Abstract
The purpose of the present study was to investigate the effects of blueberry extracts (BBE) supplementation on physical performance and exercise-induced oxidative stress. 40 male Swiss mice were randomly divided into four groups (three BBE supplementation groups and one control group). The control group was gavaged with distilled water and BBE supplementation groups were gavaged with BBE (1, 2 and 4 ml respectively). After 21 days, forced swimming test was performed and the result shows that BBE supplementation could extend the swimming time to exhaustion of the mice, decreasing the blood lactic acid and MDA levels, while increasing SOD, CAT and GPX activities. It was concluded that BBE supplementation improves physical performance (swimming time) and decreases oxidative stress. Key words: Blueberries extract, physical performance, oxidative stress, mice.
Highlights
Physical exercise provides many benefits to human health, is recommended for the prevention and management of many chronic diseases and for the maintenance of optimal health (Chang et al, 2007)
The purpose of the present study was to investigate the effects of blueberry extracts (BBE) supplementation on physical performance and exercise-induced oxidative stress. 40 male Swiss mice were randomly divided into four groups
The swimming time until exhaustion of mice was considered as an index of physical performance
Summary
Physical exercise provides many benefits to human health, is recommended for the prevention and management of many chronic diseases and for the maintenance of optimal health (Chang et al, 2007). Intense physical exercise can increase oxygen consumption by up to 10- to 20-fold over resting levels to meet energy demands, and oxygen uptake in active skeletal muscle increase 100- to 200-fold (Sureda et al, 2009). Intense physical exercise is generally recognized as a factor inducing an oxidative stress. Some studies have indicated that antioxidant supplementations, such as vitamins C and E, attenuate oxidative stress and prevent strenuous exercise-induced oxidative injury in human subjects and rats (Goldfarb, 1999; Sacheck et al, 2003; Bloomer et al, 2006)
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