Abstract
Pregnancy is a complicated physiological process and the growth and development of fetus in the uterine depend on immunological modulation in maternal-fetal interface. Regulatiry T cells (Treg) usually mediate immunological tolerance and Th17 cells often act as proinflammatory cells. Treg and Th17 cells are homologous in origin and antagonistic in function. Under certain circumstances, Treg cells are able to transdifferentiate into Th17 cells and vice versa. The imbalance of Treg/Th17 may be associated with unexplained recurrent spontaneous abortion (URSA). So we here summarize the recent advances of Treg/Th17 in URSA. Key words: Regulatory T cell (Treg); Th17 cell; Recurrent spontaneous abortion; Immunological tolerance
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