Baldness Research Articles

Traumatic Baldness of the Tongue

Traumatic Baldness of the Tongue

History of baldness from magic to medicine

History of baldness from magic to medicine

Re: Dardour: Treatment of Male Baldness Reply

Re: Dardour: Treatment of Male Baldness Reply

Beards, baldness, and sweat secretion.

The hypothesis according to which male common baldness has developed in the human species as a compensation for the growth of a beard in order to achieve heat loss has been tested. In 100 clean-shaven men direct measurement of the area of glabrous skin on the forehead and calvaria was found to be proportional to that of the hairy skin on the lips, cheeks, chin and neck. During light hyperthermia the evaporation rate on the bald scalp was 2 to 3 times higher than on the hairy scalp. Conversely the evaporation rate was practically equal on the foreheads and chins of women and unbearded young men, while in adult clean-shaven bearded men it was 40% less on the chin than the forehead. These results support the hypothesis that male baldness is a thermoregulatory compensation for the growth of a beard in adults.

Alopecia and hirsuties. Current concepts in pathogenesis and management.

Hirsuties and androgenic alopecia are the patterns of hair growth in women which develop in a similar manner to that normally seen in men. This process is mediated by androgens. It may be due to increased hormone production or increased target organ sensitivity. The majority of patients with hirsuties may be adequately managed with a careful explanation of their condition and advice about depilatory techniques. Some will benefit from a course of systemic antiandrogen therapy, but hair growth will resume on cessation of therapy. There have been few objective studies to evaluate the benefits of antiandrogen therapy in female baldness and none with minoxidil.

Treatment of male baldness.

Indications for surgical treatment of male baldness are usually related to the capability and experience of the surgeon. Based on the technique of the one-stage vertical flap described at the VIIIth International Congress of Plastic Surgery (Montreal 1983), we propose a system for management of the different types of baldness. Flaps, bald scalp resections, and punch grafts are used, depending on the extent and the topography of the baldness.

There is Growing Evidence That Minoxidil Improves Baldness

There is Growing Evidence That Minoxidil Improves Baldness

Mini-reduction surgery for the correction of baldness.

Mini-reduction surgery for the correction of baldness.

Male Baldness

Over a period of 5 years we have performed 62 rotations of long temporo-parieto-occipital flaps in a single stage without delay for both frontal and occipital male baldness. The technique and indications are reported and discussed. The good results obtained permit us to affirm that this surgical technique is safe, simple to perform, and rapidly effective. It has clear advantages over techniques involving delay, including guaranteed survival and vitality of the rotated flap, speedier execution, and few, if not minimal, complications.

Stimulating Trials in Male Baldness

Stimulating Trials in Male Baldness

Topically applied minoxidil in baldness.

Topically applied minoxidil in baldness.

Scalp, Hair, Baldness, and Surgery

Scalp, Hair, Baldness, and Surgery

One-stage correction of male frontotemporal baldness with two temporo-occipital flaps

One-stage correction of male frontotemporal baldness with two temporo-occipital flaps

Two new methods for treating baldness

Two new methods for treating baldness

Bears, Baldness, and the Double Spirit: The Identity of Donatello's Zuccone

Bears, Baldness, and the Double Spirit: The Identity of Donatello's Zuccone

Reversal of Baldness in Patient Receiving Minoxidil for Hypertension

Reversal of Baldness in Patient Receiving Minoxidil for Hypertension

Baldness in Young Males

Baldness in Young Males

Progression of tumours and non-neoplastic disorders as manifestations of a common biological phenomenon

The thesis is presented that progression, in both neoplastic and non-neoplastic disorders, involves a common biological mechanism. This view derives from the author's unified theory of growth and age-dependent disease. Diseases that show a reproducible age-dependence, satisfying certain statistical criteria, are held to be initiated by random ‘errors’ — somatic gene mutations — in stem cells of the central system of growth-control. A specifically mutant stem cell propagates a ‘forbidden clone’ (Burnet) of descendant cells and these cells, or their secreted humoral products, attack target cells at one or more anatomical locations. In certain disorders, a single forbidden clone gives rise to the first detectable symptoms and signs but progression of the disease depends on the (random) formation of further forbidden clones that attack additional target cells. This theory is discussed with reference to the neoplastic disorders, familial intestinal polyposis and tumours of the uterine cervix; and the non-neoplastic disorders, Parkinsonism and male baldness. Related problems concerning the fatal crisis in malignant disease and the unifocal/multifocal origin of tumours are also discussed.

Rosacea of common male baldness.

Four patients are described in whom rosacea occurred on the face, and on the scalp in patches of baldness. Histopathological examination of the specimens taken from the scalp showed changes of rosacea and basophilic degeneration of collagen. In two patients direct immunofluorescence studies were performed; in one of these the presence of IgG deposits at the dermo-epidermal junction was demonstrated. These observations confirm the influence of external factors in the pathogenesis of rosacea.

Sleep characteristics and endocrine response in progeria

T ~ I E P R O O ~ R I A of Hutchinson-Gi l ford is a car ica ture of normal senescence tha t begins in the first year of life wi th fai lure to ga in weight, loss of subcutaneous fat, ba lding, a t rophy of the skin, and wizened facies and ends in dea th from atheroscterosis before age 20; menta t ion is un impai red . A recent extensive metabol ic invest igat ion of 2 pat ients ind ica ted a lack of g rowth hormone response to insul ininduced hypoglycemia and relat ive insulin resis tance? Growth hormone unresponsiveness to insul ininduced hypoglycemia has been described in ano ther report , 2 bu t the pa t i en t was la ter repor ted to have g rowth hormone response to the infusion of a rg in ine? T h e present study was under taken to extend previous endocr ine observations, to eva lua te growth hormone release as a funct ion of sleep stage, and to determine whe ther