Similar Papers

Paper Title
Journal
Date
Author
NR2F1-induced NR2F1-AS1 promotes esophageal squamous cell carcinoma progression via activating Hedgehog signaling pathway
Yaowen Zhang ... Pengyu Yang
Biochemical and Biophysical Research Communications | VOL. 519
Yaowen Zhang, et. al.Yaowen Zhang ... Pengyu Yang
14 Sep 2019
Data from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma
Aibin Liu ... Xi Lin
-
Aibin Liu, et. al.Aibin Liu ... Xi Lin
31 Mar 2023
Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma
Hui Gong ... Jun Li
Clinical Cancer Research | VOL. 19
Hui Gong, et. al.Hui Gong ... Jun Li
01 Mar 2013
Data from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma
Aibin Liu ... Xi Lin
-
Aibin Liu, et. al.Aibin Liu ... Xi Lin
31 Mar 2023
View more papers

More From: Neoplasma

Paper Title
Journal
Date
Author
hsa_circ_0021727 facilitates esophageal squamous cell carcinoma progression by stabilizing GBX2 mRNA through interacting with EIF4A3
Jie Lin ... Fanlin Zeng
Neoplasma | VOL. -
Jie Lin, et. al.Jie Lin ... Fanlin Zeng
01 Jan 2025
hsa_circ_0021727 facilitates esophageal squamous cell carcinoma progression by stabilizing GBX2 mRNA through interacting with EIF4A3
Jie Lin ... Fanlin Zeng
Neoplasma | VOL. 71
Jie Lin, et. al.Jie Lin ... Fanlin Zeng
01 Jan 2025
Negative prognostic significance of primary cilia and cytoplasmic β-catenin expression in non-small cell lung cancer.
Blanka Rosova ... Josef Dvorak
Neoplasma | VOL. 71
Blanka Rosova, et. al.Blanka Rosova ... Josef Dvorak
01 Jan 2025
MTHFD2 promotes breast cancer cell proliferation through IFRD1 RNA m6A methylation-mediated HDAC3/p53/mTOR pathway.
Qingqing Zhang ... Lianhong Li
Neoplasma | VOL. 71
Qingqing Zhang, et. al.Qingqing Zhang ... Lianhong Li
01 Dec 2024
View more papers

Disclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.