Epidemic Spreading In Multiplex Networks Research Articles

The coevolution of the spread of a disease and competing opinions in multiplex networks

The COVID-19 pandemic has resulted in a proliferation of conflicting opinions on physical distancing across various media platforms, which has had a significant impact on human behavior and the transmission dynamics of the disease. Inspired by this social phenomenon, we present a novel UAP-SIS model to study the interaction between conflicting opinions and epidemic spreading in multiplex networks, in which individual behavior is based on diverse opinions. We distinguish susceptibility and infectivity among individuals who are unaware, pro-physical distancing and anti-physical distancing, and we incorporate three kinds of mechanisms for generating individual awareness. The coupled dynamics are analyzed in terms of a microscopic Markov chain approach that encompasses the aforementioned elements. With this model, we derive the epidemic threshold which is related to the diffusion of competing opinions and their coupling configuration. Our findings demonstrate that the transmission of the disease is shaped in a significant manner by conflicting opinions, due to the complex interaction between such opinions and the disease itself. Furthermore, the implementation of awareness-generating mechanisms can help to mitigate the overall prevalence of the epidemic, and global awareness and self-awareness can be interchangeable in certain instances. To effectively curb the spread of epidemics, policymakers should take steps to regulate social media and promote physical distancing as the mainstream opinion.

Suppressing traffic-driven epidemic spreading in multiplex networks by effective traffic-flow assignment strategy

Despite extensive research on the interplay between traffic dynamics and epidemic spreading, the control of epidemic spreading in multiplex networks by routing strategies has not received adequate attention. In this paper, we study the impact of effective traffic-flow assignment strategy on epidemic spreading in multiplex networks. Interestingly, epidemic spreading can be effectively controlled by tuning the routing assignment in a certain layer. In particular, by adjusting the routing parameters, the epidemic can be completely suppressed. Moreover, an increase in the average network connectivity of one layer of the multiplex networks can also suppress the outbreak of epidemic spreading. By both simulation and theoretical analysis, the epidemic suppression effect is confirmed on multiplex networks composed of Erdős–Rényi random and Scale-Free layers.

The coupled dynamics of information dissemination and SEIR-based epidemic spreading in multiplex networks

The outbreak of coronavirus disease 2019 (COVID-19) threatens the health and safety of all humanity. This disease has a prominent feature: the presymptomatic and asymptomatic viral carriers can spread the disease. It is crucial to estimate the impact of this undetected transmission on epidemic outbreaks. Currently, disease-related information has been widely disseminated by the mass media. To investigate the impact of both individuals and mass media information dissemination on the epidemic spreading, we establish a new UAU-SEIR (Unaware–Aware–Unaware–Susceptible–Exposed–Infected–Recovered) model with mass media on two-layer multiplex networks. In the model, E-state individuals denote asymptomatic infections, and a single node connecting to all individuals denotes the mass media. In this work, we use the Microscopic Markovian Chain Approach (MMCA) to derive the epidemic threshold. Comparing the MMCA theoretical results with Monte Carlo (MC) simulations, we find that the MMCA has a good consistency with MC simulations. In addition, we also analyze the impact of model parameters on epidemic spreading and epidemic threshold. The results show that reducing the proportion of asymptomatic infections, accelerating the dissemination of information between individuals and the dissemination of information via the mass media can effectively inhibit the epidemic spreading and raise the epidemic threshold.

Traffic-driven epidemic spreading in multiplex networks.

Recent progress on multiplex networks has provided a powerful way to abstract the diverse interaction of a network system with multiple layers. In this paper, we show that a multiplex structure can greatly affect the spread of an epidemic driven by traffic dynamics. One of the interesting findings is that the multiplex structure could suppress the outbreak of an epidemic, which is different from the typical finding of spread dynamics in multiplex networks. In particular, one layer with dense connections can attract more traffic flow and eventually suppress the epidemic outbreak in other layers. Therefore, the epidemic threshold will be larger than the minimal threshold of the layers. With a mean-field approximation, we provide explicit expressions for the epidemic threshold and for the onset of suppressing epidemic spreading in multiplex networks. We also provide the probability of obtaining a multiplex configuration that suppresses the epidemic spreading when the multiplex is composed of: (i) two Erdős-Rényi layers and (ii) two scale-free layers. Therefore, compared to the situation of an isolated network in which a disease may be able to propagate, a larger epidemic threshold can be found in multiplex structures.

