Toxic Chemical Exposure Research Articles

Approximating decision trees with priority hypotheses

This paper addresses the problem of creating decision trees for identifying hypotheses, also known as entities, in a setting where the cost of an action is dependent on the true hypothesis. Specifically, we consider the scenario where n hypotheses are divided into m groups based on their priority levels. Taking an action on a higher priority hypothesis incurs a higher cost. This is relevant to many real-world applications where cost-sensitive decisions need to be made. For example, in a medical diagnosis task, the goal is to take a series of actions (such as medical tests) to identify a cause. Each action in this process requires conducting a test on the patient and observing the outcome, which can take anywhere from a few minutes to several weeks depending on the test. In this case, the cost (the result of waiting for the outcome) is higher if the true hypothesis is more time-sensitive. For example, if the true hypothesis is toxic chemical exposure (as opposed to a chronic disease such as diabetes), a delay of a few minutes could significantly increase the patient's risk of mortality. We propose a group greedy algorithm to solve this problem. We demonstrate that under worst-case scenarios, our algorithm has an approximation ratio of O(mlog⁡n). Importantly, when m=1, meaning there is only one group of hypotheses, our result is consistent with the logarithmic approximation bound for the traditional optimal decision tree problem.

Effect of Intranasal Insulin on Olfactory Recovery in Patients with Anosmia .

Olfactory dysfunction, defined as either a complete or partial impairment of smell, is a common condition that may substantially affect a person’s quality of life and is frequently difficult to treat. Physical activity, heredity, nutrition, smoking, head trauma, medical treatments, and virus exposure all influence one’s capacity to smell. Hyposmia or anosmia, or loss of smell, affects 3% to 20% of the population. Intranasal insulin can improve cognitive performance, reduce food intake, and boost olfactory thresholds in healthy individuals. As people age, they are more likely to experience olfactory dysfunction, which may be affected by a variety of reasons, such as acute and chronic sino-nasal illness, upper respiratory infections, toxic chemical exposure, and degenerative diseases. About ten percent of those over 65 develop olfactory impairment, which can range from mild loss to total anosmia. Insulin resistance in the central nervous system has been linked to conditions like type II diabetes, Alzheimer’s disease, obesity, and poor odor identification. Reduced odor perception may contribute to reduced olfactory function in anosmic patients.

Xylene-Free Papanicolaou Staining.

Xylene (XL) is the most commonly used clearing agent in Papanicolaou staining. XL is hazardous and toxic chemical and prolonged exposure to XL can cause many ill-health effects. The health risk due to XL can be minimized by substituting XL with less hazardous clearing reagents such as Pine Oil (PO), Eucalyptus oil (EO), or Limonene (LM). The objective of this study was to compare the clearing ability, staining quality, preservation of morphology, physical properties, and cost of XL, PO, EO, and LM. Four smears were prepared from each of 50 serous effusions and were subjected to Papanicolaou stain. Out of four smears, one each was exposed to clearing specifically with XL (control), PO (test), EO (test), and LM (test). Test smears were compared with control for clearing, staining and morphology; graded as excellent, good or fair and further scored as 3, 2, 1, and the quality index (QI) was calculated. Statistical analysis was performed and the p value was calculated. In addition, the physical properties and cost of all the reagents were compared. QI was 0.96 for both XL and PO, whereas 0.92 and 0.54 for EO and LM, respectively. Compared to XL, the quality of staining, clearing and morphology of PO and EO were statistically not significant, whereas the difference was statistically significant with LM (p = 0.005). Physical properties such as volatility, flammability, miscibility with alcohol and DPX and the refractive indices of all the reagents were almost similar and all were recyclable. Odor was pungent for XL and EO but was pleasant for PO and LM. The cost was less for PO as compared to others. PO was a natural, less hazardous, less toxic, and economical clearing agent and can be considered as a substitute for XL.

Editorial: Challenges and emerging issues on firefighter’s toxic chemical exposure: smoke chemicals, contaminated PPE, and off-gassing

Editorial: Challenges and emerging issues on firefighter’s toxic chemical exposure: smoke chemicals, contaminated PPE, and off-gassing

Combat-related peripheral nerve injuries.

