Abstract
There have been several reports on the effects of tobacco mosaic virus (TMV) infection in Nicotiana tabacum leaf tissue. TMV infection does not appear to have an exact or precise infective selectivity, with respect to particular tissues. The tissue abnormalities such as lignification, suberization, vein banding, and vein clearing, may not be direct tissue responses to TMV infection. However, it has been suggested that plasmodesmata and possibly ectodesmata, may represent the actual sites of TMV infection, especially the plasmodesmata of the basal septum.TMV infection has also been implicated in host resistance vs. the levels of phenolic-type enzymes. A rise in oxidative metabolism has been associated with TMV infection, thus causing an increase in the, activity of peroxidase, polyphenol oxidase and phenylalanine ammonia-lyase. In addition, TMV infection may cause a lowering of o-dihydric phenols such as chlorogenic acid in some tissues.
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