Distance Runners Research Articles

Acute Effects of Taurine Supplementation on Maximal Fat Oxidation and FATmax in Recreational Endurance Runners: A Randomized, Placebo-Controlled, Crossover, and Triple-Blinded Study.

Taurine (TAU) has been shown to improve time to exhaustion (TTE) and fat oxidation during exercise; however, no studies have examined the effect of acute TAU supplementation on maximal fat oxidation (MFO) and related intensity to MFO (FATmax). Our study aimed to investigate the effect of acute TAU supplementation on MFO, FATmax, VO2peak, and TTE. Eleven recreationally trained male endurance runners performed three incremental running tests. The first visit included a familiarization to the test, followed by two subsequent visits in which exercise was performed 90min after ingestion of either 6-g TAU or placebo (PLA) using a triple-blind randomized crossover design. There was no effect of TAU on MFO (p = .89, d = -0.07, TAU: 0.48 ± 0.22g/min; PLA: 0.49 ± 0.15g/min or FATmax (p = .26, d = -0.66; TAU: 49.17 ± 15.86 %V˙O2peak; PLA: 56.00 ± 13.27 %V˙O2peak). TTE was not significantly altered (TAU: 1,444.8 ± 88.6s; PLA: 1,447.6 ± 87.34s; p = .65, d = -0.04). TAU did not show any effect on V˙O2peak in comparison with PLA (TAU: 58.9 ± 8.4ml·kg-1·min-1; PLA: 56.5 ± 5.7ml·kg-1·min-1, p = .47, d = 0.48). However, V˙O2 TAU at most stages of exercise with large effect sizes (ηp2=.43). The acute ingestion of 6 g of TAU before exercise did not enhance MFO, FATmax, or TTE. However, it did increase the oxygen cost of running fixed intensities in recreationally trained endurance runners.

HouShiHeiSan attenuates sarcopenia in middle cerebral artery occlusion (MCAO) rats

Ethnopharmacological relevancePhysical therapy is the main clinical treatment for limb symptoms after ischemic stroke, and there is a lack of reliable drug intervention programs. HouShiHeiSan (HS)comes from “Synopsis of the Golden Chamber”, where it is recorded: “seauelae of wind stroke and heaviness of limbs”, indicating this formulae is a promising opion for clinical practice. Aim of the studyThe aim of this study is to explore the therapeutic effect of HS on sarcopenia after ischemic stroke (ISS) by using the middle cerebral artery occlusion (MCAO) rats. Materials and methodsAfter 7 days of adaptive feeding Sprague-Dawley (SD) rats were randomly divided into sham and MCAO surgery groups. After MCAO operation, the agreement of the models was evaluated with a laser speckle instrument, and then, treatment groups were administered HS and related solvent. During the 7 days treatment period, the Zea-Longa score was used to assess the neural function, the treadmill for exercise capacity and traction instrument for grip strength. Besides, the physiological electrical signal system was used to record muscular electrical signals, while the muscle thickness was measured by ultrasound. After data acquisition on the 7th day after MCAO operation, the soleus muscle was dissected, and the indexes of length, weight of whole muscle tissue and cross-sectional area of muscular cells by H&E were recorded. Subsequently, mechanistic indicators were examined. MuRF1 and MAFbx expression was detected by immunohistochemistry (IHC). Furthermore, the expression level of more related indicators of muscular differentiation and cellular proterin balance, including mTOR, p-mTOR, AKT, p-AKT, p70s6k, p-p70s6, FOXO1, p-FOXO1, MyoD1, Myostatin, MuRF1 and MAFbx, were tested via Western blot. ResultsHS improved motor performance and promoted muscle regeneration in MCAO rats. In terms of motor ability, HS mixed with alcohol significantly improved the neurological function damage, reduce the weight loss, increase the running distance per unit time and increase the grip strength. The postoperative muscle electrical signal intensity increased, and muscle thickness, weight, and length were maintained. The HS with alcohol group significantly maintained the cross-sectional size of muscle cells and reduced the number of MyoD1 and myostatin-positive cells in the muscle tissue. It simultaneously promoted the expression of p-mTOR, p-AKT, p-p70s6k, and MyoD1 to promote the synthesis of muscle proteins and inhibited the expression of p-FOXO1, myostatin, MAFbx, and MuRF1 to reduce muscle protein degradation. ConclusionHS can enhance muscle protein synthesis and decrease protein breakdown by activating the AKT/mTOR/FOXO1 pathway, thereby preserving muscle health and enhancing motor performance following stroke in rats.

