Matter Asymmetry Research Articles

Oil price-inflation pass-through in OECD countries

Purpose This paper aims to assess the relationship between oil price and inflation in the Organization for Economic Co-operation and Development (OECD) countries. This paper contributes to knowledge in a number of ways. Design/methodology/approach First, we carry out a comparative analysis between the developed and developing countries of the OECD. Second, we check if the global financial crisis (GFC) of 2008 altered the oil price–inflation relationship. We further extend our analysis to capture asymmetries using the non-linear autoregressive distributed lag model. Lastly, we use the Campbell and Thompson (2008) forecast evaluation test to comparatively assess the predictive ability of the symmetric (restricted) and asymmetric (unrestricted) models. Findings Our results show that asymmetries matter in the oil price–inflation nexus. Also, the effect of the GFC of 2008 is stronger for the developed countries in the short run, and the developing countries in the long run. Lastly, accounting for asymmetries in oil price yields a better forecast for inflation in both groups. Originality/value The paper adds some interesting innovations to the oil price–inflation relationship in the OECD countries. It is the study with the widest scope for such country group under two classifications of developed and developing countries. It also inculcates the role of asymmetries, financial crisis, as well as the predictive ability of oil price on inflation.

Abnormal Rich Club Organization in Hemispheric White Matter Networks of ADHD

Objective: Brain network studies have revealed abnormal topology asymmetry of white matter (WM) in ADHD. Recently, rich club organization was proposed to be a key feature of brain network topology. However, abnormalities in the rich club organization of hemispheric WM networks in ADHD remain unclear. Method: Forty ADHD patients and 51 normal controls participated in this study. Structural networks were reconstructed based on diffusion tensor imaging (DTI) and analyzed with graph theory. Results: The two groups exhibited different patterns of asymmetry in connectivity measures of rich club connections. ADHD patients showed more feeder connections than normal controls. Reduced rightward asymmetry was observed in connectivity measures of local connections involving several peripheral regions of the ADHD patients. In addition, abnormal regional asymmetry scores were associated with ADHD symptoms. Conclusion: The topological changes in rich club organization provide a novel insight into the alteration of WM connections in ADHD.

Contribution of the chiral vortical effect to the evolution of the hypermagnetic field and the matter-antimatter asymmetry in the early Universe

In this paper, we study the contribution of the chiral vortical effect, in addition to that of the chiral magnetic effect, to the evolution of the hypermagnetic field and the matter-antimatter asymmetry in the symmetric phase of the early Universe in the temperature range 100GeV < T < 10TeV. We choose a fully helical Chern-Simons wave configuration for the velocity and the hypermagnetic vector potential fields. The latter makes the plasma force-free in the absence of viscosity. We show that the most pronounced effect of the chiral vorticity is the production and initial growth of the hypermagnetic field. In particular, we show that in the presence of a non-zero matter asymmetry, the hypermagnetic field can grow from zero initial value only in the presence of a non-zero vorticity field. Moreover, we show that larger initial growths not only result in larger maximum values of the hypermagnetic field, but also cause the saturation of the hypermagnetic field and the conversion of the lepton-baryon asymmetry to occur more quickly, i.e., at a higher temperature. We show that the damping of the vorticity due to the presence of viscosity, which typically occurs extremely rapidly, does not significantly affect the evolution.

White Matter Asymmetry: A Reflection of Pathology in Traumatic Brain Injury.

Comparisons of white matter (WM) fractional anisotropy (FA) values between mild traumatic brain injury (mTBI) patients and controls have revealed inconsistencies in the directions of the resulting FA changes. To address these discrepancies, we examined hemispheric FA symmetry levels across WM tracts in 150 mTBI patients relative to 96 military controls. Automated fiber quantification was used to extract 18 WM tracts with 100 FA values, which were used to compute correlation strengths between the nine bilateral tract pairs. The Fisher z-transformed Pearson's r values were entered into an analysis of covariance examining the effects of group (mTBI and controls) and age on symmetry levels within each tract pair. The mTBI group displayed lower symmetry levels in the corticospinal tract and the inferior longitudinal fasciculus. Interactions between age and group were detected in the inferior fronto-occipital (IFOF), uncinate (UF), and superior longitudinal fasciculi (SLF). A similar pattern emerged in the IFOF and the UF, revealing age-related symmetry decreases in the mTBI patients despite stable levels of symmetry across ages in controls. In contrast, although the control group's symmetry levels actually increased with age in the SLF, no age-related symmetry changes were detected across the mTBI participants. Here, we proposed WM symmetry measures as a potential means of circumventing directional inconsistencies of trauma-related FA changes, as well as capturing more within-tract and within-subject variances of diffusion tensor imaging (DTI) metrics. Further, we demonstrated the method's utility in detecting mTBI-specific effects and their associated interactions with age.

