THE first record of diverticulum of the esophagus was made in 1764 by Ludlow, a surgeon in England. He reported it as a case of obstructed deglutition from a prenatal dilatation, and later similar lesions were reported by the Italian and German writers. In 1877 Zenker and Von Ziemissem made a careful study and corrected many erroneous ideas and made the proper classification of pulsion and traction diverticula. This same classification still obtains with the majority of writers. Under the pathological classification they are spoken of as simple cylindrical and sacculated dilatations. The simple cylindrical dilatations are usually the result of long-continued stenosis of the esophagus or at the cardiac end of the stomach. The dilated portion shows thickened walls, with sometimes polypoid growths which may later ulcerate. The sacculated dilatations are classified into groups of pulsion and traction diverticula: pulsion due to pressure from within; traction due to traction from without. The pulsion diverticula are more commonly found in the upper portion of the esophagus on a level with the cricoid cartilage and arising from the posterior wall, varying in size from that of a pea to a large sausage-shaped dilatation, hanging down between the esophagus and the vertebral column. When located on the posterior wall, we find that the muscle tissue is not so strong at this point. The longitudinal fibers of the esophageal muscles are inserted above the cricoid cartilage, so that, in order to reach these other fibers, they become divided to the right and left before reaching the cricoid. The area between these fibers must be filled in with others, called transverse fibers. These are thin, so that when the muscles contract to force down a bolus of food, the upper longitudinal and transverse fibers pull on this thin point or area; then with an added traumatism, due to a large mass of imperfectly masticated food, the area gives way and a dilatation is formed. While small at first, it gradually becomes larger, due to a repetition of the same force, until food accumulates in the sac, ferments, and a more rapid dilatation results. We must bear in mind that the physiology of deglutition plays a rather important part in the etiology of esophageal diverticula. The bolus of food on the upper surface of the tongue is compressed by the tongue against the soft palate, assisted by esophageal-pharyngeal muscles, and is shot rapidly down the pharynx into the upper esophagus. No doubt, there is considerable force brought against the poorly supported region. This added insult does not by any means help the strength at that point. Some writers think it is a congenital defect. Diverticula of the pulsion type are nearly always found in the cervical region, and generally on the left side. Those of small size are spherical in form, but become oblong or pear-shaped, increasing in size until sometimes they become large enough to hold eight ounces or more.