Abstract
THAT incompatibility of the Rh factor between mother and child may result in brain damage in children who survive the acute phase of the disease in the neonatal period is a well established fact. The clinical picture of this sequela is essentially one of mental deficiency, choreoathetosis and extrapyramidal spasticity.1Pathologically, there is evidence of brain damage, usually most pronounced in regions which are affected in kernicterus, i. e., the subthalamic nuclei and the lenticular bodies.2More recently, the possibility has been considered that in the absence of Rh incompatibility, erythroblastosis fetalis, jaundice and kernicterus may result occasionally from maternal isoimmunization during pregnancy by the A or B factor.3Possible neurologic sequelae of this condition have been the object of a few reports,4but as yet no pathologic description of the brain damage underlying these sequelae is available. It is the purpose of this report
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