Kinetic mixing effect in noncommutative B \u2212 L gauge theory

It is well established that the SU(P)L gauge symmetry for P ≥ 3 can address the question of fermion generation number due to the anomaly cancellation, but it neither commutes nor closes algebraically with electric and baryon-minus-lepton charges. Hence, two U(1) factors that determine such charges are required, yielding a complete gauge symmetry, SU(P)L ⊗ U(1)X ⊗ U(1)N, apart from the color group. The resulting theory manifestly provides neutrino mass, dark matter, inflation, and baryon asymmetry of the universe. Furthermore, this gauge structure may present kinetic mixing effects associated to the U(1) gauge fields, which affect the electroweak precision test such as the ρ parameter and Z couplings as well as the new physics processes. We will construct the model, examine the interplay between the kinetic mixing and those due to the symmetry breaking, and obtain the physical results in detail.

P2‐373: ASYMMETRICAL THALAMIC SUBNUCLEI IN ALZHEIMER'S DISEASE ASSOCIATED WITH GREATER DISEASE SEVERITY

Asymmetrical patterns of cerebral atrophy have been observed across a wide range of neurodegenerative diseases, including Alzheimer's disease (AD). While investigations of the whole thalamus have found little evidence of asymmetry in AD, it is increasingly being recognised that different subnuclei of the thalamus have distinct function and neural connectivity. Using a novel probabilistic atlas (Iglesias et al., 2018), we investigated the clinical associations of thalamic subnuclei asymmetry in healthy controls (HC), participants with mild cognitive impairment (MCI), and with AD. The thalami of 77 participants (33 HC, 25 MCI, 19 AD) were segmented by subnuclei using FreeSurfer (Fig. 1). Asymmetry between hemispheric subnuclei volumes were calculated as a laterality index, LI = [(Left-Right)/(Left+Right)] x 100%, hence taking into account absolute subnuclei volumes. ANCOVA was conducted to compare the extent of subnuclei asymmetry between diagnostic groups, controlling for gender, age, intracranial volumes (to account for head size), and total grey matter asymmetry (to account for overall brain asymmetry). Multiple regression analysis, controlling for the same covariates, was conducted to investigate the associations of subnuclei asymmetry with cognition and neuropsychiatric symptoms. While no main effects were observed with regard to overall thalamic asymmetry, the three groups displayed significantly different extents of asymmetry in the ventrolateral thalamic subnucleus (F(2, 70=5.28, p=.007). Specifically, AD displayed greater ventrolateral asymmetry compared to HC (p=.009) and MCI (p=.040) favouring left-dominant atrophy (Fig. 2 and Fig. 3). In cognitively impaired participants (MCI and AD), but not HC, greater asymmetry of the ventrolateral subnucleus was associated with poorer RAVLT scores (p=.034), higher NPI scores (p=.046), and higher CDR (p=.007) (Fig. 4).

Quantification of pulsed saturation transfer at 1.5T and 3T.

To compare magnetization transfer (MT) and CEST effects between 1.5T and 3T in phantom and in vivo experiments. A pulsed saturation scheme using block-shaped pulses separated by gaps was used to overcome the single RF amplifier duty cycle limitations of a clinical 1.5T scanner. Modeling was performed by incorporating the extended phase graph formalism into a Bloch-McConnell simulation. Two saturation pulse types (with long and short pulses) were used. Estimated parameters for MT (the semi-solid pool fraction, M0B ; the semi-solid transverse relaxation time, T2B ) and CEST (asymmetry; areas) were compared between 1.5T and 3T in phantoms and in the healthy brain. Improved fits were shown after inclusion of extended phase graphs. Semi-solid pool fractions in phantom (for agar with ammonium chloride) were higher for short compared to long pulses at 3T (by 19% over all concentrations) and higher at 1.5T compared to 3T (by 5%) using short pulses. In the in vivo experiments, differentiation of white and gray matter was seen in the brain at both field strengths with improved white-gray matter contrast at 3T. In white matter, the mean semi-solid fractions were 18 ± 2% at 3T and 15 ± 2% at 1.5T. The CEST asymmetry in white matter was negative (-4.9 ± 0.4%) at 3T and zero (0.0 ± 0.3%) at 1.5T. The pulsed saturation method with short pulses, using the extended phase graph formalism in the Bloch McConnell simulations, led to improved model fits to the data, when compared to those without extended phase graphs.

