Abstract
Cardiac arrest occurs in more than 2 million individuals annually worldwide. Because of their limited capacity for repair, the heart and the brain are the organs receiving the most deleterious effects from cardiac arrest. Rapid efforts to achieve return of spontaneous circulation using better quality chest compressions and early defibrillation in order to limit ischemia are critical but are only a first step in ensuring optimal neurological outcomes. Over the past decade, the use of hypothermia as a postarrest therapy has dramatically enhanced the chances for victims of cardiac arrest to achieve good neurological outcomes. However, its proper use must be simultaneously coupled with intensive goal-directed hemodynamic therapy to prevent rearrest. Despite these improvements, significant work still remains to improve outcomes.
Published Version
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