Abstract
Millions worldwide suffer from OCD, which begins in adolescence and impairs daily life. Many genetic and environmental variables induce OCD. It examines heredity and environment as OCD causes. OCD is connected to serotonin and dopamine gene polymorphisms. Serotonin and glutamate transporter gene polymorphisms increase OCD risk. OCD is caused by a neurochemical imbalance that affects emotional control and behavior, according to these genetics. Childhood trauma, stress, poor life experiences, and genetics promote OCD symptoms. Neurodevelopment and the stress response system are affected by emotional, physical, or sexual abuse, rendering youngsters more prone to OCD. Early unpleasant experiences can dysregulate the stress-regulating hypothalamic-pituitary-adrenal (HPA) axis, generating anxiety and compulsivity. Life experiences and biological stresses may induce OCD, including prenatal effects, maternal stress, and infections. Genetics and environment complicate OCD's biochemical and experiential factors. This knowledge is essential for OCD therapy and prevention. Early detection of at-risk individuals, especially those with a genetic predisposition to OCD or trauma, permits concentrated therapy. CBT, especially exposure and response prevention, can cure OCD by changing maladaptive cognitive processes and behaviors. Some people's OCD symptoms improve with SSRIs' neurochemical balance. OCD treatment should incorporate genetic and environmental factors, according to this paper. Researchers and doctors can improve patient-specific treatment strategies by analyzing these complex relationships. Genetic testing, environmental assessments, and therapy tactics affect OCD treatment outcomes and quality of life. Genetic and environmental research is needed to understand OCD and develop new treatments and preventative methods.
Published Version
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