The role of arthroscopy in temporomandibular disorders (TMDs) has given us an opportunity to glean new knowledge and thus develop new technology and treatments. Arthroscopy of the temporomandibular joint (TMJ) suggests that, contrary to existing thought, TMDs are not predominantly caused by an anteriorly displaced disk. Instead we find a variety of pathologic conditions: adhesions, degenerative changes, loose bodies, bruxism dents, cartilage flaps, perforations, anterior tears, and posterior tears. A posterior tear or anterior displaced disk occurs 12% of the time. To treat every patient with TMD with an anterior repositioning splint by manipulation would appear to be using the wrong method of treatment for 88% of the pathologies found. There are two types of cartilage in human joints: hyaline, which is load-bearing, and fibrocartilage, which is the cartilage lining the condylar fossa, the articular eminence, and the TMJ disk. Fibrocartilage is histologically and chemically lacking the percentage of proteoglycans (present in hyaline cartilage) to withstand compressive loading. Therefore, loading fibrocartilage causes degeneration. When fibrocartilage receives compressive loading, matrix degeneration occurs, which leads to fibrillation and cartilage death. Because the human body heals by fibrin/clot formation and cartilage has no blood supply, cartilage will not heal. Cartilage will only wear or degenerate. Cartilage wearing is not remodeling. Loaded excessively, degeneration will occur. A series of corrected tomographs and computed tomographic scans clearly demonstrate the deleterious effects of loading (see Alpern et al.1).
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