White matter asymmetries in human situs inversus totalis.

Diffusion weighted imaging (DWI) was used to investigate white matter asymmetries in participants with situs inversus totalis (SIT) and matched controls. Regardless of visceral condition, hemispheric differences were found for the arcuate fasciculus (ARC) and the superior longitudinal fasciculus (SLF), which are involved in language and visuospatial functioning, respectively. The ARC appears lateralized to the left hemisphere, analogous to the left lateralization of functional areas associated with language. The SLF, on the other hand, is lateralized to the right, corresponding with rightward lateralization of visuospatial functioning. Interestingly, SIT participants show a significantly lower number of streamlines in the Uncinate Fasciculus (UNC). In addition, UNC volume appears associated with measures of cognitive performance, a finding in line with previously reported performance differences between SIT participants and controls.

Structural Asymmetry in the Frontal and Temporal Lobes Is Associated with PCSK6 VNTR Polymorphism.

The nodal cascade influences the development of bodily asymmetries in humans and other vertebrates. The gene PCSK6 has shown a regulatory function during left-right axis formation and is therefore thought to influence bodily left-right asymmetries. However, it is not clear if variation in this gene is also associated with structural asymmetries in the brain. We genotyped an intronic 33bp PCSK6 variable number tandem repeat (VNTR) polymorphism that has been associated with handedness in a cohort of healthy adults. We acquired T1-weighted structural MRI images of 320 participants and defined cortical surface and thickness for each HCP region. The results demonstrate a significant association between PCSK6 VNTR genotypes and gray matter asymmetry in the superior temporal sulcus, which is involved in voice perception. Heterozygous individuals who carry a short (≤ 6 repeats) and a long (≥ 9 repeats) PCSK6 VNTR allele show stronger rightward asymmetry. Further associations were evident in the dorsolateral prefrontal cortex. Here, individuals homozygous for short alleles show a more pronounced asymmetry. This shows that PCSK6, a gene that has been implicated in the ontogenesis of bodily asymmetries by regulating the nodal cascade, is also relevant for structural asymmetries in the human brain.

Minimal system including weak sphalerons for investigating the evolution of matter asymmetries and hypermagnetic fields

We study simultaneous evolution of large scale hypermagnetic fields and the asymmetries of quarks, leptons and Higgs boson in the temperature range from 10TeV to 100GeV. Above 10TeV, we identify all of the major fast interactions and use the associated conservation laws as constraints on the initial conditions at 10TeV. Below 10TeV, we identify the major processes which fall out of equilibrium or emerge as non-negligible processes and derive the relevant evolution equations. These include the Abelian anomalies which violate fermion numbers, direct and inverse Higgs decays that change the chiralities of fermions, and weak sphalerons which violate the left-handed fermion numbers. We also consider the contributions of all fermionic chemical potentials to the UY(1) Chern-Simons term which affects the evolution through the AMHD equations. Thus, we present a minimal set of self-consistent initial conditions and evolution equations which respect all constraints coming from conservation laws, fast processes and charge neutrality of the plasma. We solve the coupled evolution equations and find that initial large hypermagnetic field can produce matter asymmetries starting from zero initial value, and vice versa provided an initial seed of hypermagnetic field is present and the rate of the electron Yukawa processes is lower. We find that our model yields acceptable values for baryon asymmetry and magnetic field. However, the scale of the magnetic field obtained is much smaller than the observational data, even when the turbulence driven inverse cascade mechanism in the broken phase is taken into account.

Reduced Interhemispheric White Matter Asymmetries in Medial Temporal Lobe Epilepsy With Hippocampal Sclerosis.

