Syndrome In Adult Men Research Articles

Interactive associations of smoking and physical activity with metabolic syndrome in adult men in Korea.

This study aimed to investigate the association of smoking and physical activity (PA) with metabolic syndrome (MetS) in adult men in Korea. This study analyzed data of 7,229 adult men aged 19-64 years obtained from the 2014-2021 Korea National Health and Nutrition Examination Survey (KNHANES). Information on smoking habits was obtained using KNHANES data, while that on total PA (TPA), leisure-time PA (LTPA), and occupational PA (OPA) was collected using the Global Physical Activity Questionnaire. Smoking status was classified into non-smokers and smokers, and PA was categorized into three groups (total, leisure time, and occupational) according to the time spent engaging in moderate or high-intensity PA areas. The diagnosis of MetS was based on the Adult Treatment Program III of the National Cholesterol Education Program and Koreans' waist circumference criteria. Logistic regression revealed that the risk of MetS was significantly lower in non-smokers than in smokers, even after adjusting for all covariates. The risk of MetS was significantly lower in individuals who engaged in at least 150 min of moderate- and high-intensity TPA or LTPA per week than in those who did not engage in PA. Furthermore, smokers who engaged in at least 150 min of moderate- to high-intensity TPA and LTPA per week had a significantly lower risk of MetS than those who did not engage in PA. Meanwhile, OPA was not associated with MetS. The findings suggest that engaging in moderate- to high-intensity TPA or LTPA for at least 150 min per week attenuates the risk of MetS caused by smoking.

Evaluation of Applicability of Novel Markers of Metabolic Syndrome in Adult Men.

There is a continuous worldwide increase in incidences of metabolic syndrome (MetS) reaching about a quarter of the world’s population. Thus, studies that allow for a robust diagnosis of MetS are of paramount importance from an economic and medical point of view. This study was carried out in a group of men diagnosed with MetS using consensus definition criteria that included the definitions of the International Diabetes Foundation and Diabetes Federation and the American Heart Association/National Heart, Lung, and Blood Institute. The control group consisted of men for whom the parameters that define the MetS were in the norm. This study analyzed statistical differences between MetS and healthy men and the correlations between the set of 14 potential markers of MetS, that is, between body mass index, total cholesterol, high-density lipoprotein cholesterol, low-density lipoprotein, triglycerides, cortisol, adiponectin, monocyte chemotactic protein-1 (MCP-1), C-reactive protein (CRP), adipsin, leptin, resistin, and plasminogen activator inhibitor-1 (PAI)-1. This report revealed a significant difference between MetS and healthy men in most of the parameters studied. Furthermore, a strong positive correlation between cortisol levels and body mass index was demonstrated. Furthermore, MCP-1 levels in men with MetS were significantly higher than their levels in healthy men. Finally, a strong positive correlation was also observed between adiponectin and adipsin in Mets men. Thus, this study reveals the potential usefulness of adiponectin, MCP-1, adipsin, leptin, resistin, and PAI-1 as markers of MetS in adult men.

Association between unhealthy plant-based diets and the metabolic syndrome in adult men and women: a population-based study in South Korea.

No studies have investigated the associations between established plant-based diet indices and the metabolic syndrome (MetS). We evaluated the associations between an overall plant-based diet index (PDI), healthy PDI (hPDI), unhealthy PDI (uPDI) and the MetS in a nationally representative sample using data from 14450 Korean adults (≥19 years) in the Korea National Health and Nutrition Examination Survey 2012-2016. Dietary intakes were assessed by a semi-quantitative FFQ. In the PDI, all plant foods received positive scores. In the hPDI, only healthy plant foods received positive scores. In the uPDI, only unhealthy plant foods received positive scores. All indices reverse scored animal food intake. Multivariable logistic regression models were used to examine the associations between three PDI and the MetS by sex, adjusting for potential risk factors. A total of 23·3 % of Korean adults had the MetS. In the overall study population, individuals in the highest quintile of uPDI had greater odds (OR 1·54, 95 % CI 1·28, 1·86, Ptrend < 0·001) of the MetS than those in the lowest quintile. Higher uPDI score was associated with higher odds of hypertriacylglycerolaemia in men and abdominal obesity, high fasting glucose and hypertriacylglycerolaemia in women. No significant associations were observed between PDI, hPDI and the MetS. Greater adherence to unhealthy plant-based diets was associated with greater odds of the MetS and its components suggesting the importance of the quality of plant-based diet in South Korean adults. Sex differences may be considered when recommending plant-based diets for the prevention and management of metabolic diseases.

