Abstract
Deterministic and stochastic models for polyribosome breakdown into monosomes are developed simulating two situations. 1. (i) Polyribosome degradation by ribonuclease 2. (ii) Polyribosome degradation by mechanical forces. Another model is developed simulating the modification of polyribosome distribution in acellular protein synthesis systems in the absence of significant initiation. The hypothetical mechanisms of these three phenomena are discussed in light of the comparison between the results of the simulations and experimental data available in the literature.
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