Weight Status In Adulthood Research Articles

Body weight in childhood, adolescence, and young adulthood in relation to later risk of disabilities and early retirement among Danish female nurses.

Obesity is now the most common health problem in the younger population in Western societies and obesity rates are higher in lower socioeconomic status (SES) groups. We investigated whether overweight in childhood, independently of overweight in adulthood, influenced adult employment status and later risk of having disabilities. Using data from the Danish Female Nurse Cohort study, we examined associations between overweight in childhood/adolescence, and young adulthood and disabilities and early retirement in later adulthood (>44 years) and whether it was influenced by menopausal age (<or ≥52 years). We analysed data from 10,363 female nurses recruited in 1999, who reported whether they, as children, were larger or of similar weight size as their peers at any age below 13 years, between 13 and 19 years, their weights, and heights at 25 years, their current work situation and whether they had had disabilities for more than 6 months. Our results showed that overweight in childhood, adolescence and young adulthood was associated with an increased risk of disabilities and early retirement. Especially childhood overweight that did not persist into adulthood was associated with an increased risk of disabilities (OR = 1.82, 95% CI = 1.26-2.63) and early retirement (OR = 2.05, 95% CI = 1.38-3.03) in the postmenopausal group. A similar increased risk for disabilities (OR = 1.76, 95% CI = 1.26-2.47) was seen for adolescent overweight that did not persist into adulthood. The results show that in a well-educated population of women, overweight in childhood and/or adolescence had adverse socioeconomic consequences for later risk of disabilities and early retirement irrespective of weight status in adulthood.

Maternal pea fiber supplementation to a high calorie diet in obese pregnancies protects male offspring from metabolic dysfunction in adulthood.

We investigated the influence of maternal yellow-pea fiber supplementation in obese pregnancies on offspring metabolic health in adulthood. Sixty newly-weaned female Sprague-Dawley rats were randomized to either a low-calorie control diet (CON) or high calorie obesogenic diet (HC) for 6-weeks. Obese animals were then fed either the HC diet alone or the HC diet supplemented with yellow-pea fiber (HC + FBR) for an additional 4-weeks prior to breeding and throughout gestation and lactation. On postnatal day (PND) 21, 1 male and 1 female offspring from each dam were weaned onto the CON diet until adulthood (PND 120) for metabolic phenotyping. Adult male, but not female, HC offspring demonstrated increased body weight and feed intake vs CON offspring, however no protection was offered by maternal FBR supplementation. HC male and female adult offspring demonstrated increased serum glucose and insulin resistance (HOMA-IR) compared with CON offspring. Maternal FBR supplementation improved glycemic control in male, but not female offspring. Compared with CON offspring, male offspring from HC dams demonstrated marked dyslipidemia (higher serum cholesterol, increased number of TG-rich lipoproteins, and smaller LDL particles) which was largely normalized in offspring from HC + FBR mothers. Male offspring born to obese mothers (HC) had higher hepatic TG, which tended to be lowered (p = 0.07) by maternal FBR supplementation.Supplementation of a maternal high calorie diet with yellow-pea fiber in prepregnancy and throughout gestation and lactation protects male offspring from metabolic dysfunction in the absence of any change in body weight status in adulthood.

Parental absence during childhood and weight status in adulthood among middle-aged community dwellers in rural Vietnam.

While adverse childhood experiences (ACEs) have been shown to be associated with adulthood obesity, less is known about their association with underweight. We examined the associations between parental absence (i.e., a major component of ACEs) and both underweight and excess weight among middle-aged rural community dwellers in Vietnam, where experiences of parental absence was not uncommon during and after the Vietnam War (1955-1975). Data came from 3000 middle-aged adults who participated in the baseline survey of Khánh Hòa Cardiovascular Study. Parental absence was defined as parental absence due to death, divorce, or out-migration. Using information on the timing of such events, we categorized participants into those who experienced parental absence before the age of 3, between the ages of 3 and 15, and those without such experiences. BMI was calculated based on measured height and weight (kg/m2 ) and categorized into three groups: underweight <18.5; normal 18.5-24.9; excess weight ≥ 25. Multinomial logistic regression was then used to investigate the association between parental absence and adult weight status. Parental absence that occurred before the age of 3 was marginally significantly associated with underweight (relative risk ratio [RRR]=1.44, 95% confidence interval [CI] 0.95, 2.20) but not with overweight/obesity. Parental divorce was associated with overweight/obesity (RRR=2.48, 95% CI 1.28, 4.81), but not parental absence due to migratory work. While previous studies in Western settings focused almost exclusively on the risk of obesity in relation to exposure to ACEs, our findings point to the potential importance of considering the risk of underweight in low- and middle-income countries.

