Abstract
Alzheimer’s disease (AD) is the most common neurodegenerative disorder with multifactorial etiology. However, there is no potential therapeutics until now. Curcumin with its ample therapeutic benefits and substantial ability to bind to amyloid beta (Aβ) plaques has been considered the best therapeutic agent. This chapter highlights manifold pathways through which curcumin counters the pathological events involved in AD. Primarily, this chapter throws light on the potential pathological amyloid beta binding efficacy, chemistry, and pathways.
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