Prevalence of Metabolic Syndrome in Adult Men of the Dschang Health District in Western-Cameroon

In developing countries, increasing urbanization and lifestyle changes are helping to increase the prevalence of the metabolic syndrome among the population through the development of cardiometabolic diseases. The objective was to determine the metabolic syndrome prevalence of men aged from 30 to 60 years. A total of 254 male, initially in apparent good health who consented freely were divided into three age groups: 30-40, 41-50 and 51-60 years. They were randomly selected in 5 urban areas and 5 rural areas. After obtaining ethical clearance, patterns of diet and physical activity, data on socio-demographic conditions, alcoholism and smoking were identified by a questionnaire in a survey. Nurses measured anthropometric parameters, blood pressure and fasting blood glucose. Transaminases, uric acid, creatinine and lipids were assayed. The data collected was submitted to the EPI-InfoTM software version 7.1.5.0. The metabolic syndrome prevalence was 38.98% in the Dschang Health District and we found 31.97% and 48.6% in the rural and urban areas respectively. This prevalence of metabolic syndrome among the participants in this study showed a close association with the lifestyle, as in the drinkers with a prevalence of 71.53% and 41.67% among smokers. Only 19.3% of the university with the metabolic syndrome were identified, while those with high salt consumption had a prevalence of 34.93%. A significant number of participants had metabolic syndrome in the Dschang Health District. This high prevalence might be related to tobacco consumption, alcoholism, reduced physical activity, low educational level and poor diet.

Association between Thyroid Hormone and Risk Factors of Metabolic Syndrome in Adult Men of Normal Thyroid Function

Association between Thyroid Hormone and Risk Factors of Metabolic Syndrome in Adult Men of Normal Thyroid Function

Association between testosterone levels and the metabolic syndrome in adult men

Objective: To evaluate the relationship between testosterone levels and the metabolic syndrome (MS) in men older than 45 years.Methods: Six hundred and sixty men (45–70 years) selected from 2906 participants of a population screening for prostate cancer were included in this study. Testosterone and the components of MS were assessed in all men. MS was diagnosed according to NCEP-ATP III criteria. Triglycerides (TG)/HDL-cholesterol (chol) index was calculated.Results: The presence of MS was inversely associated with testosterone (χ2, p < 0.001), independently of age (OR 0.802, CI 95%: 0.724–0.887, p < 0.0001). Hypertension was the most frequent abnormality observed followed by elevated TG and waist circumference (WC). Testosterone correlated positively with HDL-chol (r: 0.14, p < 0.0001) and negatively with body mass index (BMI)(r: −0.29, p < 0.0001), WC (r: −0.26, p < 0.0001), TG (r: −0.20, p < 0.0001), TG/HDL-chol (r: −0.20, p < 0.0001), glucose (r: −0.11, p = 0.005) and MS score (r: −0.23, p < 0.0001).Conclusions: Our results show that in men older than 45 years, as long as testosterone levels decline, the prevalence of MS increases, independently of age. The correlations found between testosterone and four of the five components of MS, as well as with BMI and TG/HDL-chol ratio, a surrogate marker of insulin resistance, suggest considering male hypogonadism as a determinant of developmental abnormalities typical of MS.

Binge drinking and metabolic syndrome.

Dear Editor, We would like to discuss the topic 'binge and metabolic syndrome.'1) Im et al.1) noted for positive relationship between frequency of and metabolic syndrome in adult men. In an animal model study, the effect of on insulin metabolism can be seen. Lindtner et al.2) reported that binge induces systemic insulin resistance by impairing hypothalamic insulin action and that this effect can be prevented by inhibition of brain protein tyrosine phosphatase 1B. However, an important consideration of the present report by Im et al.1) is the confounding factor that can lead to the problem of metabolic syndrome. The drinker usually demonstrates other concomitant risk behaviors such as smoking,3) hence, it is difficult to conclude the effect of without consideration of other factors. Finally, it should be noted that measurement of binge drinking should be discussed. Normally, the problem of reliability and accuracy of determination is common. Matano et al.4) noted that accurate methods for assessing appear to be by directly asking for the largest number of drinks consumed in a single session. Yours Sincerely

