Abstract
Renal organogenesis is a complex interaction between gene stimulation of planned growth and complementary apoptosis allowing appropriate development of the functioning renal organ system, vascular bed, and intertwined genital system. The renin-angiotensin system (RAS) is critical to normal renal development through delicate interaction between the maternal–placental RAS and the developing fetal RAS that has central hormone-specific and sex-specific configurations that help the developing vascular and renal organogenesis. Preterm birth (<37 weeks gestation), as well as low birth weight (LBW), are associated with chronic kidney disease (CKD), and other associated adult diseases, and the improved survival in these groups warrant more research into long-term health optimization of these patients.
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