Influence of geometric correlations on epidemic spreading in multiplex networks

Real-world multiplex networks are not randomly connected by several single-layer networks. In fact, there exist significant geometric correlations among different layers. In this paper, the influence of the geometric correlations, including the radial correlation and the angular correlation, on epidemic spreading on multiplex networks is investigated. The results show that the radial correlation and/or the angular correlation can reduce the epidemic threshold and lead to smaller scale of outbreak on the artificial networks. Moreover, although geometric correlations and overlapped links are positively correlated, the influence of geometric correlations on epidemic spreading cannot be replaced by overlapped links. Simulations on a real-world multiplex network indicate that the geometric correlations can produce lower threshold and smaller scale of outbreak.

Epidemic spreading in multiplex networks with heterogeneous infection rate

The awareness behaviour prevents infection, and the epidemic spreading affects awareness spreading. The interactions between human awareness and epidemic spreading are often described in terms of multiplex networks. Many studies have revealed the heterogeneity of infection rates due to different health habits, social conditions, physical strength, and contact frequency between individuals. In this paper, we consider the epidemic spreading process in multiplex networks with heterogeneous infection rate. We construct a heterogeneous infection rate function whose tunable power exponent is related to the degree of the node. The epidemic threshold is obtained by the Markov chain and mean-field approach. Numerical simulations of scale-free, small-world and random networks show that the fraction of infected individuals and epidemic threshold are affected by the power exponent, and the negative heterogeneous infection rate is more conducive to preventing epidemic. Immunization of hub nodes is a good method to mitigate an epidemic.

A colored mean-field model for analyzing the effects of awareness on epidemic spreading in multiplex networks.

We study the impact of susceptible nodes' awareness on epidemic spreading in social systems, where the systems are modeled as multiplex networks coupled with an information layer and a contact layer. We develop a colored heterogeneous mean-field model taking into account the portion of the overlapping neighbors in the two layers. With theoretical analysis and numerical simulations, we derive the epidemic threshold which determines whether the epidemic can prevail in the population and find that the impacts of awareness on threshold value only depend on epidemic information being available in network nodes' overlapping neighborhood. When there is no link overlap between the two network layers, the awareness cannot help one to raise the epidemic threshold. Such an observation is different from that in a single-layer network, where the existence of awareness almost always helps.

Pair quenched mean-field approach to epidemic spreading in multiplex networks

• A pair-quenched mean-field approach to epidemic spreading on multiplex networks is developed. • The condition for the epidemic threshold in a static multiplex network is derived. • Gillespie algorithm-based simulations staged on various multilayered network topologies were conducted. • Theoretical results are in agreement with numerical simulations on 2-layer and 3-layer complex networks. • Our findings highlight the efficiency of the presented approach for assessing epidemic thresholds in multiplex networks. Using the law of total probability, we extend the pair quenched mean-field approach for epidemic spreading in monoplex networks to the scenario of contagion outbreaks in multiplex networks. By means of the quasi-static approximation, we derive the condition for the epidemic threshold in a static multiplex network overlapped by the randomly connected subnetwork without clustering. Our theoretical results are in good agreement with continuous-time Gillespie algorithm-based simulations for 2-layer and 3-layer multiplex networks, revealing the advantage of our model in the prediction of epidemic spreading relative to the quenched mean-field (QMF) approach. Importantly, our study demonstrates that unlike the standard QMF approach, the pair QMF model can be used to assess the influence of the link overlap on the epidemic threshold, thereby carrying vital implications for future epidemiological research and policy development.

Suppressing epidemic spreading in multiplex networks with social-support

Although suppressing the spread of a disease is usually achieved by investing in public resources, in the real world only a small percentage of the population have access to government assistance when there is an outbreak, and most must rely on resources from family or friends. We study the dynamics of disease spreading in social-contact multiplex networks when the recovery of infected nodes depends on resources from healthy neighbors in the social layer. We investigate how degree heterogeneity affects the spreading dynamics. Using theoretical analysis and simulations we find that degree heterogeneity promotes disease spreading. The phase transition of the infected density is hybrid and increases smoothly from zero to a finite small value at the first invasion threshold and then suddenly jumps at the second invasion threshold. We also find a hysteresis loop in the transition of the infected density. We further investigate how an overlap in the edges between two layers affects the spreading dynamics. We find that when the amount of overlap is smaller than a critical value the phase transition is hybrid and there is a hysteresis loop, otherwise the phase transition is continuous and the hysteresis loop vanishes. In addition, the edge overlap allows an epidemic outbreak when the transmission rate is below the first invasion threshold, but suppresses any explosive transition when the transmission rate is above the first invasion threshold.

