Abstract
宫颈癌作为威胁女性生命的恶性肿瘤,其发生和细胞异常凋亡与高危型人乳头状瘤病毒( HPV)感染密切相关.人端粒酶逆转录酶( hTERC)基因的扩增可激活端粒酶活性而维持端粒长度,使细胞逃避凋亡而发生无限制增殖,进而发生癌变.HPV感染可致细胞DNA发生突变而致癌.我们分析了hTERC基因扩增和高危型HPV感染在宫颈上皮癌变过程中的意义及相关性,以期为临床早诊早治、监测病情和评估预后提供一定的科学依据。
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