Can Trabecular Bone Score Enhance Fracture Risk Assessment in Long-Distance Runners With Bone Stress Injuries?

Objective: To evaluate the trabecular bone score (TBS) Z scores in long-distance runners with bone stress injuries (BSIs) in whom the bone mineral density (BMD) Z score is more than −1.0 (Aim 1) and whether the number of runners with abnormal TBS Z scores would be higher in those with BSI in trabecular-rich sites as compared with cortical-rich sites (Aim 2). Design: Retrospective cohort study. Setting: Institutional sports medicine center. Patients: We analyzed Z scores derived from the TBS and BMD in 44 long-distance runners aged 18 to 40 years who had sustained BSIs between 2017 and 2023 and undergone a dual-energy x-ray absorptiometry scan with reported TBS measurements within 1 year before or 2 years after their injuries. Independent Variables: Bone mineral density and TBS Z scores. Main Outcome Measures: Evaluation of BMD and TBS Z scores in long-distance runners with BSIs, further stratified according to cortical-rich and trabecular-rich BSIs. Results: Bone mineral density assessments revealed an average Z score of −0.96 ± 0.99. In this cohort of 44 runners, 55% (n = 24) of runners were reported to have BMD Z score more than −1.0. Within this subgroup, 54% (n = 13) displayed abnormal TBS. Stratifying BSIs by trabecular- (n = 23) and cortical-rich (n = 19) sites, we found no significant difference in sex, mean BMD, or the number of runners with abnormal TBS between the 2 groups. Conclusions: Trabecular bone score Z scores in sports medicine may provide complementary information to BMD in long-distance runners with BSI, particularly when the BMD Z scores are more than −1.0. However, further research is needed to validate the usefulness of TBS Z scores in this context. Study Design: Cohort study. Level of Evidence: Retrospective Case Series, IV.

Erythrocyte nicotinamide adenine dinucleotide concentration is enhanced by systematic sports participation

BackgroundNicotinamide adenine dinucleotide (NAD+), nicotinamide adenine dinucleotide phosphate (NADP+), and their reduced forms (NADH and NADPH) are the vital cofactors for most cellular oxidation/reduction reactions and therefore influence most critical pathways in cellular metabolism. This study aimed to predict the trends of age-related changes in erythrocyte NAD+ and NADP+ concentrations in elite athletes compared to untrained controls and to assess whether life-long physical training stimulates favorable adaptations in erythrocyte NAD(P)+ concentrations.MethodsErythrocyte concentrations of NAD+ and NADP+ were measured in 68 elite endurance runners (20–81 years), 58 elite sprinters (21–90 years), and 62 untrained individuals (20–68 years). Linear regression analyses were performed to estimate longitudinal relationships and cross-sectional rates of change between age and erythrocyte NAD+ and NADP+ levels. One-way analysis of variance was used to determine differences between the studied groups.ResultsIn all three groups, the erythrocyte NAD+ and NADP+ concentrations significantly decreased with advancing age, suggesting gradual deterioration of NAD-related regulatory functions in older individuals. However, the concentration of erythrocyte NAD(P)+, regardless of age category, was higher in the athletic groups compared to less active controls.ConclusionsOur research shows that systematic sports participation, especially of a sprint-oriented nature, can be treated as a natural and effective strategy promoting cellular NAD(P)+ anabolism and thus cells’ energy and redox metabolism.Trial registrationThe study was retrospectively registered in the clinical trials registry on 2021-11-09 (NCT05113914).

Power or speed: Which metric is more accurate for modelling endurance running performance on track?

This study aimed to compare the accuracy of the power output, measured by a power meter, with respect to the speed, measured by an inertial measurement unit (IMU) and a global navigation satellite system (GNSS) sport watch to determine the critical power (CP) and speed (CS), work over CP (W') and CS (D'), and long-duration performance (i.e., 60 min). Fifteen highly trained athletes randomly performed seven time trials on a 400m track. The CP/CS and W'/D' were defined through the inverse of time model using the 3, 4, 5, 10, and 20 min trials. The 60 min performance was estimated through the power law model using the 1, 3, and 10 min trials and compared with the actual performance. A lower standard error of the estimate was obtained when using the power meter (CP: 2.7 [2.1-3.3] % and W': 13.8 [10.4-17.3] %) compared to the speed reported by the IMU (CS: 3.4 [2.5-4.3] %) and D': 20.7 [16.6-24.7] %) and GNSS sport watch (CS: 3.4 [2.5-4.3] % and D': 20.6 [16.7-24.7] %). A lower coefficient of variation was also observed for the power meter (4.9 [3.7-6.1] %) Regarding the speed reported by the IMU (10.9 [7.1-14.8] %) and GNSS sport watch (10.9 [7.0-14.7] %) in the 60 min performance estimation, the power meter offered lower errors than the IMU and GNSS sport watch for modelling endurance performance on the track.