Binge Drinking and Its Relation to Metabolic Syndrome in Korean Adult Men

BackgroundIt is reported that heavy drinking increases the risk of metabolic syndrome. But there have been few studies on the relationship between the intensity of drinking and metabolic syndrome when drinking the same amount of alcohol. This study aimed to assess the relationship between the frequency of binge drinking and metabolic syndrome in Korean adult men.MethodsFrom the database of the 4th and 5th Korea National Health and Nutrition Examination Survey conducted in 2007-2010, data of 8,305 adult men (≥19 years of age) was included in this analysis. Cross-sectional relationship between the frequency of binge drinking and metabolic syndrome was investigated adjusting for pure alcohol consumed per day.ResultsAdjusting for various confounders including pure alcohol consumed per day, the adjusted odds ratio for metabolic syndrome in those in higher frequency (more than 1/wk) binge drinking group was 1.62 (95% confidence interval, 1.30 to 2.03; P for trend = <0.001) compared to those in the non-binge drinking group. Through analysis of the relationship between pure alcohol consumed per day and metabolic syndrome, it was found that pure alcohol consumed per day had a positive relation to metabolic syndrome in the higher frequency binge drinking group (P for trend = 0.041). The relationship was inverse in the non-binge drinking group (P for trend = 0.002).ConclusionOur study found a positive relationship between frequency of binge drinking and metabolic syndrome in adult men. And the effect of drinking on metabolic syndrome may depend on the frequency of binge drinking. Further studies are required to confirm this association.

A moderate carbohydrate‐restricted diet results in weight loss and improves clinical parameters of metabolic syndrome in adult men and women and addition of egg yolk further improves inflammation

Men and women (n = 18) with metabolic syndrome (MetS) were instructed to follow a carbohydrate (CHO)‐restricted diet (&lt;30% energy) and randomly assigned to consume either 3 whole eggs (EGG, n = 9) or egg substitute (SUB, n = 9) per day for 12 wk. Dietary intake, parameters of MetS, inflammatory markers and body composition by DXA were assessed at baseline, wk 6 and wk 12. Total calories (p&lt;0.0001), total CHO (p&lt;0.0001), % CHO energy (p&lt;0.001), and glycemic index (p&lt;0.0001) decreased in all subjects over time. Reductions in weight (p&lt;0.0001), body mass index (p&lt;0.0001), fat mass (p&lt;0.0001), and trunk fat (p&lt;0.0001) were also observed. MetS parameters were improved with increases in plasma HDL‐cholesterol (p&lt;0.01) and decreases in waist circumference (p&lt;0.01), plasma glucose (p&lt;0.05) and plasma triglycerides (TG) (p&lt;0.01) with no differences between groups. Reductions in total CHO correlated with reductions in weight (0.484, p&lt;0.05) and plasma TG (0.483, p&lt;0.05). The EGG group consumed more cholesterol (p&lt;0.0001) and choline (p&lt;0.0001) than the SUB group. There was a greater reduction in plasma tumor necrosis factor‐α (TNFα) in EGG vs. SUB groups (p&lt; 0.05) and increases in adiponectin (0.419 vs 0.014 mg/L). These results suggest that the addition of egg yolk does not suppress the beneficial effects of carbohydrate‐restriction in MetS and may modulate plasma TNFα and adiponectin. [Supported by the American Egg Board].Grant Funding Source: American Egg Board/Egg Nutrition Center