Epidemic spreading in multiplex networks influenced by opinion exchanges on vaccination.

Through years, the use of vaccines has always been a controversial issue. People in a society may have different opinions about how beneficial the vaccines are and as a consequence some of those individuals decide to vaccinate or not themselves and their relatives. This attitude in face of vaccines has clear consequences in the spread of diseases and their transformation in epidemics. Motivated by this scenario, we study, in a simultaneous way, the changes of opinions about vaccination together with the evolution of a disease. In our model we consider a multiplex network consisting of two layers. One of the layers corresponds to a social network where people share their opinions and influence others opinions. The social model that rules the dynamic is the M-model, which takes into account two different processes that occurs in a society: persuasion and compromise. This two processes are related through a parameter r, r < 1 describes a moderate and committed society, for r > 1 the society tends to have extremist opinions, while r = 1 represents a neutral society. This social network may be of real or virtual contacts. On the other hand, the second layer corresponds to a network of physical contacts where the disease spreading is described by the SIR-Model. In this model the individuals may be in one of the following four states: Susceptible (S), Infected(I), Recovered (R) or Vaccinated (V). A Susceptible individual can: i) get vaccinated, if his opinion in the other layer is totally in favor of the vaccine, ii) get infected, with probability β if he is in contact with an infected neighbor. Those I individuals recover after a certain period tr = 6. Vaccinated individuals have an extremist positive opinion that does not change. We consider that the vaccine has a certain effectiveness ω and as a consequence vaccinated nodes can be infected with probability β(1 − ω) if they are in contact with an infected neighbor. In this case, if the infection process is successful, the new infected individual changes his opinion from extremist positive to totally against the vaccine. We find that depending on the trend in the opinion of the society, which depends on r, different behaviors in the spread of the epidemic occurs. An epidemic threshold was found, in which below β* and above ω* the diseases never becomes an epidemic, and it varies with the opinion parameter r.

Cooperative spreading processes in multiplex networks.

This study is concerned with the dynamic behaviors of epidemic spreading in multiplex networks. A model composed of two interacting complex networks is proposed to describe cooperative spreading processes, wherein the virus spreading in one layer can penetrate into the other to promote the spreading process. The global epidemic threshold of the model is smaller than the epidemic thresholds of the corresponding isolated networks. Thus, global epidemic onset arises in the interacting networks even though an epidemic onset does not arise in each isolated network. Simulations verify the analysis results and indicate that cooperative spreading processes in multiplex networks enhance the final infection fraction.

Two-stage effects of awareness cascade on epidemic spreading in multiplex networks.

Human awareness plays an important role in the spread of infectious diseases and the control of propagation patterns. The dynamic process with human awareness is called awareness cascade, during which individuals exhibit herd-like behavior because they are making decisions based on the actions of other individuals [Borge-Holthoefer et al., J. Complex Networks 1, 3 (2013)]. In this paper, to investigate the epidemic spreading with awareness cascade, we propose a local awareness controlled contagion spreading model on multiplex networks. By theoretical analysis using a microscopic Markov chain approach and numerical simulations, we find the emergence of an abrupt transition of epidemic threshold β(c) with the local awareness ratio α approximating 0.5, which induces two-stage effects on epidemic threshold and the final epidemic size. These findings indicate that the increase of α can accelerate the outbreak of epidemics. Furthermore, a simple 1D lattice model is investigated to illustrate the two-stage-like sharp transition at α(c)≈0.5. The results can give us a better understanding of why some epidemics cannot break out in reality and also provide a potential access to suppressing and controlling the awareness cascading systems.

Dynamical Interplay between Awareness and Epidemic Spreading in Multiplex Networks

We present the analysis of the interrelation between two processes accounting for the spreading of an epidemic, and the information awareness to prevent its infection, on top of multiplex networks. This scenario is representative of an epidemic process spreading on a network of persistent real contacts, and a cyclic information awareness process diffusing in the network of virtual social contacts between the same individuals. The topology corresponds to a multiplex network where two diffusive processes are interacting affecting each other. The analysis using a microscopic Markov chain approach reveals the phase diagram of the incidence of the epidemics and allows us to capture the evolution of the epidemic threshold depending on the topological structure of the multiplex and the interrelation with the awareness process. Interestingly, the critical point for the onset of the epidemics has a critical value (metacritical point) defined by the awareness dynamics and the topology of the virtual network, from which the onset increases and the epidemics incidence decreases.