NEUMOCONIOSIS, and more particularly P silicosis, has been the outstanding occupational disease disability problem of American industry for many years. It was of such magnitude that, for the period 1914 to 1940, most of the effort of the Industrial Hygiene Division of the Public Health Service was devoted to pneumoconiosis research; in this effort, many cooperative studies were done with the U. S. Bureau of Mines. Although the basic research era began in 1914, it was not until 1924 that instrumentation and diagnostic procedures had developed to the point where environmental conditions could be correlated with pathologic findings. With the development of the impinger and the mobile x-ray unit the Public Health Service in 1924 embarked upon a series of epidemiologic studies that culminated around 1940.'-'' By this time, three disabling types of pneumoconiosis had been recognized-silicosis, with and without complicating tuberculosis ; anthracosilicosis ; and asbestosis. The etiology, epidemiology, pathology and methods of prevention had been well defined, and it was believed that, through the application of developed engineering and medical principles, pneumoconiosis could be prevented. The period 1935 to 1940 also was one in which industry began to apply these developed preventive techniques on a very wide scale. This research had led to certain conclusions regarding the pneumoconioses, which need not be repeated at this time; however, attention is directed to two conclusions: (1) that some nonsilica components in industrial dust, especially iron oxide, may prevcnt or modify the action of silica in the body and may alter the tendency toward complicating tuberculosis; and (2) that asbestos was the only dust other than free sitica that had been shown to cause a lung