Abstract
Vitamin B6 exists as a family of chemical species that ultimately can be converted metabolically to the coenzyme pyridoxal 5′-phosphate (PLP). PLP serves as a coenzyme in almost all aspects of human metabolism, including amino acid metabolism, one-carbon metabolism, glycogenolysis, and gluconeogenesis. Recent evidence indicates that inflammation suppresses PLP in the plasma and liver. Inflammation also increases vitamin B6 catabolism. However, the mechanistic connection between vitamin B6 insufficiency and chronic disease remains unclear.
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