Atrioventricular reentrant nodal tachycardia (AVNRT) and right atrial flutter could have a common area in the perinodal myocardium. We studied the occurrence of inducible flutter in pediatric patients with AVNRT or atrioventricular reentrant tachycardia (AVRT). Moreover we studied the effect of slowpathway ablation on flutter inducibility. We included 110 children (mean age = 12 ± 4 years), without underlying heart disease, previous ablation or history of atrial flutter or fibrillation, who were referred for supraventricular tachycardia ablation. Thirty-seven (34%) patients had AVNRT and 73 (66%) had AVRT. A standardized protocol of flutter induction was used in all these patients at baseline and after ablation. All patients with AVNRT had immediate successfull slowpathway ablation. Ninety-nine percent of patients with AVRT had immediate successfull accessory pathway ablation. Pediatric patients with AVNRT had inducible flutter in 14% of cases whereas no patient with AVRT had inducible flutter (p = 0.001). After slowpathway ablation, including a line between the low tricuspid valve and the coronary sinus ostium, no inducible flutter was found in the AVNRT and AVRT group. In the AVNRT group, patients with inducible flutter had shorter baseline AH interval (67 ± 14 vs. 88 ± 21 ms, p = 0.04), AV Wenckebach (294 ± 67 vs. 404 ± 101 ms, p = 0.02) and VA Wenckebach (298 ± 48 vs. 403 ± 98 ms, p = 0.04) compared to other AVNRT patients. These results suggest that AVNRT and right atrial flutter could share a common area located in the perinodal myocardium. However the slowpathway may not correspond to the slow conduction area during atrial flutter. Large slowpathway ablation could abolish flutter inducibilty.