Atrial fibrillation (AF) is the most common and increasingly common rhythm disorder . There is the AF classification model accepted by the European Society of Cardiology (ESC) and the European Heart Rhythm Society (EHRA) and the EHRA score (mEHRA score) for the evaluation of AF symptoms. Apart from its carbohydrate role, dietary fructose (as sucrose or high fructose corn syrup) has come to the fore in nutrition and dietetic studies whether it can be considered harmful or even a toxin by nature. High fructose consumption induces oxidative stress and thus causes mitochondrial damage in cells. In the light of this information, it is thought that high fructose consumption triggers mitochondrial dysfunction, which causes skeletal specific cells to enter the apoptosis pathway, by oxidative stress. Chronic fructose intake is thought to cause metabolic diseases and heart inflammation. Fructose consumption is the main dietary sugar that causes the progression of cardiovascular diseases and cardiac arrhythmias. AF patients admitted to the outpatient clinic were retrospectively scanned from the archive. Four groups of 40 patients were formed randomly according to EHRA scoring. Participants used a dietary record to provide details of nutrient consumption and all of the data from the 2 groups were compared. This study revealed statistically that a relationship between fructose consumption level and increased AF symptoms and mEHRA score (p<0,001), left atrium diameter and mEHRA score (p<0,001). Increased fructose consumption has been associated with increased AF symptoms. Excessive fructose consumption may play a role in AF pathophysiology.