Active-duty service members (ADSM) and military Veterans represent a population with increased occupational risk for nerve injuries sustained both during training operations and wartime. Mechanisms of war-related nerve injuries have evolved over time, from the musket ball-related traumas described by S.W. Mitchell to complex blast injuries and toxic exposures sustained during Middle East conflicts in the 21st century. Commonly encountered nerve injury etiologies in this population currently include compression, direct trauma, nutritional deficits, traumatic limb amputation, toxic chemical exposures, or blast-related injuries. Expeditious identification and comprehensive, interdisciplinary treatment of combat-associated neuropathies, as well as prevention of these injuries whenever possible is critical to reduce chronic morbidity and disability for service members and to maintain a well-prepared military. However, diagnosis of a combat-associated nerve injury may be particularly challenging due to comorbid battlefield injuries or delayed presentation of neuropathy from military toxic exposures. Advances in imaging for nerve injury, including MRI and ultrasound, provide useful tools to compliment EMG in establishing a diagnosis of combat-associated nerve injury, particularly in the setting of anatomic disruption or edema. Surgical techniques can improve pain control or restoration of function. In all cases, comprehensive interdisciplinary rehabilitation provides the best framework for optimization of recovery. Further work is needed to prevent combat-associated nerve injuries and promote nerve recovery following injury.

Tracking and optimizing toxic chemical exposure pathways through food trade: A case study in SCCPs contaminated seafood in China

Abstract Food safety is related to human health and sustainable development. International food trade poses food safety risks through the collateral transport of toxic chemicals that are detrimental to human health. Domestic interprovincial trade has similar effects within countries but has not been comprehensively investigated previously. Here we assessed the effects of interprovincial trade on food safety and human dietary exposure to short-chain chlorinated paraffins (SCCPs), a group of emerging persistent toxic chemicals, in seafood across China by synthesizing data from field observation and various models. Our findings indicate that there is a higher level of SCCPs exposure risk in coastal provinces compared to inland provinces. Approximately 70.3% of human exposure to SCCPs through seafood consumption in China was embodied in the interprovincial seafood trade in 2021. Specifically, the domestic trade led to a remarkable increase in SCCPs exposure in the coastal provinces in South China, attributable to low SCCPs pollution in these provinces and imported seafood from those provinces with high SCCPs pollution. In contrast, human exposure to SCCPs decreased in those coastal provinces in East China due to importing seafood from those provinces with low SCCPs concentrations. The interprovincial seafood trade routes were optimized by linear programming to minimize human exposure to SCCPs considering both shipping cost and health risk constraints. The optimized trade routes reduced the national per capita SCCPs exposure through seafood consumption by over 12%. This study highlights the importance of interprovincial food trade in the risk assessment of toxic chemicals.

The Impact of the Aryl Hydrocarbon Receptor on Antenatal Chemical Exposure-Induced Cardiovascular-Kidney-Metabolic Programming.

Early life exposure lays the groundwork for the risk of developing cardiovascular-kidney-metabolic (CKM) syndrome in adulthood. Various environmental chemicals to which pregnant mothers are commonly exposed can disrupt fetal programming, leading to a wide range of CKM phenotypes. The aryl hydrocarbon receptor (AHR) has a key role as a ligand-activated transcription factor in sensing these environmental chemicals. Activating AHR through exposure to environmental chemicals has been documented for its adverse impacts on cardiovascular diseases, hypertension, diabetes, obesity, kidney disease, and non-alcoholic fatty liver disease, as evidenced by both epidemiological and animal studies. In this review, we compile current human evidence and findings from animal models that support the connection between antenatal chemical exposures and CKM programming, focusing particularly on AHR signaling. Additionally, we explore potential AHR modulators aimed at preventing CKM syndrome. As the pioneering review to present evidence advocating for the avoidance of toxic chemical exposure during pregnancy and deepening our understanding of AHR signaling, this has the potential to mitigate the global burden of CKM syndrome in the future.

Assessing chronic gestational exposure to environmental chemicals in pregnant women: Advancing the co-PBK model

