Data on the clinical outcomes of traumatically intruded, young, permanent teeth in Chinese children and adolescents are absent. The aim of this study was to examine the probability of spontaneous re-eruption of injured teeth, to investigate the incidence of pulp necrosis with infection of the root canal system and replacement root resorption and to analyse possible factors related to healing complications after injury. Clinical data from 6- to 18-year-old patients who sustained intrusive luxation from 2007 to 2016 were reviewed. Teeth were treated by awaiting re-eruption, orthodontic repositioning or surgical repositioning. The incidences of spontaneous re-eruption, pulp necrosis with infection, replacement resorption and marginal bone loss were calculated, and factors related to these complications were analysed using Cox regression and Kaplan-Meier analyses. Data from 79 teeth in 58 patients (mean age 9.19±2.34years) were examined over follow-up periods from 7 to 87months (median 18months). Of the 50 teeth awaiting re-eruption, the incidences of complete re-eruption and partial re-eruption were 40.0% and 34.0%, respectively. Teeth intruded <3mm had a higher complete re-eruption rate (57.1%) than those with a 3-7mm of intrusion (18.2%) (hazard ratio [HR]=4.15). Of the 52 teeth observed for more than 12months, pulp necrosis with infection, replacement resorption and marginal bone loss occurred in 57.4%, 15.4% and 61.5% of the teeth, respectively. Teeth with 3-7mm (60.9%, HR=2.97) or >7mm (100%, HR=6.44) of intrusion and teeth with uncomplicated crown fracture (85.7%, HR=5.19) were more likely to develop pulp necrosis with infection. Teeth that received orthodontic or surgical repositioning showed higher incidences of replacement resorption (23.1%, HR=5.72; 25.0%, HR=11.68, respectively). Spontaneous re-eruption of intruded teeth was significantly related to intrusion depth. Intrusion depth and crown fracture had strong relationships with pulp necrosis with infection, whereas the choice of treatment influenced the development of replacement resorption.