Abstract
The origin(s) of amyloid, or specifically Aβ, is of the utmost importance to the information presented in the book. As presented, there are a myriad of plausible extraneuronal sources of Aβ including other cells in the brain, as well as peripheral sources of Aβ that may gain entry into the brain through a dysfunctional BBB. How extraneuronal Aβ is internalized in neurons is also presented, and includes Aβ binding proteins, as well as a variety of Aβ binding receptors.
Published Version
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