There is a long, ongoing controversy about the potential health effects of bisphenol A in humans (Bucher et al., 2009 ). There are two, diametrically opposed views. One side argues vociferously, based on animal experiments, that bisphenol A may exert numerous adverse effects in humans, ranging from reproductive development disorders through obesity effects to cancer induction/progression. The other side argues, also based on animal experiments, that bisphenol A poses no significant health risks to humans. This stand-off is not simply about ‘scientists squabbling about who is right or wrong’, as there are similar differences in reaction between authorities, as Canada and some US states have chosen to ban use of bisphenol A in baby feeding bottles, whereas others (e.g. EU) have said there is nothing for consumers to worry about. What is not disputed is that we are all exposed daily to bisphenol A (Calafat et al., 2008 ); our exposure is mainly via bisphenol A that has leached into food from containers (Kang et al., 2006 ), as it is used in the making of (heat-resistant) polycarbonate plastics that are used widely as containers for food and beverages as well as in tin-can linings. As with all ‘environmental’ chemicals, the question is whether our exposure is sufficient to cause adverse health effects. Based on the same evidence, different scientists and different authorities appear to have deciphered a very different answer to this question (Bucher et al., 2009 ), so the noninvolved outsider has every reason to be confused.