Abstract
Abnormal triglycerides or cholesterol plasma levels characterize dyslipidemia. These changes may occur as a result of gene mutations or as a consequence of environmental factors such as food, medicine, a sedentary lifestyle, or disease. Proper maintenance of healthy concentrations of plasma lipid levels involves homeostatic fine control of various gene expressions, including epigenetics mechanisms. In this chapter, we review the main genes involved in the uptake, synthesis, metabolism, and clearance of lipids associated with the development of dyslipidemias.
Full Text 
Sign-in/Register to access full text options
Sign-in/Register to access full text options 
Published version (
Free)
Free) 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call