Abstract MP015: Resistance Exercise and Incident Metabolic Syndrome in Men

Introduction: There is little evidence on the association between resistance exercise and incident metabolic syndrome. Hypothesis: We examined the hypothesis that resistance exercise reduces the risk of developing metabolic syndrome in men. Methods: Participants were 6,098 men aged 18 to 83 years (mean age, 46) who received an extensive preventive medical examination during 1987–2006, and were free of cardiovascular disease, cancer, and metabolic syndrome at baseline. Resistance exercise was assessed by self-reported muscle strengthening activities using either free weights or weight training machines. We used the frequency of resistance exercise (0, 1, 2, 3, 4, and ≥5 days/week) as our main exposure categories. Metabolic syndrome was defined as the presence of ≥3 of the following criteria: waist circumference &gt;102 cm, triglycerides ≥150 mg/dL, high-density lipoprotein cholesterol &lt;40 mg/dL, systolic/diastolic blood pressure ≥130/85 mm Hg or physician-diagnosed hypertension, and fasting glucose ≥100 mg/dL or physician-diagnosed diabetes according to the National Cholesterol Education Program Adult Treatment Panel III. Follow-up was calculated from the baseline examination to the first event of metabolic syndrome or the last examination through 2006. Cox regression analysis was used to estimate the hazard ratio (HR) and 95% confidence interval (CI) of incident metabolic syndrome across resistance exercise. Age, examination year, body mass index, current smoking, heavy alcohol drinking (&gt;14 drinks/week), abnormal electrocardiography, and aerobic exercise (inactive, low, medium, and high) were included in the multivariate regression model. Results: During the 5-year mean follow-up, 1,088 men developed metabolic syndrome. About 62% of men did not participate in resistance exercise, 3% of men participated in 1 day/week of resistance exercise, and 35% of men met the Physical Activity Guidelines for resistance exercise (≥2 days/week). Resistance exercise was inversely associated with incident metabolic syndrome after adjusting for possible confounders including aerobic exercise (trend P &lt;0.01). Compared with no resistance exercise, the HRs (95% CIs) of developing metabolic syndrome were 0.83 (0.55–1.26), 0.88 (0.70–1.11), 0.85 (0.72–1.02), 0.62 (0.43–0.88), and 0.74 (0.51–1.07) for 1, 2, 3, 4, and ≥5 days/week of resistance exercise, respectively. Men meeting the recommended resistance exercise (≥2 days/week) had 18% lower risk of developing metabolic syndrome (HR=0.82, 95% CI=0.71–0.94) compared with men not meeting the recommendation (0 or 1 day/week of resistance exercise). Conclusions: Participating in resistance exercise may reduce the risk of developing metabolic syndrome in adult men.

Excessive fructose intake induces the features of metabolic syndrome in healthy adult men: role of uric acid in the hypertensive response

Excessive fructose intake causes metabolic syndrome in animals and can be partially prevented by lowering the uric acid level. We tested the hypothesis that fructose might induce features of metabolic syndrome in adult men and whether this is protected by allopurinol. A randomized, controlled trial of 74 adult men who were administered 200 g fructose daily for 2 weeks with or without allopurinol. Primary measures included changes in ambulatory blood pressure (BP), fasting lipids, glucose and insulin, homeostatic model assessment (HOMA) index, body mass index and criteria for metabolic syndrome. The ingestion of fructose resulted in an increase in ambulatory BP (7+/-2 and 5+/-2 mm Hg for systolic (SBP) and diastolic BP (DBP), P<0.004 and P<0.007, respectively). Mean fasting triglycerides increased by 0.62+/-0.23 mmol l(-1) (55+/-20 mg per 100 ml), whereas high-density lipoprotein cholesterol decreased by 0.06+/-0.02 mmol l(-1) (2.5+/-0.7 mg per 100 ml), P<0.002 and P<0.001, respectively. Fasting insulin and HOMA indices increased significantly, whereas plasma glucose level did not change. All liver function tests showed an increase in values. The metabolic syndrome increased by 25-33% depending on the criteria. Allopurinol lowered the serum uric acid level (P<0.0001) and prevented the increase in 24-h ambulatory DBP and daytime SBP and DBP. Allopurinol treatment did not reduce HOMA or fasting plasma triglyceride levels, but lowered low-density lipoprotein cholesterol relative to control (P<0.02) and also prevented the increase in newly diagnosed metabolic syndrome (0-2%, P=0.009). High doses of fructose raise the BP and cause the features of metabolic syndrome. Lowering the uric acid level prevents the increase in mean arterial blood pressure. Excessive intake of fructose may have a role in the current epidemics of obesity and diabetes.

