Convulsive Phase Research Articles

Effects of adenosinetriphosphate on electrical activity of the brain during the preconvulsive and convulsive phases induced by various convulsants.

ArticleEffects of Adenosinetriphosphate on Electrical Activity of the Brain During the Preconvulsive and Convulsive Phases Induced by Various ConvulsantsClara TordaClara TordaFrom the Department of Medicine (Neurology), Cornell University Medical College and New York Hospital, New York CityPublished Online:01 Jul 1954https://doi.org/10.1152/ajplegacy.1954.178.1.123MoreSectionsPDF (1 MB)Download PDF ToolsExport citationAdd to favoritesGet permissionsTrack citations ShareShare onFacebookTwitterLinkedInWeChat Previous Back to Top Next Download PDF FiguresReferencesRelatedInformation Cited ByChemical Transmission in the Central Nervous System12 April 2011 | Journal of Pharmacy and Pharmacology, Vol. 12, No. 1Soman toxicity during and after exposure to different environmental temperaturesJournal of Toxicology and Environmental Health, Vol. 26, No. 3Model of Molecular Mechanism Able to Generate a Depolarization-Hyperpolarization CycleTHE PROBLEM OF NON-CHOLINERGIC TRANSMISSION IN THE CENTRAL NERVOUS SYSTEM More from this issue > Volume 178Issue 1July 1954Pages 123-128 Copyright & PermissionsCopyright © 1954 by American Physiological Societyhttps://doi.org/10.1152/ajplegacy.1954.178.1.123PubMed13180725History Received 15 July 1953 Published online 1 July 1954 Published in print 1 July 1954 Metrics

Observations on the Electrically-Produced Epileptic Convulsion. Part II: Pupillary Phenomena in Normal and Pathological Pupils

There are three particular pupillary movements in the course of the electrically-produced epileptic fit: dilatation during the convulsion, constriction in the post-convulsive stage, and spasmodic dilatation towards the final stages of the post-convulsive stage. The pupillary dilatation in the convulsive phase is identical with that found in the spontaneous epileptic fit, where it is generally considered as an expression of sympathetic activity, either the consequence of a general (Bumke, 1914) or of a focal cortical excitation (Foerster, 1936; Penfield and Erickson, 1941), or, an opinion held more recently, due to a hypothalamic mechanism (Delay, 1946).

STUDIES ON EXPERIMENTAL HYPERTENSION

STUDIES ON EXPERIMENTAL HYPERTENSION

STUDY OF THE WATER AND MINERAL BALANCES IN EPILEPTIC CHILDREN

That there is some significant relationship between the water balance of the body and the occurrence of convulsive seizures in epilepsy has been demonstrated by the clinical observations of Fay 1 and others and by our experimental studies 2 and those of Gamble and Hamilton. 3 There is no agreement of opinion, however, regarding the underlying nature of this relationship, although several tentative explanations have been offered. Gamble and Hamilton, who were primarily concerned with the problem of the total acid-base metabolism, observed that urinary water, as well as total base and acid, was considerably increased during the convulsive phase as contrasted with the nonconvulsive phase of the disease. The increment in both base and water excretion was interpreted as being incidental to the elimination of the extra acid released from the body tissues during the convulsions. Since the extra total acid excreted was composed chiefly of chloride ion and