Loss of atrial natriuretic peptide signaling causes insulin resistance, mitochondrial dysfunction, and low endurance capacity.

Type 2 diabetes (T2D) and obesity are strongly associated with low natriuretic peptide (NP) plasma levels and a down-regulation of NP guanylyl cyclase receptor-A (GCA) in skeletal muscle and adipose tissue. However, no study has so far provided evidence for a causal link between atrial NP (ANP)/GCA deficiency and T2D pathogenesis. Here, we show that both systemic and skeletal muscle ANP/GCA deficiencies in mice promote metabolic disturbances and prediabetes. Skeletal muscle insulin resistance is further associated with altered mitochondrial function and impaired endurance running capacity. ANP/GCA-deficient mice exhibit increased proton leak and reduced content of mitochondrial oxidative phosphorylation proteins. We further show that GCA is related to several metabolic traits in T2D and positively correlates with markers of oxidative capacity in human skeletal muscle. Together, these results indicate that ANP/GCA signaling controls muscle mitochondrial integrity and oxidative capacity in vivo and plays a causal role in the development of prediabetes.

Insight on Exercise-Induced Heart Remodeling in Different Track and Field Disciplines.

Background: The foot racing disciplines include sprints, middle distances, and long distances, which vary in terms of intensities, duration of training, and metabolic demands. The aim of our study was to evaluate the differences in morpho-functional parameters describing cardiac remodeling in a large cohort of Olympic athletes practicing the different track subspecialties. Methods: We evaluated 140 track and field (52.1% males, mean age 26.3 ± 4.3 years) Olympic athletes divided into four groups according to the distance performed: Group A (46, 32.9%): 100 and 200 mt; Group B (34, 24.3%): 400 mt; Group C (25, 17.9%): 800, 1500, and 3000 mt; Group D (35, 24.9%): 5000, 10,000 mt, and marathon distance. The athletes underwent a pre-participation screening, which included transthoracic echocardiography and exercise stress testing. Results: In Group A and in Group B, most athletes presented normal cardiac geometry (41/46, 89.1% in Group A and 31/34, 91.2% in Group B, p < 0.0001). Instead, in Groups C and D, more than half presented eccentric cardiac remodeling (13\25, 52% in Group C and 23\35, 65.7% in Group D). No significant differences were found between subspecialties in LVEF (p = 0.587), diastolic function (p = 0.431), and training hours/week (p = 0.078). Conclusions: In conclusion, the presence and extent of cardiac remodeling vary according to the distance of the discipline practiced, with the largest dimensional increase in both left and right ventricles and atria in mid- and long-distance runners and the lowest in sprinters.

The Race Within a Race: Starting Together, Finishing Apart

Every four years the world’s best athletes come together to compete in the Olympic games, electrifying audiences with incredible feats of speed, strength, endurance and skill as personal best performances and new records are set. However, the exceptional talent that underpin such performances is incomprehensible to most casual observers who often cannot appreciate how unique these athletes are. In this regard, endurance running, specifically the marathon, a 42.195 km foot race, provides one of the few occasions in sport outside of Olympic, world and national competitions, that permits sport scientists and fans alike to directly compare differences in the physiology between recreational and elite competitors. While these individuals may all cover the same distance, on the same course, on the same day – their experience and the physiological and psychological demands placed upon them are vastly different. There is, in effect, a “race within a race”. In the current review we highlight the superior physiology of the elite endurance athlete, emphasizing the gap between elite competitors and well-trained, but less genetically endowed athletes. We draw attention to a range of inconsistencies in how current sports science practices are understood, implemented, and communicated in terms of the elite and not-so-elite endurance athlete.

The Influence of Adolescent Physical Activity on Bone Mineral Density among Adult Runners.