Association of Lower Urinary Tract Symptoms with Irritable Bowel Syndrome in Adult Men - An Internet-based Survey

Patients with irritable bowel syndrome (IBS) are more likely to experience certain urinary symptoms. The aims of this study were to investigate the association between lower urinary tract symptoms (LUTS) and IBS, and to determine their impact on the quality of life. E-mails were sent to 23,594 men who were registered at an internet survey company. Subjects were requested to fill out the questionnaires regarding IBS and LUTS assessed by the International Prostate Symptom Score (IPSS). Among 601 subjects (mean age, 35.5+/-8.4) included in the final analysis, 118 (19.6%) fulfilled the Rome II criteria for the diagnosis of IBS. The total mean IPSS of IBS subjects was 9.6, which was significantly higher than the 7.0 of non-IBS subjects (p<0.01). When IPSS was subcategorized into mild, moderate, and severe symptom categories, the proportions with the moderate and severe symptoms among IBS subjects were 33.9% and 13.6% respectively, which were significantly higher than those of non-IBS subjects; (26.9 and 5.2%) (p<0.01). In multivariate analysis, statistically significant association was found between IBS and moderate to severe LUTS (OR, 1.91; 95% CI, 1.27-2.88). IBS subjects also showed a poorer quality of life score than non-IBS subjects (2.24 vs. 1.65, p< 0.001). LUTS in adult men with IBS are more severe and have a more negative impact on the quality of life than in non-IBS subjects.

대사증후군을 가진 성인남녀의 영양소 섭취상태와 혈액성상에 관한 연구

The purpose of this study is to evaluate the nutrient intake and selected blood parameters of adults with metabolic syndrome (MS) and to provide data in forming a dietary guideline for the prevention of chronic diseases. Subjects were recruited and divided into two groups according to the NCEP?ATP Ⅲ criteria and WHO Asia?Pacific Area criteria for obesity. MS group was defined as subjects who have three or more risk factors and control group was defined as those with two or less of the risk factors. The average age, height, weight, body mass index (BMI) were 58.8 years, 158.0 ㎝, 66.3 ㎏, 26.5 ㎏/㎡, respectively, in the MS group; and 58.4 years, 158.9 ㎝, 59.6 ㎏, 23.5 ㎏/㎡, respectively, in the control group. The weight and BMI in the MS group were significantly higher than those in the control (p＜0.001). There was no significant difference in the food and nutrient intake between the MS and control group. Male subjects in the MS group showed significantly higher intake of mushrooms than those in the control (p＜0.05). Egg consumption in the MS group was significantly lower than those in the control (p＜0.01). Consumption of vegetables and fiber was significantly lower for female subjects in the MS group than those in the control (p＜0.05). Serum GPT, AI and WBC count in the MS group (27.8 IU/L, 3.7, 5964.2 μ/L) were significantly higher than those in the control (22.6 IU/L, 3.2, 5250.0 μ/L; p＜0.01, p＜0.001, p＜0.01). In conclusion, consuming fiber and vegetables may prevent and reduce metabolic syndrome in adult men and women, and this study demonstrates the need for proper dietary management for them.

Impact of parental smoking on diabetes, hypertension and the metabolic syndrome in adult men and women in the San Antonio Heart Study

In the San Antonio Heart Study (SAHS) we investigated the effects of exposure to parental smoking on diabetes, hypertension and the metabolic syndrome in adult offspring aged 25-64 years. In a retrospective cohort study the parental smoking status during childhood, obtained through a postal questionnaire, determined a person's exposure status. Logistic regression models were used to calculate odds ratios for diabetes, hypertension and the metabolic syndrome at the baseline SAHS examination in relation to parental smoking status. All models were adjusted for age, sex, ethnicity, education years, personal smoking status (current, former or never-smoker), BMI and, in the case of diabetes, a family history of diabetes. Of the 2,371 participants who returned the mailing, 44.5, 5.4, 20.0 and 30.1% reported that their father, mother, both or neither parent smoked, respectively. Participants reporting that both parents smoked were 1.60 (95% CI: 0.95-2.69) times more likely to have diabetes, 1.55 (95% CI: 1.05-2.28) times more likely to have hypertension, and 1.46 (95% CI: 1.01-2.10) times more likely to have the metabolic syndrome than participants reporting that neither parent smoked during their childhood. Odds ratios, after limiting the population to younger participants (i.e. <or=50 years) to reduce survivor bias, were 2.53 (95% CI: 1.21-5.31), 1.29 (95% CI: 0.78-2.16), and 1.41 (95% CI: 0.89-2.22) for diabetes, hypertension and the metabolic syndrome, respectively. These results provide evidence that early exposure to parental smoking may be associated with an increased risk of diabetes and perhaps hypertension and the metabolic syndrome.

