Anterior chest wall is composed of soft tissue and skeletal supporting structures. The soft tissues are largely the skin, respiratory muscles and the parietal pleura. The ribs offer rigidity which functions to protect the intra-thoracic structures and more importantly aids in coordinating respiratory movements and lung expansion. Adequate replacement of these anatomical components of the chest wall is critical to restoration of function following a defect. Anterior chest wall defects could be either full thickness or partial thickness in terms of tissue lost. There are several aetiological factors that can result in chest deformities namely tumor resection, infections, radiation necrosis, congenital malformations and trauma [1]. High voltage electrical injury is a traumatic form of injury that can affect many organ systems with protean presentation. The chest wall and its contents can be damaged by electrical current with resultant high necrotic load and superimposed infection. Interestingly, the increased metabolic demands placed on a major electrical burn due to