Mesial temporal lobe epilepsy (MTLE), one of the most common types of refractory focal epilepsy, has shown white matter abnormalities both within and beyond the temporal lobe. In particular, the white matter abnormalities in the ipsilateral hemisphere are more obvious than those in the contralateral hemisphere in MTLE, that is, the abnormalities present asymmetrical characteristics. However, very few studies have characterized the white matter microstructure asymmetry in MTLE patients specifically. Thus, we performed diffusion tensor imaging (DTI) to investigate the white matter microstructure asymmetries of patients with MTLE with unilateral hippocampal sclerosis (MTLE-HS). We enrolled 25 MTLE-HS (left MTLE-HS group, n = 13; right MTLE-HS group, n = 12) and 26 healthy controls (HC). DTI data were analyzed by tract-based spatial statistics (TBSS) to test the hemispheric differences across the entire white matter skeleton. We also conducted a two-sample paired t-test for 21 paired region of interests (ROIs) parceled on the basis of the ICBM-DTI-81 white-matter label atlas of bilateral hemispheres to test the hemispheric differences. An asymmetry index (AI) was calculated to further quantify the differences between the left and right paired-ROIs. It was found that the asymmetries of white matter skeletons were significantly lower in the MTLE-HS groups than in the HC group. In particular, the asymmetry traits were moderately reduced in the RMTLE-HS group and obviously reduced in the LMTLE-HS group. In addition, AI was significantly different in the RMTLE-HS group from the LMTLE-HS or HC group in the limbic system and superior longitudinal fasciculus (SLF). The current study found that the interhemispheric white matter asymmetries were significantly reduced in the MTLE-HS groups than in the HC group. The interhemispheric white matter asymmetries are distinctly affected in left and right MTLE-HS groups. The differences in AI among RMTLE-HS, LMTLE-HS, and HC involved the limbic system and SLF, which may have some pragmatic implications for the diagnosis of MTLE and differentiating LMTLE-HS from RMTLE-HS.

White Matter Changes in Cervical Dystonia Relate to Clinical Effectiveness of Botulinum Toxin Treatment.

In a previous report showing white matter microstructural hemispheric asymmetries medial to the pallidum in focal dystonias, we showed preliminary evidence that this abnormality was reduced 4 weeks after botulinum toxin (BTX) injections. In the current study we report the completed treatment study in a full-size cohort of CD patients (n = 14). In addition to showing a shift toward normalization of the hemispheric asymmetry, we evaluated clinical relevance of these findings by relating white matter changes to degree of symptom improvement. We also evaluated whether the magnitude of the white matter asymmetry before treatment was related to severity, laterality, duration of dystonia, and/or number of previous BTX injections. Our results confirm the findings of our preliminary report: we observed significant fractional anisotropy (FA) changes medial to the pallidum 4 weeks after BTX in CD participants that were not observed in controls scanned at the same interval. There was a significant relationship between magnitude of hemispheric asymmetry and dystonia symptom improvement, as measured by percent reduction in dystonia scale scores. There was also a trend toward a relationship between magnitude of pre-injection white matter asymmetry and symptom severity, but not symptom laterality, disorder duration, or number of previous BTX injections. Post-hoc analyses suggested the FA changes at least partially reflected changes in pathophysiology, but a dissociation between patient perception of benefit from injections and FA changes suggested the changes did not reflect changes to the primary “driver” of the dystonia. In contrast, there were no changes or group differences in DTI diffusivity measures, suggesting the hemispheric asymmetry in CD does not reflect irreversible white matter tissue loss. These findings support the hypothesis that central nervous system white matter changes are involved in the mechanism by which BTX exerts clinical benefit.

Do T asymmetries for neutrino oscillations in uniform matter have a CP-even component?

Observables of neutrino oscillations in matter have, in general, contributions from the effective matter potential. It contaminates the CP violation asymmetry adding a fake effect that has been recently disentangled from the genuine one by their different behavior under T and CPT. Is the genuine T-odd CPT-invariant component of the CP asymmetry coincident with the T asymmetry? Contrary to CP, matter effects in uniform matter cannot induce by themselves a non-vanishing T asymmetry; however, the question of the title remained open. We demonstrate that, in the presence of genuine CP violation, there is a new non-vanishing CP-even, and so CPT-odd, component in the T asymmetry in matter, which is of odd-parity in both the phase δ of the flavor mixing and the matter parameter a. The two disentangled components, genuine AαβT;CP and fake AαβT;CPT, could be experimentally separated by the measurement of the two T asymmetries in matter (να ↔ νβ) and ( {overline{nu}}_{alpha}leftrightarrow {overline{nu}}_{beta } ). For the (νμ ↔ νβ) transitions, the energy dependence of the new AμeT;CPT component is like the matter-induced term AμeCP;CPT of the CP asymmetry which is odd under a change of the neutrino mass hierarchy. We have thus completed the physics involved in all observable asymmetries in matter by means of their disentanglement into the three independent components, genuine AαβCP;T and fake AαβCP;CPT and AαβT;CPT.