Epidemiology of the metabolic syndrome in 2045 French military personnel (EPIMIL study)

The Metabolic syndrome is considered to be an important public health problem, but few epidemiological studies have defined the present situation in France. EPIMIL is a prospective epidemiological study that began on February 1, 2003. This study was designed to identify the clinical and laboratory parameters of metabolic syndrome and cardiovascular risk factors in a population of 2045 male military personnel based in the Paris region. The initial 1-year cross-sectional study will be followed by a 10-year follow-up and patient care. The 2045 subjects included 185 (9%) presented at least 3 of the 5 NCEP ATP III criteria defining metabolic syndrome. They were significantly older (42.2 +/- 8.5 yrs) than the other subjects (37.3 +/- 8.7 yrs, P<0.001), had a higher BMI (29.5 +/- 3.4 vs 24.8 +/- 2.9 kg/m2, P<0.001) and a greater body weight at age 20 (75.4 +/- 11 vs 70.4 +/- 8.5 kg, P<0.01). Smoking, little physical activity and family histories of diabetes and arterial hypertension were more frequent in these men. While levels of the cholesterol and CRP us were higher, Lp(a) and homocysteine concentrations were normal. Plasma insulin and BMI (r = 0.456 P<0.0001) and plasma insulin and waist circumference (r = 0.446 P<0.0001) were well correlated. Lastly, plasma insulin, free fatty acids and cardiovascular risk increased steadily with the increase in the number of metabolic syndrome criteria. These results in a large and particularly uniform population of men show the prevalence of metabolic syndrome in adult men, and demonstrate its link with insulin resistance. Men with several risk factors require specific care, particularly for hypertension and dyslipidemia; the effectiveness of this care will be evaluated during the follow-up period.

Metabolic Syndrome in a Screened Worksite Sample: Prevalence and Predictors

Relatively limited contemporary information is available about the magnitude of, and factors associated with, the metabolic syndrome in adult men and women. The purpose of our observational study was to describe the prevalence and predictors of the metabolic syndrome in a sample of employed adults attending a worksite cardiovascular screening program. The study sample consisted of 871 men and women between the ages of 21 and 77 years from 6 locations of the parent company. These individuals attended an employer-sponsored cardiovascular screening and wellness program during 2003. A standardized questionnaire was administered to all study participants and a number of different coronary risk factors were measured. Approximately 27% of the study sample was classified as having the metabolic syndrome. Men, persons with a history of hypertension, heart disease, or stroke, sedentary individuals, and those with an increased heart rate and higher levels of C-reactive protein were associated with presence of the metabolic syndrome. A relatively similar risk factor profile was noted in persons without a self-reported history of prior cardiovascular disease. The results of our cross-sectional observational study suggest that the prevalence of the metabolic syndrome is considerable. A number of demographic, comorbid, and other factors are associated with this syndrome. Increased attention to the metabolic syndrome, and modification of predisposing factors, remains of considerable public health and clinical importance.

