Severe Bouts Research Articles

Peripheral radiculoneuritis complicating pregnancy

This case of peripheral radiculoneuritis complicating pregnancy was relieved by the termination of the pregnancy by cesarean section, with favorable results. The patient did not respond to intensive vitamin therapy, as suggested in previous papers, and had no history of the severe and/or protracted bouts of vomiting that had been reported in this type of case prior to this writing.

Rupture of pulmonary aneurysm accompanying patent ductus arteriosus; occurrence in a 67 year old woman.

The rupture of a pulmonary aneurysm has been infrequently reported. More unusual is its occurrence secondary to patent ductus arteriosus in a patient who is beyond the age of 67. The concomitant finding of a dissecting aneurysm of the thoracic aorta is of extreme interest. This combination of major vascular lesions is most unusual and warrants presentation. REPORT OF CASE The patient was a 67 year old white woman who was admitted for the last time to Milwaukee County General Hospital on Sept. 5, 1943 with headaches, mental confusion and severe bouts of dizziness of about twenty-four hours' duration. Past history revealed that the patient had been known to be hypertensive since 1928, at which time she had first had headaches and dizzy spells. Since 1934 she had been observed in the Out-Patient Medical Department of the Milwaukee County Dispensary, where she had been treated for benign hypertension. Her first

CARBOHYDRATE METABOLISM

In the course of some observations made five years ago on children recovering from severe bouts of vomiting, I noted a failure of elevation of the respiratory quotient following the intravenous administration of glucose. Because such states involve so many variables, viz., an accompanying infection, emotion, alteration in the acid-base balance or similar influences, the present studies were undertaken in order to analyze further the factors affecting the utilization of intravenously administered glucose. Lehmann 1 in 1874 and Hofmeister 2 in 1890 described diabetes due in the first instance to either arsenic poisoning or starvation and in the second to starvation. The similarity of the states induced by arsenic poisoning and by starvation naturally suggested that the site of the metabolic flaw was in the liver. This idea was further developed by the work of Barrenschen 3 in 1914 and by that of Nagasuye 4 in 1925. The reproduction of