Abstract
The main mechanism of control of dengue has been through the fight against Aedes aegypti , the transmitter more competent. The fight is primarily to eliminate the potential breeding, health education and use of synthetic insecticides. These insecticides have performance but are highly toxic to vertebrates and the environment. With the emergence of resistance of mosquitoes to them, opens up an opportunity to insecticides of botanical origin. This study looks at the larvicidal activity of a solution of 3-?-acetoxylabdan-8(17)-13-dien-15-oico acid a solution of 3-?-acetoxylabdan-8(17)-13-dien-15-oico acid extracted from the medicinal plant Copaifera reticulata Ducke (Leguminosae) on the 3rd stage larvae of Ae. aegypti . Larvae were subjected to acid 9ppm for up to 24h. Those that reached lethargic state were collected and fixed in 4% in the paraformaldhyde buffer 0.1 M sodium cacodilate pH 7.2, included in resin and the slides stained with hematoxylin-eosin technique and examined by light microscope. This acid caused the death of the Ae. aegypti larvae by cell destruction in the midgut, by the high cytoplasmic vacuolation, cells and nuclei hypertrophy, degeneration of the edge brush, formation vesicular apical with release of cytoplasmic contents of cells, stratification of the epithelium and folds in the peritrophic matrix.
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