Tan EM Research Articles

AB0539 Prevalence of Primary SjÖGren's Syndrome in Byelorussian Population and Secondary SjÖGren's Syndrome Frequency in Rheumatoid Arthritis, Systemic Lupus Erythematosus and Systemic Sclerosis Patients

BackgroundThe prevalence of Sjögren's syndrome (Ss), both primary and secondary, varies from 0.1% to 0.6% in European Union countries [1]. The prevalence of Ss has been studied in Belarus neither...

AB1238 A comparison between automated and manual methods for detection antinuclear autoantibodies on human epithelial-2 cells

BackgroundA variety of antinuclear antibodies (ANAs) are found in serum samples obtained from patients with rheumatic autoimmune diseases1. Identification of the corresponding antigens and measurement of the antibody titer are...

Hepatocellular carcinoma presenting as polymyositis: a paraneoplastic syndrome

Polymyositis as a paraneoplastic syndrome associated with hepatocellular carcinoma is quite rare; only a few cases have been reported. We report a case of a 50-year-old female who presented with subacute quadriparesis, neck muscle weakness, elevated creatinine phosphokinase, a myogenic pattern on the EMG and was diagnosed as having polymyositis, a paraneoplastic syndrome associated with hepatocellular carcinoma with negative hepatic viral markers and a positive ANA. Improvement in patient symptoms and areduction in creatinine phosphokinase, which occurred after lobectomy, supports this rare association.

AUTHORS REPLY

International Journal of UrologyVolume 5, Issue 4 p. 336-336 Free Access AUTHORS REPLY Tetsuo Yamada, Tetsuo Yamada MD Department of Urology National Sagamihara Hospital Sagamihara, JapanSearch for more papers by this author Tetsuo Yamada, Tetsuo Yamada MD Department of Urology National Sagamihara Hospital Sagamihara, JapanSearch for more papers by this author First published: 02 July 2007 https://doi.org/10.1111/j.1442-2042.1998.tb00362.xAboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onFacebookTwitterLinkedInRedditWechat No abstract is available for this article. References 1 Kzol JA, Clark DC, Gittes RF, Tan EM. The natural history of interstitial cystitis. J Urol 1993; 149: 465– 469. 2 Alagiri M, Chottiner S, Ratner V, Slade D, Hanno P. Interstitial cystitis: unexplained associations with other chronic disease and pain syndromes. Urology 1993; 49: 52– 57. 3 Johansson SL, Fall M. Clinical featurs and spectrum of light microscopic changes in interstitial cystitis. J Urol. 1990; 143: 1118– 1124. 4 Holm-Bentzen M. Pathology and pathophysiology of painful bladder diseases. Urol Int 1989; 44: 327– 331. 5 Messing EM. Interstitial cystitis. In: PC Walsh, AB Retik, TA Stamey, ED Vaughan (eds) Campbell's urology. 6th ed. Philadelphia : Saunders , 1991; 985– 986. Volume5, Issue4July 1998Pages 336-336 ReferencesRelatedInformation

Does Hospitalizing Patients with Tuberculosis Ensure Compliance with Drug Regimens?

Bangladesh, the strain has rapidly spread to many parts of the world. The spread of the India strain of V. cholerae 0139 to southern China was confirmed here by both serological and molecular means. This is not unexpected, given the increasing cross-border trading in southwestern China and poor hygienic conditions made worse by the influx of millions of workers from northern China every year. The India and the Bangladesh strains of V. cholerae 0139 were distinguished with use of molecular typing and biotyping methods. The Guangdong strain described herein was identical to the India strain with respect to biotype, serotype, antibiogram, enterotoxin production, and the hybridization pattern of the ct.A cholera toxin gene. Nevertheless, the cholera toxin gene pattern in V. cholerae is known to be an unstable trait. Ribotyping with a broad-spectrum rRNA probe revealed no difference so far in hybridization pattern among all the 0139 strains (unpublished data), despite the presence of different ctxA gene patterns. It is therefore possible that all of the 0139 strains belonged to the same clonal linage, with subsequent divergence in terms of cholera toxin gene arrangement. The presence of two copies of cholera toxin gene in the 0139 strains was also documented in this case. Ou results agree with those of previous investigators who identified tan em repeats of the cholera toxi gene in V. cholerae 0139. This characteristic probably accounts or the huge quantities of toxin produced by this organism that result in infections of the same severity as V. cholerae 01.

Pharmacogenetic perspectives: genes, drugs and disease.

Pharmacogenetic perspectives: genes, drugs and disease.

Serologic studies in a family with heterozygous C2 deficiency: McCarty DJ, Tan EM, Zvaifler NJ, Koethe S, Duquesnoy RJ: Am J Med 1981; 71: 945–948

Serologic studies in a family with heterozygous C2 deficiency: McCarty DJ, Tan EM, Zvaifler NJ, Koethe S, Duquesnoy RJ: Am J Med 1981; 71: 945–948

Arthritis: A Quarter Century of Research

Journal Article Arthritis: A Quarter Century of Research Get access Arthritis: A Quarter Century of Research. Edited by Tan EM, Ziff M.Atlanta, The Arthritis Foundation, 1977, cloth, 248 pp, illus, $9 Mary L. Suddath Mary L. Suddath Search for other works by this author on: Oxford Academic Google Scholar Physical Therapy, Volume 59, Issue 3, March 1979, Page 352, https://doi.org/10.1093/ptj/59.3.352 Published: 01 March 1979