Ultraviolet completion of a composite asymmetric dark matter model with a dark photon portal

Composite asymmetric dark matter scenarios naturally explain why the dark matter mass density is comparable with the visible matter mass density. Such scenarios generically require some entropy transfer mechanism below the composite scale; otherwise, their late-time cosmology is incompatible with observations. A tiny kinetic mixing between a dark photon and the visible photon is a promising example of the low-energy portal. In this paper, we demonstrate that grand unifications in the dark and the visible sectors explain the origin of the tiny kinetic mixing. We particularly consider an ultraviolet completion of a simple composite asymmetric dark matter model, where asymmetric dark matter carries a B − L charge. In this setup, the longevity of asymmetric dark matter is explained by the B − L symmetry, while the dark matter asymmetry originates from the B−L asymmetry generated by thermal leptogenesis. In our minimal setup, the Standard Model sector and the dark sector are unified into SU(5)GUT × SU(4)DGUT gauge theories, respectively. This model generates required B − L portal operators while suppressing unwanted higher-dimensional operators that could wash out the generated B − L asymmetry.

Baryogenesis from neutron-dark matter oscillations

It was recently suggested that dark matter consists of ~GeV particles that carry baryon number and mix with the neutron. We demonstrate that this could allow for resonant dark matter-neutron oscillations in the early universe, at finite temperature, leading to low-scale baryogenesis starting from a primordial dark matter asymmetry. In this scenario, the asymmetry transfer happens around 30 MeV, just before big bang nucleosynthesis. We illustrate the idea using a model with a dark U(1)' gauge interaction, which has recently been suggested as a way of addressing the neutron lifetime anomaly. The asymmetric dark matter component of this model is both strongly self-interacting and leads to a suppression of matter density perturbations at small scales, allowing to mitigate the small-scale problems of cold dark matter cosmology. Future CMB experiments will be able to consistently probe, or firmly exclude, this scenario.

Hemispheric asymmetries in cortical gray matter microstructure identified by neurite orientation dispersion and density imaging

Histological studies have reported microstructural hemispheric asymmetries in several cortical areas of the human brain, but reliable in vivo assessment methods have been lacking so far. Here, we used neurite orientation dispersion and density imaging (NODDI) to examine microstructural asymmetries in in vivo and determine if findings are in accordance with what has been reported in histological studies. We examined intra-neurite volume fraction (INVF), neurite orientation dispersion (ODI), and isotropic volume fraction (ISO) asymmetries in two independent samples of healthy adults (n = 269 and n = 251). Over both samples, we found greater left-hemispheric INVF in early auditory, inferior parietal and temporal-parietal-occipital areas. In contrast, we found greater right-hemispheric INVF in the fusiform and inferior temporal gyrus, reflecting what has been reported in histological studies. ODI was asymmetric towards the left hemisphere in frontal areas and towards the right hemisphere in early auditory areas. ISO showed less pronounced asymmetries. There were hardly any effects of sex or handedness on microstructural asymmetry as determined by NODDI. Taken together, these findings suggest substantial microstructural asymmetries in gray matter, making NODDI a promising marker for future genetic and behavioral studies on laterality.

Tribonacci Quantum Cosmology: Optimal Non-Antipodal Spherical Codes &amp;amp; Graphs

Degrees of freedom in deep learning, quantum cosmological, information processing are shared and evolve through a self-organizing sequence of optimal , non-antipodal , spherical codes, . This Tribonacci Quantum Cosmology model invokes four codes: 1-vertex, 3-vertex (great circle equilateral triangle), 4-vertex (spherical tetrahedron) and 24-vertex (spherical snub cube). The vertices are einselected centres of coherent quantum information that maximise their minimum separation and survive environmental decoherence on a noisy horizon. Twenty-four 1-vertex codes, , self-organize into eight 3-vertex codes, , which self-organize into one 24-vertex code, , isomorphic to dimensions of 24-spacetime and 12(2) generators of SU(5). Snub cubical 24-vertex code chirality causes matter asymmetries and the corresponding graph-stress has normal and shear components relating to respective sides of Einstein’s tensor equivalence . Cosmological scale factor and Hubble parameter evolution is formalized as an Ostwald-coarsening function of time, scaled by the tribonacci constant (T≈1.839) property of the snub cube. The 24-vertex code coarsens to a broadband 4-vertex code, isomorphic to emergent 4-spacetime and antecedent structures in 24-spacetime metamorphose to familiar 4-spacetime forms. Each of the coarse code’s 4-vertices has 6-fold parallelized degrees of freedom (conserved from the 24-vertex code), , so 4-spacetime is properly denoted 4(6)-spacetime. Cosmological parameters are formalized: CMB h=H0/100=Tlog(3)/3≈0.674, Distance Ladder , , and . Due to 6-fold parallelization, the total matter density parameter is 6-fold heavier than the baryon density parameter, . A torrent of information-equivalent energy downloads from 6-fold faster 24-spacetime to 4(6)-spacetime. Consequent stress on 4(6)-spacetime causes it to resize its dynamic memory, expanding its cosmological scale. Ultimate coarsening of reality to , isomorphic to eternal time, is imminent for each observing agent in a Wheelerian participatory universe. DNA perhaps evolved from an 8 × 3-nucleotide primeval molecular code on the model’s 24 shared dimensions.

