Abstract
<em>Helicobacter Pylori</em> affects many individuals in developing countries. Inflammation caused by <em>Helicobacter Pylori</em> differs depending on the virulence factors, density and host tissue response of the bacteria. This study is intended to investigate the relation between density of <em>Helicobacter Pylori</em> colonization in gastric mucosa in biopsy specimens and gastric mucosal inflammation severity.
Highlights
Helicobacter pylori (H. pylori/Hp) is a microaerophilic gram negative bacilli initially detected in the antral gastric mucosa by Warren and Marshall [1]
The inflammation caused by helicobacter pylori depends on the virulence factors, the density and the host tissue of the bacterium [3]
H. pylori has been shown to be associated with gastritis, peptic ulcer, gastric carcinoma and Mucosal Associated Lymphoid Tissue (MALT) lymphoma [4,5,6]
Summary
Helicobacter pylori (H. pylori/Hp) is a microaerophilic gram negative bacilli initially detected in the antral gastric mucosa by Warren and Marshall [1]. Around half of the world’s population is infected with Helicobacter pylori [2]. Helicobacter pylori is most commonly found in the antrum. H. Pylori causes mucosal inflammation with proinflammatory factors that it released and it spoils mucus layer with released enzymes. The inflammation caused by helicobacter pylori depends on the virulence factors, the density and the host tissue of the bacterium [3]. H. pylori has been shown to be associated with gastritis, peptic ulcer, gastric carcinoma and Mucosal Associated Lymphoid Tissue (MALT) lymphoma [4,5,6]
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