Variation in the effects of family background and birth region on adult obesity: results of a prospective cohort study of a Great Depression-era American cohort.

BackgroundStudies have identified prenatal and early childhood conditions as important contributors to weight status in later life. To date, however, few studies have considered how weight status in adulthood is shaped by regional variation in early-life conditions, rather than the characteristics of the individual or their family. Furthermore, gender and life course differences in the salience of early life conditions to weight status remain unclear. This study investigates whether the effect of family background and birth region on adult obesity status varies by gender and over the life course.MethodsWe used data from a population-based cohort of 6,453 adults from the Health and Retirement Study, 1992–2008. Early life conditions were measured retrospectively at and after the baseline. Obesity was calculated from self-reported height and weight. Logistic models were used to estimate the net effects of family background and birth region on adulthood obesity risk after adjusting for socioeconomic factors and health behaviors measured in adulthood. Four economic and demographic data sets were used to further test the birthplace effect.ResultsAt ages 50–61, mother’s education and birth region were associated with women’s obesity risk, but not men’s. Each year’s increase in mother’s education significantly reduces the odds of being obese by 6 % (OR = 0.94; 95 % CI: 0.92, 0.97) among women, and this pattern persisted at ages 66–77. Women born in the Mountain region were least likely to be obese in late-middle age and late-life. Measures of per capita income and infant mortality rate in the birth region were also associated with the odds of obesity among women.ConclusionsWomen’s obesity status in adulthood is influenced by early childhood conditions, including regional conditions, while adulthood health risk factors may be more important for men’s obesity risk. Biological and social mechanisms may account for the gender difference.Electronic supplementary materialThe online version of this article (doi:10.1186/s12889-015-1870-7) contains supplementary material, which is available to authorized users.

Overweight in Childhood, Adolescence and Adulthood and Cardiovascular Risk in Later Life: Pooled Analysis of Three British Birth Cohorts

BackgroundOverweight and obesity in adulthood are established risk factors for adverse cardiovascular outcomes, but the contribution of overweight in childhood to later cardiovascular risk is less clear. Evidence for a direct effect of childhood overweight would highlight early life as an important target for cardiovascular disease prevention. The aim of this study was to assess whether overweight and obesity in childhood and adolescence contribute to excess cardiovascular risk in adults.Methods and findingsData from three British birth cohorts, born in 1946, 1958 and 1970, were pooled for analysis (n = 11,447). Individuals were categorised, based on body mass index (BMI), as being of normal weight or overweight/obese in childhood, adolescence and adulthood. Eight patterns of overweight were defined according to weight status at these three stages. Logistic regression models were fitted to assess the associations of patterns of overweight with self-reported type 2 diabetes, hypertension, and coronary heart disease (CHD) in adulthood (34–53 years). Compared to cohort members who were never overweight, those who were obese in adulthood had increased risk of all outcomes. For type 2 diabetes, the odds ratio was higher for obese adults who were also overweight or obese in childhood and adolescence (OR 12.6; 95% CI 6.6 to 24.0) than for those who were obese in adulthood only (OR 5.5; 95% CI 3.4 to 8.8). There was no such effect of child or adolescent overweight on hypertension. For CHD, there was weak evidence of increased risk among those with overweight in childhood. The main limitations of this study concern the use of self-reported outcomes and the generalisability of findings to contemporary child populations.ConclusionsType 2 diabetes and to a lesser extent CHD risk may be affected by overweight at all stages of life, while hypertension risk is associated more strongly with weight status in adulthood.

Parents, Television and Children's Weight Status

This study scrutinizes the long-term effects of parental television socialization activities on their children's weight status measured through body mass index (BMI-score). We address the question how parental television habits and parental television mediation in childhood relate to a person's weight status in adulthood. To analyze this issue we employed data from the 2009 Family Survey of the Dutch Population with extensive retrospective information on 1,377 Dutch respondents and their parents. Structural equation models were estimated and showed long-term effects of parental television role modeling and mediation on their children's weight status in adulthood. A parental example of frequent television viewing and social coviewing lastingly increases children's weight status through two distinct pathways: via weight status in young adulthood and via educational attainment and adult television habits. Parental instructional television mediation, however, is related to a lower-weight status of their children later in life.