Vulnerable populations, such as pregnant women and their fetuses, confront potential health risks due to exposure to environmental toxic compounds. Computational methods have been popular in assessing chemical exposure to populations, contrasting with traditional cohort studies for human biomonitoring. This study proposes a screening-level approach based on physiologically based kinetic (PBK) modeling to evaluate the steady-state exposure of pregnant women to environmental chemicals throughout pregnancy. To exemplify the modeling application, naphthalene was chosen. Simulation results indicated that maternal fat exhibited significant bioaccumulation potential, with the log-transformed BTF of naphthalene at 0.51 mg kg−1 per mg d−1 in the steady state. The placenta was primarily exposed to 0.83 mg/d naphthalene for a 75.2 kg pregnant woman, considering all exposure routes. In the fetal structure, single-organ fetal PBK modeling estimated a naphthalene exposure of 123.64 mg/d to the entire fetus, while multiple-organ fetal PBK modeling further revealed the bioaccumulation highest in fat tissue. The liver identified as the vital organ for metabolism, kBioT,LiverM was demonstrated with the highest sensitivity among rate constants in the maternal body. Furthermore, the first-order kinetic rate constants related to the placenta and blood were found to impact the distribution process of naphthalene in the fetus, influencing gestational exposure. In conclusion, urgent attention is needed to develop a computational biomonitoring tool for assessing toxic chemical exposure in vulnerable populations.

3D structure-functional design of a biomass-derived photocatalyst for antimicrobial efficacy and chemical degradation under ambient conditions.

Surface sterilization and hazardous chemical degradation under ambient conditions can provide significant benefits for public and environmental health. Materials with sterilization and chemical degradation capacity under sunlight can efficiently reduce infectious disease incidence rates and toxic chemical exposure. Utilizing renewable energy for sustainable sterilization and degradation is more desirable as it reduces the potential secondary contamination. Herein, we report functional structure design using lignin, a renewable carbon heterogeneous polymer, to synthesize a highly efficient and stable photocatalyst that degrades environmentally hazardous organic compounds rapidly. Through a hydrolysis reaction between Ti-OH and the hydroxyl groups of lignin, Ti-O-C and Ti-O-Ti bonds were established and a lignin based photocatalyst with a hollow sphere structure (Clignin@H-TiO2) was formed. The presence of a homozygous carbon modified TiO2 structure contributes to the enhanced photodegradation activity with solar light. The close hetero-interfacial contact between carbonized lignin and TiO2 further improves the photocatalytic efficiency by facilitating effective charge carrier separation. After synthesis optimization, the resulting Clignin@H-TiO2 photocatalyst exhibits excellent performance in the degradation of atenolol under solar light irradiation with 100% degradation within five minutes. Additionally, it efficiently removes approximately 50% of PFOA and kills about 90% of bacteria within three hours. The uniform distribution of lignin within the crosslinking structures ensures a durable carbon modified TiO2 framework, which remains stable after 10 cycles of usage. The robustness of the lignin-based photocatalyst enables incorporating the catalyst into diversified material formats and various usages. Coating of the photocatalyst onto device surfaces shows bacterial killing efficacy under sunlight. The photocatalysts based on lignin valorization present a green chemistry approach for environmental remediation and surface sterilization, which has long-term environmental protection benefits, with broad applications in toxin treatment and health protection against pathogen infection.

SuperNOVA: Semi-Parametric Identification and Estimation of Interaction and Effect Modification in Mixed Exposures using Stochastic Interventions in R.

Environmental epidemiology studies aim to understand the impact of mixed exposures on health outcomes while adjusting for covariates. However, traditional statistical methods make simplistic assumptions that may not be applicable to public policy decisions. Researchers are ultimately interested in answering causal questions, such as the impact of reducing toxic chemical exposures on adverse health outcomes like cancer. For example, in the case of PFAS, a class of chemicals measured simultaneously in blood samples, identifying the shifts that result in the greatest reduction in thyroid cancer rates can help more directly inform policy decisions on PFAS. In mixtures, nonlinear and non-additive relationships call for new statistical methods to estimate such modified exposure policies. To address these limitations, the open-source SuperNOVA package has been developed to use data-adaptive machine learning methods for identifying variable sets that have the most explanatory power on an outcome of interest. This package applies non-parametric definitions of interaction and effect modification to these variable sets in a mixed exposure, enabling researchers to explore modified treatment policies using stochastic interventions and answer causal questions. The SuperNOVA software implements the data-adaptive discovery of variable sets and estimation using optimal estimators for stochastic interventions described in our paper "Semi-Parametric Identification and Estimation of Interaction and Effect Modification in Mixed Exposures using Stochastic Interventions" (McCoy et al., 2023).

Disparities in Toxic Chemical Exposures and Associated Neurodevelopmental Outcomes: A Scoping Review and Systematic Evidence Map of the Epidemiological Literature.