Altered frontal white matter asymmetry and its implications for cognition in schizophrenia: A tractography study.

BackgroundWhite matter (WM) alterations are well documented in schizophrenia. Abnormalities in interhemispheric fibers appear to account for altered WM asymmetry in the illness. However, the regional specificity (e.g., frontal versus occipital) of these alterations and their potential contribution to cognitive dysfunction in schizophrenia remain unknown.MethodsForty one patients with schizophrenia and 21 healthy controls (HC) underwent diffusion-weighted imaging on a 3 Tesla MRI machine. Tract-based spatial statistic (FSL) was used to assess whole brain differences in WM. Probabilistic tractography was performed in order to separately measure frontal and occipital WM tracts. Participants also completed tests of verbal memory and processing speed. Repeated measures analyses of covariance and Pearson correlation analyses were performed.ResultsA significant group x cerebral hemisphere interaction was found for fractional anisotropy (FA) (F(1,17) = 7.03; p = .017; ηp2 = 0.29) and radial diffusivity (RD) (F(1,17) = 4.84; p = .042; ηp2 = 0.22) in the frontal tract of patients versus HC. Healthy controls showed higher mean FA and lower mean RD in the left frontal tract compared to patients, who showed the opposite pattern. In patients with schizophrenia, mean FA and RD in the right frontal tract correlated with verbal memory (r = −0.68, p = .046; r = 0.77, p = .015).ConclusionsAsymmetric WM alterations were found in a frontal tract of patients with schizophrenia. Higher mean FA in the right frontal tract correlated with worse verbal memory performance, suggesting a possible contribution these brain changes to cognitive impairment in schizophrenia.

Disentangling Genuine from Matter-Induced CP Violation in Neutrino Oscillations.

We prove that, in any flavor transition, neutrino oscillation CP-violating asymmetries in matter have two disentangled components: (i)a CPT-odd T-invariant term, non-vanishing iff there are interactions with matter, and (ii)a T-odd CPT-invariant term, non-vanishing iff there is genuine CP violation. As function of the baseline, these two terms are distinct L-even and L-odd observables to separately test (i)matter effects sensitive to the neutrino hierarchy and (ii)genuine CP violation in the neutrino sector. For the golden ν_{μ}→ν_{e} channel, the different energy distributions of the two components provide a signature of their separation. At long baselines, they show oscillations in the low and medium energy regions, with zeros at different positions and peculiar behavior around the zeros. We discover a magic energy E=(0.91±0.01) GeV at L=1300 km with vanishing CPT-odd component and maximal genuine CP asymmetry proportional to sinδ, with δ the weak CP phase. For energies above 1.5GeV, the sign of the CP asymmetry discriminates the neutrino hierarchy.

Scalar dark matter, GUT baryogenesis, and radiative neutrino mass

We investigate an interesting correlation among dark matter phenomenology, neutrino mass generation and GUT baryogenesis, based on the scotogenic model. The model contains additional right-handed neutrinos $N$ and a second Higgs doublet $\Phi$, both of which are odd under an imposed $Z_2$ symmetry. The neutral component of $\Phi$, i.e. the lightest of the $Z_2$-odd particles, is the dark matter candidate. Due to a Yukawa coupling involving $\Phi$, $N$ and the Standard Model leptons, the lepton asymmetry is converted into the dark matter asymmetry so that a non-vanishing $B-L$ asymmetry can arise from $(B-L)$-conserving GUT baryogenesis, leading to a nonzero baryon asymmetry after the sphalerons decouple. On the other hand, $\Phi$ can also generate neutrino masses radiatively. In other words, the existence of $\Phi$ as the dark matter candidate resuscitates GUT baryogenesis and realizes neutrino masses.