AGAMMAGLOBULINEMIA IN ADULTS

Case Reports1 March 1957AGAMMAGLOBULINEMIA IN ADULTSJ. W. SAVACOOL, M.D., F.A.C.P., RAY P. LANDES, M.D.J. W. SAVACOOL, M.D., F.A.C.P.Search for more papers by this author, RAY P. LANDES, M.D.Search for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-46-3-628 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptThe purpose of this report is to call attention to another case of agammaglobulinemia in an adult, to emphasize a few of the features of this interesting process, and to add to the accumulating data with respect to its association with various relatively common pathologic processes.CASE REPORTA 38 year old white male clerk was admitted to Grand View Hospital May 29, 1954, with an 18 month history of recurrent episodes of fever, chills, sweats, coughs, expectoration, bilateral anterior chest pain and weakness.As a child he had apparently had good health, having had only measles. At about the...Bibliography1. SanfordFavourTribeman JPCBMS: Absence of serum gamma globulin in an adult, New England J. Med. 250: 1027-1029 (June 17) 1954. CrossrefMedlineGoogle Scholar2. Bruton OC: Agammaglobulinemia, Pediatrics 9: 722-728 (June) 1952. MedlineGoogle Scholar3. PrasadKoza ASDW: Agammaglobulinemia, Ann. Int. Med. 41: 629-639 (Sept.) 1954. LinkGoogle Scholar4. ZinnemanHallHeller HHWHBI: Acquired agammaglobulinemia, J. A. M. A. 156: 1390-1393 (Dec. 11) 1954. CrossrefMedlineGoogle Scholar5. RohmBehnkeBond RJRHWH: Acquired agammaglobulinemia and hypersplenism, J. Lab. and Clin. Med. 44: 918, 1954. Google Scholar6. GrantWallace AHWD: Agammaglobulinemia, Lancet 2: 671-673 (Oct. 2) 1954. CrossrefGoogle Scholar7. WallSaslaw RLS: Adult agammaglobulinemia, Arch. Int. Med. 95: 33-36 (Jan.) 1955. CrossrefGoogle Scholar8. Verschure JD: Gammaglobulinopenia, Nederl. tijdschr. v. geneesk. 98: 2805, 1954. MedlineGoogle Scholar9. CraigGitlinJewett JMDTC: The response of lymph nodes of normal and congenitally agammaglobulinemic children to antigenic stimulation, Am. J. Dis. Child. 88: 626 (Nov.) 1954. Google Scholar10. JanewayCraigDavidsonDowneyGitlinSullivan CAJAMWDJC: Hypergammaglobinemia associated with severe recurrent and chronic nonspecific infection, Am. J. Dis. Child. 88: 388, 1954. Google Scholar11. HamburgerPetermann FMC: Studies on hypoproteinemia. II. Familial idiopathic dysproteinemia: Blood 4: 1085-1108 (Oct.) 1949. CrossrefMedlineGoogle Scholar12. Good RA: Agammaglobulinemia: a provocative experiment of Nature, Bull. Univ. Minnesota Hospitals and Minnesota Med. Foundation 26: 1, 1954. Google Scholar13. YoungWolfson IIWQ: The agammaglobulinemia syndrome in adult men: its differentiation into familial lymphopenic agammaglobulinemia, Clin. Res. Proc. 2: 101, 1954. Google Scholar14. GoodKelley RAVC: Adrenal cortical function in patients with agammaglobulinemia, Proc. Soc. Exper. Biol. and Med. 88: 99-101 (Jan.) 1955. CrossrefMedlineGoogle Scholar15. LaskiSass-KorstakHillman BADA: Cyclic neutropenia and agammaglobulinemia, Am. J. Dis. Child. 88: 820-821, 1954. Google Scholar16. ReinerStern MKG: Electrophoretic studies on the protein distribution in the serum of multiple myeloma patients, Acta hematol. 9: 19-29 (Jan.) 1953. CrossrefMedlineGoogle Scholar17. Brownell EG: Multiple myeloma, Arch. Int. Med. 95: 699-704 (May) 1955. CrossrefGoogle Scholar18. Reimann HA: Periodic disease: report of old cases, new cases and therapy, Arch. Int. Med. 92: 494-506 (Oct.) 1953. CrossrefGoogle Scholar This content is PDF only. To continue reading please click on the PDF icon. Author, Article, and Disclosure InformationAffiliations: Philadelphia, Pennsylvania*Received for publication November 5, 1955.From the Barton Division and the Department of Medicine, Jefferson Medical College Hospital, Philadelphia, and the Grand View Hospital, Sellersville, Pennsylvania.Requests for reprints should be addressed to J. W. Savacool, M.D., 146 West Tulpehocken Street, Philadelphia 44, Pennsylvania. PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics Cited byIdiopathic Acquired Agammaglobulinemia Associated with ThymomaHOST-PARASITE RELATIONSHIPS IN PATIENTS WITH DYSPROTEINEMIAS 1 March 1957Volume 46, Issue 3Page: 628-637KeywordsAntibioticsChild healthFeversHospital medicineIschemiaMeaslesPneumoniaProteinsSweatUpper respiratory tract infections Issue Published: 1 March 1957 PDF downloadLoading ...

Chronic meningococcemia.