Children are routinely exposed to chemicals known or suspected of harming brain development. Targeting Environmental Neuro-Development Risks (Project TENDR), an alliance of leading scientists, health professionals, and advocates, is working to protect children from these toxic chemicals and pollutants, especially the disproportionate exposures experienced by children from families with low incomes and families of color. This scoping review was initiated to map existing literature on disparities in neurodevelopmental outcomes for U.S. children from population groups who have been historically economically/socially marginalized and exposed to seven exemplar neurotoxicants: combustion-related air pollution (AP), lead (Pb), mercury (Hg), organophosphate pesticides (OPs), phthalates (Phth), polybrominated diphenyl ethers (PBDEs), and polychlorinated biphenyls (PCBs). Systematic literature searches for the seven exemplar chemicals, informed by the Population, Exposure, Comparator, Outcome (PECO) framework, were conducted through 18 November 2022, using PubMed, CINAHL Plus (EBSCO), GreenFILE (EBSCO), and Web of Science sources. We examined these studies regarding authors' conceptualization and operationalization of race, ethnicity, and other indicators of sociodemographic and socioeconomic disadvantage; whether studies presented data on exposure and outcome disparities and the patterns of those disparities; and the evidence of effect modification by or interaction with race and ethnicity. Two hundred twelve individual studies met the search criteria and were reviewed, resulting in 218 studies or investigations being included in this review. AP and Pb were the most commonly studied exposures. The most frequently identified neurodevelopmental outcomes were cognitive and behavioral/psychological. Approximately a third (74 studies) reported investigations of interactions or effect modification with 69% (51 of 74 studies) reporting the presence of interactions or effect modification. However, less than half of the studies presented data on disparities in the outcome or the exposure, and fewer conducted formal tests of heterogeneity. Ninety-two percent of the 165 articles that examined race and ethnicity did not provide an explanation of their constructs for these variables, creating an incomplete picture. As a whole, the studies we reviewed indicated a complex story about how racial and ethnic minority and low-income children may be disproportionately harmed by exposures to neurotoxicants, and this has implications for targeting interventions, policy change, and other necessary investments to eliminate these health disparities. We provide recommendations on improving environmental epidemiological studies on environmental health disparities. To achieve environmental justice and health equity, we recommend concomitant strategies to eradicate both neurotoxic chemical exposures and systems that perpetuate social inequities. https://doi.org/10.1289/EHP11750.

Antioxidant activity, total phenolic content, and total flavonoid content of potato milk – A novel functional beverage developed as an alternative to cow milk

The fundamental component of food functionality is antioxidant activity, which can scavenge and neutralize free radicals released by the body's biochemical system in response to toxic chemical exposure. The chemical composition of potato milk include 1 ± 0.087% total sugar, 0.3 ± 0.001% fat, 2.2 ± 0.0365% crude protein, 1.9 ± 0.234% dietary fiber, 0 ± 0.000% lactose, 1.031 ± 0.021 density, 92.66 ± 0.032% moisture, 7.34 ± 0.002% total milk solid, 7.04 ± 0.013% solid non fat. The sensory evaluation was carried out with a 7-point hedonic scale with 25 panelists, and the overall acceptability was 5.62 ± 1.54. Analysis of the antioxidant ability is vital to ascertain the functionality of the newly developed potato milk so that it can be employed as a biofunctional beverage. By using standard procedures, the following outcome was attained. The total phenolics present in the methanolic extract of potato milk were 620 ± 0.046 mM Gallic acid/ml at 500 µg/ml concentration. At 100 µg/ml, the total flavonoid content was 131.11 ± 0.046 mM Quercetin/ml, percentage inhibition in the ABTS assay was 88.6 ± 0.053%, according to DPPH activity was 86.16 ± 0.030%, the ferric-reducing antioxidant power was 205.2 ± 0.057 µM Fe (II)/ml, the CUPRAC of potato milk was found to be 203.67 ± 0.001 mM ascorbic acid/ml, the reducing power assay revealed a value of 132.55 ± 0.037 mM ascorbic acid/. At 250 µg/ml, the total antioxidant capacity was 289 ± 0.053 mM ascorbic acid/ml. Thus, the novel potato milk has significant antioxidant potential and may help to protect the body against a broad spectrum of disorders.