This study aimed to determine the influence of sport type and training volume during adolescence on low bone mineral density (BMD) in long-distance runners. In total, 125 competitive long-distance runners (64 males; 21.3±4.2 years, 61 females; 21.4±3.1 years) participated in this retrospective cohort study. We collected training history data during adolescence using a questionnaire and measured BMD of the lumbar spine and whole body using dual-energy X-ray absorptiometry (DXA). Male runners with low BMD ran for more hours (p=0.02) and had a smaller proportion of multidirectional loading activity (p=0.03) in elementary school than those with normal BMD. Although female runners with low BMD performed multidirectional activity for more hours in middle school than those with normal BMD (p=0.01), running volume was similar between groups. There was an increasing trend in the total number of hours of physical activity in middle school in female runners with low BMD (p=0.05). These results suggest that avoiding specialization in long-distance running, increasing the proportion of multidirectional sports, and preventing overload during the bone growth phase may reduce the risk of low BMD among runners.

Development of an endurance training model in athletic performance: a case study on long distance running in Indonesia

This research aims to create innovative and effective endurance training materials tailored for long-distance runners aged 15-18. Utilizing the research and development (RnD) approach with the Borg and Gall model, the study incorporates feedback from expert validators and athletes along with their coaches from Sorong, West Papua. The research procedure encompasses ten stages, starting with problem identification and culminating in the implementation of the final product, involving 80 athletes. These stages include initial analysis, planning, initial product development, preliminary field testing, primary product revision, main field testing, operational product revision, operational field testing, final product revision, and dissemination and implementation. The involvement of both experts and practitioners ensures the training materials are theoretically sound and practically applicable. Data analysis was conducted using SPSS version 21, employing a normality test, a homogeneity test, and a paired-sample t-test to ensure the reliability and validity of the findings. The normality test confirmed the data followed a normal distribution, while the homogeneity test ensured the sample groups were comparable. The paired-sample t-test results indicated the newly developed book-based endurance training model significantly enhanced athletes' performance (p-value 0.000), showing a more substantial improvement in training outcomes compared to conventional methods (p-value 0.014). This improvement is attributed to the structured and comprehensive nature of the training materials, which include interactive elements and technology integration, making the training process more engaging and effective for young athletes. In conclusion, adopting interactive and technology-based training models can significantly aid in developing endurance skills in young long-distance runners, addressing specific needs and optimizing their training regimen. Keywords: Athletics, Endurance Training, Exercise Media, Physical Education

7284 Ablation of hexose-6-phosphate dehydrogenase in mice causes skeletal muscle atrophy and dramatically impairs exercise capacity

Abstract Disclosure: M. Zogg: None. A.D. Grötsch: None. J. Birk: None. P. Pelczar: None. J. Bouitbir: None. A. Odermatt: None. Hexose-6-phosphate dehydrogenase (H6PD) catalyzes the first two steps of the pentose-phosphate pathway (PPP) within the endoplasmic reticulum (ER), generating the redox equivalent NADPH. It has been shown that mice lacking H6PD develop defects mainly in skeletal muscles [1]. Nevertheless, the mechanisms underlying the myopathy in H6PD KO mice is unknown. Here, we further assessed muscle responses to H6PD deficiency and aimed to uncover the mechanisms that may cause myopathy in mice. We conducted a detailed characterization of the muscle structure and function as well as energy metabolism in oxidative and glycolytic muscles of H6PD KO mice. At the age of 15 weeks, H6PD KO mice displayed lower body weight than WT mice, despite similar food and water intake. Grip strength was significantly lower in H6PD KO than in WT mice. This was associated with decreased weight of the mixed (gastrocnemius, quadriceps) and fast fiber type rich muscles of the hind leg (extensor digitorum longus, tibialis anterior) in H6PD KO animals. Atrogin1 and Murf1 mRNA expression, markers of atrophy, increased only in the white (glycolytic) quadriceps. In this context, fast-twitch fibers in WT mice displayed higher H6pd mRNA expression than slow-twitch fibers. Electron microscopy analysis of red and white gastrocnemius muscles displayed the presence of large intramyofibrillar vacuoles, and morphologic changes of the sarcoplasmic reticulum and mitochondria in H6PD KO muscle. Metabolically, H6PD KO muscles presented an accumulation of glycogen granules and significantly decreased mRNA expression of glucose transporter Glut4, glucose-6-phosphate transporter G6pt and glycogen phosphorylase Pygm. The exercise capacity, measured by maximal running distance, decreased by more than 50% in H6PD KO mice. Upon exercise, both WT and H6PD KO animals were able to mobilize the majority of glycogen stored in their muscles. However, immediately after exercise, there was a significant increase in plasma lactate and glucose levels in H6PD KO animals. Disturbances in glucose metabolism were further supported by in vitro experiments using stable C2C12 H6PD KO myoblasts, revealing a shift in basal state mitochondrial fuel oxidation usage from glucose to fatty acid and glutamine in metabolic dependency assays. In conclusion, our findings revealed that H6PD seems to play a key role in proper function and energy metabolism of skeletal muscles.

