Abstract
Sister-chromatid exchanges (SCE), phenomena of incompletely understood mechanism or significance, are often enumerated as a measure of genotoxicity, as their frequency is clearly elevated by exposure to many, if not most, chemical mutagens and carcinogens (Latt et al., 1984). However, SCE arise spontaneously, and in fairly large numbers per cell on average, in the absence of known genotoxin exposure. Statistical tests to determine whether SCE levels are elevated in a population or in an individual are based on underlying assumptions regard-
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