Prävalenz von Übergewicht und Adipositas bei KantonsschülerInnen in St. Gallen

Overweight and obesity are the most important modulators in the development of chronic diseases. Body weight status in adulthood is often already determined in childhood and adolescence. In this study we evaluate possible threats and the prevalence of overweight and obesity among students of the Kantonsschule Burggraben in St.Gallen. Different anthropometric parameters (e.g. size, weight, circumference of waist and hip, body composition by BIA) were assessed among 552 students and different life-style factors as well as risk factors were assessed with a self-administered questionnaire. Using the IOTF-definitions between 6.12-8.18% of students were overweight and 1.08-2.23% were obese. Male students with the highest media consumption (MC) (around 4 h/d) had a higher BMI (kg/m2) than students with lower MC (around 2 h/d) or the lowest level of MC (approximately 1 h/d) (22.83 +/- 0.46 kg/m2 vs. 21.51 +/- 0.21 kg/m2 and 21.60 +/- 0.39 kg/m2; ANOVA p = 0.008). Similarly body composition was affected: students with the highest media consumption had the highest amount of body-fat mass: 12.69 +/- 0.66% vs. 11.05 +/- 0.34% and 10.69 +/- 0.62% (ANOVA p = 0.026) compared to medium and low MC, respectively. There was no correlation between the BMI, the body fat mass and the MC among female students. According to the level of activity male students with a high level showed a significant lower BMI than students with a lower level of physical activity (21.70 +/- 0.21 kg/m2 vs. 22.05 +/- 0.33 kg/m2; p = 0.016). The body fat mass was also lower for higher levels of physical activity (9.57 +/- 0.65% vs. 12.69 +/- 0.86%). Among female students the level of physical activity modulated the body fat mass (high level 20.66 +/- 0.52% vs. low level 23.53 +/- 0.58; p = 0.005) but had no effect on the BMI. Risk factors for obesity such as physical activity patterns are established in childhood and early adolescence. Preventive strategies have to be implemented as early as possible.

A 12-year follow-up study of treated obese children in Japan.

To assess tracking for body weight from childhood to adulthood in obese Japanese children who were treated for obesity, investigate the relation between the changes in body weight status and morbidity, and identify correlates of the changes in body weight status. Twelve-year retrospective cohort study. A sample of 276 subjects (age 23.9+/-4.1, 176 males and 100 females) who responded to a questionnaire mailed in 1998 to 1047 children (age 10.6+/-2.2) treated for obesity at Mie National Hospital in Japan between 1976 and 1992. Based on height and weight from medical records during childhood, the relative weight (RW; weight expressed as a percentage of the standard body weight for age, height, and sex) was calculated. Degrees of childhood obesity were based on RW: slight obesity (120% < or = RW<130%; n=17), moderate obesity (130% < or = RW <150%; n=131), and severe obesity (RW > or = 150%; n=128). Adult body mass index (BMI), which was obtained from the mailed questionnaires, was classified as normal, overweight and obese according to the WHO/NIH criteria. Body weight tracking by degree of obesity was evaluated. Subjects with severe obesity during childhood (n=128) were examined for their weight status in adulthood, prevalence of chronic diseases in adulthood, and factors such as parental obesity, dietary and exercise habits and obesity treatment during childhood. Childhood obesity tracked into adulthood obesity or overweight in 54.7% of all cases. Severely obese children (36.7%) were more likely to be obese as an adult than moderately obese children (16.8%). The prevalence of adult obesity tended to be greater in boys with moderate childhood obesity than in girls (29.7% in boys vs 14.9% in girls, P=0.058). Among the severely obese children who became normal-weight adults, the prevalence of chronic diseases was about one-fifth of those who remained obese in adulthood (P=0.041). Four factors were associated with changes in body weight status: maternal BMI at entry (P=0.044), the changes in dietary and exercise habits after treatment (P=0.014, P=0.030, respectively), and satisfaction with obesity treatment in childhood (P=0.035). Severely obese children have a higher risk of becoming obese adults even when they received obesity treatment in childhood. The risk of adulthood obesity was twice as high in moderately obese boys than in girls. On the other hand, many cases of childhood obesity can be corrected with obesity treatment, which in turn can decrease the risk for adult chronic diseases.