Six-year follow-up of a world record-breaking master marathon runner.

Endurance performance declines with advancing age. Of the three main physiological factors that determine endurance running performance [maximal oxygen consumption (V̇o2max), lactate threshold, and running economy (RE)], V̇o2max appears to be most affected by age. Although endurance performance declines with age, recently, endurance performance has rapidly improved in master athletes as the number of master athletes competing in endurance events has increased. Master athletes represent an intriguing model to study healthy aging. In this case study, we reassessed the physiological profile of a 76-yr-old distance runner who broke the marathon world record for men over 70 yr of age in 2018. This runner was tested a few months before breaking the world record and retested in 2024. Between 2018 and 2024, his marathon running velocity decreased significantly. Therefore, the purpose of this case study was to determine the physiological changes that explain his performance decline. RE remained similar to 2018, and while there was not a clear breakpoint in blood lactate, he still likely runs marathons at a high percentage (∼90%) of his V̇o2max. However, V̇o2max declined by 15.1%. HRmax declined by 3.2% and maximal O2 pulse declined by 12.4%, suggesting that maximal stroke volume and/or arteriovenous O2 difference decreased. Altogether, although this marathoner continues to compete at an elite level, his performance has declined since his record-breaking marathon due to a reduction in V̇o2max. This is likely caused by reductions in maximal stroke volume and/or arteriovenous O2 difference. We speculate that these changes reflect primarily age-related processes.NEW & NOTEWORTHY We performed 6-yr follow-up testing on a world record-breaking master marathon runner. We determined that his performance declined since his record-breaking marathon in 2018 primarily due to a reduction in V̇o2max. His max heart rate (HR) changed minimally, but his peak O2 pulse decreased, suggesting that his maximal stroke volume and/or arteriovenous O2 difference decreased. These changes likely reflect primarily age-related effects in the absence of an overt pathological disease process.

Physiological parameters and training characteristics of endurance runners at Ethiopian Youth Sports Academy (2400 meters above sea level) and Guna Athletics Sport Club (3100 meters above sea level) training camps: a comparative cross-sectional study.

Endurance performance is impacted by physiological, anthropometrical, diet, genetic, psychological, and training characteristics. Altitude can affect physiological parameters (like maximum oxygen utilization, arterial oxygen saturation (SaO<inf>2</inf>), heart rate, and blood pressure) and training characteristics (duration, frequency, and training load). Thus, this study compared physiological parameters and training characteristics between endurance runners at Ethiopian Youth Sports Academy (EYSA) and Guna Athletics Sport Club (GASC) located which are located at elevation of 2400 and 3100 meters, respectively. A comparative cross-sectional study design was used from September to November 2021. Data was obtained from a total of 120 eligible participants (30 runners and 30 controls at GASC, and 30 runners and 30 controls at EYSA). Sociodemographic and training characteristics were assessed using self-administered standardized questionnaires. SaO<inf>2</inf> and heart rate were measured using finger pulse-oximetry (Nellcor, Oxim N-65; Covidien, Dublin, Ireland). Blood pressure was measured by Folee Digital Blood Pressure Monitor DX-B1 (Jiangsu Folee Medical Equipment Co., Ltd., Zhenjiang, China). The runners of GASC had significantly lower resting SaO<inf>2</inf>, SaO<inf>2</inf> immediately after maximal exertion, training (frequency and load) than runners of EYSA in both sexes. Runners of Guna Athletics Sport Club (RGASC) had significantly higher maximum oxygen utilization (VO<inf>2</inf>max) than Runners Ethiopian Youth Sports Academy (REYSA) in both sexes. Severe exercise-induced hypoxemia (EIAH) and moderate EIAH were developed by male runners of GASC and EYSA respectively. Besides, female runners GASC and EYSA developed moderate and mild EIAH, respectively. Conclusively, there were significant differences in physiological parameters and training load between RGASC and REYSA. High altitude (3100 meters) may have a greater impact on the above variables than moderate hypoxia (2400 meters above sea level). Hence, endurance runner coaches should give emphasis to these variables.