Beauty retailers push for ingredient transparency

A coalition of major beauty product stores has formed to collect and share chemical hazard data on ingredients used by the personal care industry. The Know Better, Do Better Collaborative so far includes the retailers Sephora, Ulta Beauty, Credo, Beautycounter, and The Honest Company, as well as the personal care chemical maker Inolex and the Environmental Defense Fund, a nonprofit. The effort is organized by Chem- FORWARD, a nonprofit that says its goal is “creating broad access to chemical hazard data, illuminating safer alternatives, and ending toxic chemical exposure.” In addition to beauty and personal care, ChemFORWARD has initiatives in packaging and electronics. The group says an analysis it conducted in 2022 found that detailed chemical hazard data are available for only about half of the ingredients used in beauty and personal care products. “These data gaps create risk for brands, retailers, and consumers,” the group says. Other transparency efforts

Current practices addressing toxic chemical exposure and other environmental health risks during prenatal care: Results and implications of a Canada-wide survey of prenatal care providers

Prospective parents are increasingly aware of potential risks to pregnancy and child health posed by exposure to toxic chemicals. Expanding scientific research demonstrates the heightened vulnerability of the prenatal period to environmental toxicants, underscoring potential benefits of exposure reduction. To inform reduction strategies, the SOCG collaborated in a national survey to examine prenatal care providers’ environmental health knowledge, concerns, practices, and views on patient education opportunities and barriers.

Beyond Neighborhood Disadvantage: Local Resources, Green Space, Pollution, and Crime as Residential Community Correlates of Cardiovascular Risk and Brain Morphology in Midlife Adults.

Residing in communities characterized by socioeconomic disadvantage confers risk of cardiometabolic diseases. Residing in disadvantaged communities may also confer the risk of neurodegenerative brain changes via cardiometabolic pathways. This study tested whether features of communities-apart from conventional socioeconomic characteristics-relate not only to cardiometabolic risk but also to relative tissue reductions in the cerebral cortex and hippocampus. Participants were 699 adults aged 30 to 54 years (340 women; 22.5% non-White) whose addresses were geocoded to compute community indicators of socioeconomic disadvantage, as well as air and toxic chemical pollutant exposures, homicide rates, concentration of employment opportunities, land use (green space), and availability of supermarkets and local resources. Participants also underwent assessments of cortical and hippocampal volumes and cardiometabolic risk factors (adiposity, blood pressure, fasting glucose, and lipids). Multilevel structural equation modeling demonstrated that cardiometabolic risk was associated with community disadvantage ( β = 0.10, 95% confidence interval [CI] = 0.01 to 0.18), as well as chemical pollution ( β = 0.11, 95% CI = 0.02 to 0.19), homicide rates ( β = 0.10, 95% CI = 0.01 to 0.18), employment opportunities ( β = -0.16, 95% CI = -0.27 to -0.04), and green space ( β = -0.12, 95% CI = -0.20 to -0.04). Moreover, cardiometabolic risk indirectly mediated the associations of several of these community features and brain tissue volumes. Some associations were nonlinear, and none were explained by participants' individual-level socioeconomic characteristics. Features of communities other than conventional indicators of socioeconomic disadvantage may represent nonredundant correlates of cardiometabolic risk and brain tissue morphology in midlife.

Development on Animal Models for Drug/Chemical Induced Liver Injury

In the human body, the largest gland is the liver and does a lot of essential work of the body. Liver damage is the cause of mortality and increasing day by day. Liver disease is caused by multiple factors, such as an autoimmune condition, toxic chemical exposure, viral infection, and dietary factors. Drug-induced liver injury (DILI) is a critical issue in drug development because DILI causes failures in clinical trials and the withdrawal of approved drugs from the market and leading to pathological changes result, including increase in SGOT, SGPT and bilirubin as well as the free radical generation. In this review, contains the animal model of hepatotoxicity with a different cause, their action mech., and procedure with dose. These models include the toxicity caused by chemical, drug, radiation, metal, diet, and high-fat this will lead to pathological changes resulting in hepatotoxicity.

Resveratrol Butyrate Ester Supplementation Blunts the Development of Offspring Hypertension in a Maternal Di-2-ethylhexyl Phthalate Exposure Rat Model.