Case Reports1 July 1956CHRONIC MENINGOCOCCEMIAMAURICE GORE, M.D.MAURICE GORE, M.D.Search for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-45-1-142 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptChronic meningococcemia without the presence of meningitis is an uncommon clinical entity, particularly when this occurs in an isolated instance. During local outbreaks or in endemic areas, meningococcemia occurs frequently and can be easily diagnosed. According to Feddersen,1the first description of chronic meningococcemia was published by Solomon.2Feddersen reported that there were over 100 cases in the European literature3-6up to 1945, and stated that there were fewer cases in the American literature7-11because full investigations were not the usual procedure. With the advent of sulfonamide therapy, treatment was often started before adequate diagnostic studies were made. Since meningococcemia...Bibliography1. Feddersen AS: Meningeal septicemia, M. J. Australia 2: 47-48 (July 14) 1945. CrossrefGoogle Scholar2. Solomon H: Ueber Meningokokkenseptikämie, Berl. klin. Wchnschr. 39: 1045 (Nov. 10) 1902. Google Scholar3. Denison SB: Chronic meningococcic septicemia treated with 2-(p-aminobenzene-sulfonamide) pyridine, Lancet 2: 424 (Aug. 20) 1938. Google Scholar4. StottCopeman AWWS: Chronic meningococcic septicemia associated with an outbreak of cerebro-spinal fever, Lancet 1: 617 (June 22) 1940. Google Scholar5. Lane J: Chronic meningococcemia, M. J. Australia 2: 617 (Dec. 9) 1944. CrossrefGoogle Scholar6. DicksonMcKinnonMaguerMcGillwray RCNEDNB: Meningococcal infection, Lancet 2: 631 (Nov. 22) 1941. CrossrefGoogle Scholar7. SchwentkerGelmanLong FFSPJ: The treatment of meningococcic meningitis with sulfonamides, J. A. M. A. 58: 1407 (Apr. 24) 1937. CrossrefGoogle Scholar8. ZendelGreenberg JFD: Meningococcemia: treatment with sulfanilamide and Prontosil. Report of two cases, New York State J. Med. 37: 1744 (Oct. 15) 1937. Google Scholar9. Norman JK: Chronic meningococcemia, Mil. Surgeon 97: 455-457 (Dec.) 1945. MedlineGoogle Scholar10. CarconellCampbell AE: Prolonged meningococcemia, Arch. Int. Med. 61: 646 (Apr.) 1938. CrossrefGoogle Scholar11. Appelbaum E: Chronic meningococcus septicemia, Am. J. M. Sc. 193: 96-108 (Jan.) 1937. CrossrefGoogle Scholar12. Bruton OC: Agammaglobulinemia, Pediatrics 9: 722-728 (June) 1952. CrossrefMedlineGoogle Scholar13. BrutonAptJaneway OCLC: Absence of serum gamma globulins, Am. J. Dis. Child. 84: 632-636 (Nov.) 1952. Google Scholar14. WallSaslaw RLS: Adult agammaglobulinemia, Arch. Int. Med. 95: 33-36 (Jan.) 1955. CrossrefGoogle Scholar15. YoungWolfson IIQ: The agammaglobulinemia syndrome in adult men; its differentiation into familial lymphopenic agammaglobulinemia and familial non-lymphopenic dysglobulinemia, Clin. Res. Proc. 2: 101 (Apr.) 1954. Google Scholar16. SanfordFavourTribeman JPCBMS: Absence of serum gamma globulin in an adult, New England J. Med. 250: 1027-1029 (June) 1954. CrossrefMedlineGoogle Scholar This content is PDF only. To continue reading please click on the PDF icon. Author, Article, and Disclosure InformationAffiliations: Chicago, Illinois*Received for publication April 29, 1955.Requests for reprints should be addressed to Maurice Gore, M.D., 104 South Michigan Avenue, Chicago-3, Illinois. PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics Cited byDurch Gram-negative Bakterien bedingte Hauterscheinungen bei septischen KrankheitenMeningococcal arthritisChronic meningococcemiaChronic Meningococcemia 1 July 1956Volume 45, Issue 1Page: 142-145KeywordsArthritisErythemaMeningitisMeningococcemiaSulfonamide ePublished: 1 December 2008 Issue Published: 1 July 1956 PDF downloadLoading ...