Right-sided Pleuritic Chest Pain Research Articles

Forgotten foreign bodies in bronchial tree in adult (A report of two cases and review of literature)

Forgotten foreign bodies in bronchial tree in adult (A report of two cases and review of literature)

Pulmonary amyloidosis: treatment with laser therapy and systemic steroids

A 47-year-old housewife presented with a 3-month history of right-sided pleuritic chest pain, repeated colds and chest infections, 2 stone weight loss, poor appetite and night sweats. She was a smoker of 3 cigarettes day ’ but had smoked 20-40 cigarettes dayi. Six years previously, she had been investigated for haemoptysis and had a bronchoscopy which showed bronchial mucosal inflammation but no endobronchial abnormality. In her past medical history, she had whooping cough as a child, a hysterectomy and recurrent urinary tract infections. In her family history, her mother had Crohn’s disease and her father had renal disease and died in his thirties. Physical examination was unremarkable. Chest radiograph showed right upper lobe shadowing with loss of volume and a prominent right hilum (Plate 1). A computed tomographic (CT) scan showed a soft tissue mass around the right hilum extending into the precarinal space (Plate 2). There was some calcification within the mass. Bronchoscopy was performed which showed fleshy ‘tumour’ occluding the right upper lobe bronchus and extending into the bronchus intermedius, which bled easily with biopsy. The biopsies showed a

Unilateral Hilar Mass

A 32-year-old nonsmoking woman was admitted to our hospital with progressive dyspnea on exertion and right-sided pleuritic chest pain for the past three months. The patient had been well until 2 years earlier when she developed periodic upper airway infections. She had not lost weight over the past year. On physical examination, the right basal region of the lung manifested dull percussion and at auscultation breath sounds were diminished. The central venous pressure was elevated. Cardiac examination revealed no murmur. There was no peripheral edema, clubbing, or cyanosis. Laboratory tests revealed an elevated erythrocyte sedimentation rate of 43 mm after 1 h and anemia (hemoglobin, 7.8 mmol/L). Pulmonary function tests disclosed no abnormalities.

Aortic dissection complicated by a mediastinal haematoma and haemoptysis

The patient A 76 year old hypertensive woman presented with massive haemoptysis and right-sided pleuritic chest pain. Investigations A chest radiograph and computed tomography (CT) were performed. Comment In the majority of cases of thoracic aortic dissection, the predominant presenting symptom is chest pain, occurring in over 90% ofcases. Haemoptysis is a rare but well-documented complication, occurring in 6% of cases.' It may rarely be the sole presenting feature2 and may occur in association with a mediastinal haematoma.

Progressive Dyspnea, Pleurisy, and Hemoptysis of Three Weeks' Duration in a 48-Year-Old Woman

A 48-year-old previously healthy woman presented with a 3-week history of blood-tinged sputum, right-sided pleuritic chest pain, and dyspnea. A local physician, diagnosing asthma and a cold, prescribed theophylline and an inhaled beta-agonist. The patient presented 2 weeks later with worsening dyspnea at rest. She had a 40- to 50-pack/year history of cigarette smoking and prior heavy alcohol use. A purified protein derivative test performed 1 year earlier was negative.

Rhabdomyolysis Associated With Pleural Tuberculosis

To the Editor. —Rhabdomyolysis associated with infectious diseases, although uncommon, is now being increasingly recognized.1-7We report a case of rhabdomyolysis in a patient with tuberculosis. To the best of our knowledge, this association has not been previously reported. Report of a Case. —A 27-year-old man was in good health until one month prior to admission, when he started to experience malaise, anorexia, and an 8-kg weight loss. Three weeks later he developed a cough, right-sided pleuritic chest pain, and fever associated with myalgia, muscular weakness, and emission of dark-brown urine. Family history was negative for known inherited muscle disease. He denied any use of alcohol or tobacco, drug abuse, recent trauma, or strenuous exercise. On admission his temperature was 38.8°C. A right-sided pleural effusion and generalized proximal muscle tenderness were observed. Results of laboratory studies disclosed the following values: hematocrit, 40% (0.40), and erythrocyte sedimentation rate, 74 mm/h.

Compression of Pulmonary Artery by Aortic Aneurysm

ANEURYSMS of the ascending aorta may involve the main pulmonary artery or its branches by compression or by rupture and formation of an aortopulmonary artery fistula. While the diagnosis may be suggested from the chest roentgenogram, large midline ascending aortic aneurysms may be present yet not radiologically detectable. Diagnosis often requires pulmonary arteriography with visualization of the levophase as described herein. Report of a Case A 67-year-old woman had severe rightsided pleuritic chest pain, dyspnea, and hemoptysis for three days. The patient had been receiving warfarin sodium (Coumadin) for phlebitis but was unreliable in taking her medications. Her history included cancer of the bladder with bone metastasis and rheumatic heart disease. During physical examination, the patient was an anxious, diaphoretic woman in severe respiratory distress with considerable splinting of the right thorax. Her respirations were 24/min and blood pressure was 170/80 mm Hg. Breath sounds at the right base were

Maternal Meconium Granulomatous Peritonitis

A 32-year-old woman underwent cesarean section because of fetal distress. Meconium spilled into the incision during delivery. The patient subsequently had a fever, right-sided pleuritic chest pain, a right lower lobe infiltrate, and a pleural effusion. Exploratory laparotomy disclosed intra-abdominal fibrosis with inflammatory mass formation. A biopsy specimen showed a granulomatous reaction around the bile-staining material similar to meconium. The patient was treated with prednisone, and her symptoms abated. During the next four years, episodes of fever, abdominal discomfort, and pleuritis recurred, which eventually responded to indomethacin therapy.

Pulmonary embolism without arterial hypoxemia.

Letters, Comments, and Corrections1 December 1971Pulmonary Embolism Without Arterial HypoxemiaJOHN MILLS, M.D., DENIS DREW, B.S.JOHN MILLS, M.D.Search for more papers by this author, DENIS DREW, B.S.Search for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-75-6-972_2 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptTo the editor: In the February 1971 issue Szucs and associates (1) reported that in 50 patients with angiographically proved pulmonary emboli the partial pressure of oxygen (PO2), with room air, was less than 80 mm Hg and that lung scans were abnormal in all patients tested. They concluded that the diagnosis of pulmonary embolism could be excluded if findings in either of these tests were normal.We have seen two patients with angiographically proved pulmonary emboli but normal arterial PO2:A 64-year-old woman was admitted because of progressive ankle edema, right-sided pleuritic chest pain, shortness of breath, and sweating,...