Resveratrol (REV) is a plant polyphenol with a plethora of beneficial properties. We previously enhanced the efficacy of REV via esterification of REV with butyrate to form resveratrol butyrate ester (RBE). Compared with REV, RBE exhibits higher bioavailability and better antioxidant effects. Hypertension can originate in early life because of maternal toxic chemical exposure. This study aims to examine the effectiveness of RBE in the protection of offspring hypertension induced by maternal di-2-ethylhexylphthalate (DEHP) exposure and to explore the underlying mechanisms. DEHP (10 mg/kg/day) was used as oral gavage to pregnant rats during gestation and lactation. The control group received the vehicle. Three groups of DEHP-exposed dams received REV (6.67 mg/kg/day), or low-dose (3.33 mg/kg/day) or high-dose (6.67 mg/kg/day) RBE in drinking water during gestation and lactation. Perinatal DEHP exposure resulted in hypertension and bodyweight gain in adult male offspring, which was prevented by high-dose RBE. REV supplementation attenuated DEHP exposure-induced increases in blood pressure but not bodyweight. High-dose RBE decreased renal oxidative damage, increased plasma butyrate concentrations, and altered short chain fatty acid receptor (SCFA) expression. Low-dose RBE treatment reduced downstream mediators of the acryl hydrocarbon receptor (AHR) signaling pathway. Moreover, DEHP exposure, REV and RBE treatment differentially shaped the offspring's gut microbiota. In particular, high-dose RBE increased the abundance of the genus Duncaniella. The beneficial effects of RBE treatment were related to reducing oxidative damage, increasing plasma butyrate concentrations, downregulating SCFA receptor expression, antagonizing AHR signaling, and altering the gut microbiota. This study provides the first evidence of RBE as a novel plant polyphenol bioproduct targeting the oxidative stress and gut microbiota to protect against maternal DEHP exposure-primed offspring hypertension.

Methamphetamine Lab Explosion: A Pediatric Emergency Medicine Case.

Children exposed to the manufacture of illegal drugs are at risk for multiple medical problems. Providers need to be able to recognize and manage the complications from these exposures because early intervention can be crucial to decreasing morbidity and mortality. In this simulation case, a 3-year-old patient is brought to the emergency department (ED) after a house fire due to a methamphetamine lab explosion. The goals of this case are to provide the learners with the training and opportunity to manage a toxic chemical exposure by applying principles and methods of decontamination, and to manage an inhalational injury with rapidly progressive airway edema. These events being rare, this simulation gives learners crucial experience with a high-stakes medical condition.

Preparedness towards Chemical, Biological, Radiological, Nuclear, and Explosive (CBRNE) threats among healthcare personnel in Pasir Gudang, Johor, Malaysia

Chemical, Biological, Radiological, Nuclear, and Explosive (CBRNE) threats are one of the major concerns related to global security and threat. It is an unexceptional CBRNE crisis in the industrial area of Pasir Gudang, Johor Bahru, Malaysia, as the industrial sector keeps growing resulted to a crisis of toxic chemical exposure which affected 6000 lives in 2019. It is then had put more concern among healthcare personnel towards CBRNE disaster. It is of critical importance to assess the extent of preparedness in CBRNE hazards. Therefore, this article is primarily aimed to evaluate the level of preparedness among healthcare personnel toward CBRNE crises. 114 responses through questionnaire had been collected from healthcare personnel of four government agencies and was analyzed using SPSS Version 27. According to the results, progressive exposure to CBRNE knowledge with more than 5 years of working experience demonstrated an advanced level of preparedness towards CBRNE disaster. The impact of knowledge and resilience level proved to be strongly connected to CBRNE preparedness. Therefore, initiatives to improve CBRNE preparedness will provide endless CBRNE awareness. This study will enable the management to measure the readiness of other essential healthcare professionals or first responders while also assisting improving the quality of service among healthcare personnel towards disaster.

Viscoelastic and Viscoplastic Glucose Theory (VGT #22): Applying the Concepts of Viscoelasticity, Viscoplasticity and Perturbation Theories to Predict Cancer Risk Percentages using the overall Lifestyle Scores and Calculated Cancer Risk Change Rates with Corresponding Lifestyle Scores as the Respective Viscosity Factors along with Studying the Relationship between Cancer Risks and Diabetic Glucoses Based on the GH-Method: Math-Physical Medicine (No. 601)

Viscoelastic and Viscoplastic Glucose Theory (VGT #22): Applying the Concepts of Viscoelasticity, Viscoplasticity and Perturbation Theories to Predict Cancer Risk Percentages using the overall Lifestyle Scores and Calculated Cancer Risk Change Rates with Corresponding Lifestyle Scores as the Respective Viscosity Factors along with Studying the Relationship between Cancer Risks and Diabetic Glucoses Based on the GH-Method: Math-Physical Medicine (No. 601)