Abstract
The objective of this study is investigating the role of excessive activation PI3K/Akt/mTORC1 in diabetes mellitus type 2. It’s known that the PI3K/Akt/mTORC1 pathway is involved in the pathogenesis of cancer. Study design was carried out in accordance with the guidelines of the Helsinki Declaration that included normal patients as control (n = 15), second group included patients with diabetes mellitus type 2 (n = 22) while, third group included patients with malignancy positive mTORC1 (n = 22). 81 women were examined and enrolled in this study with age from 40 to 75-year-old. Diabetes mellitus type 2 compensation was assessed by determining the level of HbA1c by the method of ion-exchange chromatography, using the BIO-RAD D-10 analyzer, PRAS40 was determined in units depending on the amount of protein in the blood cell lysates.
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