Seasonal variation of particle number concentration in a busy urban street with exposure assessment and deposition in human respiratory tract

Ultrafine particles (UFP) associated with air quality and health impacts are a major concern in growing urban regions. Concentrations of UFP (particles of size between 10 and 100 nm) and accumulation mode (Nacc) (particles of size >100 and up to 1000 nm), are analyzed over a highly polluted megacity, Delhi, in conjunction with vehicular flow density, during peak (morning, and evening) and non-peak hours. UFP contributes ≥60% to total particle concentration during autumn and monsoon. UFP concentrations are about 50,000 particles per cm3 in winter which reduces to about 25,000 particles during monsoon. Nacc are about 20,000 (winter) and 10,000 (monsoon) particles per cm3. UFP concentration and Nacc during peak hours are at least twice higher than those obtained in non-peak hours, confirming the dominant influence of emissions from vehicular exhaust in the study region. Seasonal analysis of UFP size distribution reveals that direct emissions dominate the particle concentrations during winter and autumn, whereas new particle formation mechanism contributes the highest in spring and summer. Assessment of inhalable particle number concentration and particle deposition in the human respiratory tract using Multiple Path Particle Dosimetry (MPPD) model, performed for the first time, shows that the order in which these particles deposit in the human respiratory tract is alveoli > bronchiole > bronchus. The deposition ranges between 10 and 18 million nanoparticles during different hours of the day, whereas the estimated inhalable particle concentration (IPN) varies between 0.5 and 1 billion. Results on the IPN during activities classified from light (walking), medium, heavy, very heavy to severe (long-distance running) provide insights into health effects on vulnerable populations. These quantitative results obtained over a megacity on hourly and seasonal variations of nanoparticles along with IPN and deposition rates for different activities are important, and are invaluable inputs for developing mitigation policies aimed to improve air quality and public health, both of which are major concerns in South Asia.

Peak tibial accelerations in different foot strike patterns during level running: an independent investigation in different cohorts.

Peak tibial accelerations are used to monitor impact severity during distance running and as input for bio-feedback. Here, peak tibial accelerations were compared between rearfoot and forefoot strikes. Two different studies were undertaken by independent research centres. Tibial acceleration and optical motion capture were collected in 14 rearfoot strikers who changed to a forefoot strike in the first centre. In the second centre, tibial acceleration of 14 other rearfoot strikers and nine forefoot strikers were collected and processed. In over-ground level running at a submaximal speed, the resultant peak tibial acceleration was greater in the instructed forefoot strike condition (Δ = 7.6 ± 1.3 g, mean ± standard error difference) and in the habitual forefoot strikers (Δ = 3.7 ± 1.1 g) than in the rearfoot strikers. The shank kinematics revealed a greater decrease in antero-posterior velocity following touchdown in the forefoot strike condition. The forefoot strikes experienced greater posterior tibial acceleration, which resulted in an increased resultant peak tibial acceleration that also occurred earlier than in the rearfoot strikes. No significant difference in axial peak tibial acceleration was found between these foot strike patterns. In conclusion, the foot strike pattern differently affects peak tibial accelerations in level running, which can have implications for monitoring and biofeedback applications.

Informing Extreme Heat Policy Development in Long Distance Running

Informing Extreme Heat Policy Development in Long Distance Running

Stressed Out: Recurrent Leg Pain And Fatigue In A Collegiate Male Long-distance Runner

Stressed Out: Recurrent Leg Pain And Fatigue In A Collegiate Male Long-distance Runner

Use Of Continuous Glucose Monitoring During Experimentally-induced Acute Low Energy Availability In Female Endurance Runners

Use Of Continuous Glucose Monitoring During Experimentally-induced Acute Low Energy Availability In Female Endurance Runners

Injury Experiences Of Collegiate Distance Runners And Implications For Future Injury Reporting

Injury Experiences Of Collegiate Distance Runners And Implications For Future Injury Reporting

Cardiorespiratory And Metabolic Responses To Blood-flow Restricted Running In Female Distance Runners

Cardiorespiratory And Metabolic Responses To Blood-flow Restricted Running In Female Distance Runners