Channel Loss Research Articles

A 32-Gb/s Dual-Mode Transceiver With One-Tap FIR and Two-Tap IIR RX Only Equalization in 65-nm CMOS Technology

This article presents an receiver (RX) only equalization (ROE) technique that eliminates feed-forward equalization (FFE) in transmitter (TX) and bandwidth improved output driver without increment of power consumption. With a help of the proposed design technique, the power consumption, circuit complexity, and design cost are improved. The proposed RX with one-tap finite-impulse response (FIR) and second-tap IIR decision feedback equalizer (DFE) removes the FFE equalization in TX by moving the sampling point of main cursor in the received data. A simpler TX architecture with nMOS only output driver (NOD) owing to the ROE facilitates a wide bandwidth and energy efficient dual-mode (differential and single-ended) operation. The proposed transceiver was fabricated in a 65-nm CMOS technology. The RX achieves bit error rate (BER) less than 10^{-12} over a 22-dB channel loss at 32 Gb/s with 0.62-pJ/bit energy efficiency, and the TX with NOD has 0.77- and 0.40-pJ/bit energy efficiency at 32 Gb/s in differential mode and single-ended mode, respectively. The occupied area of TX and RX is 0.002 and 0.024 mm <sup xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink">2</sup> , respectively.

Paradoxical hyperexcitability from NaV1.2 sodium channel loss in neocortical pyramidal cells.

SUMMARYLoss-of-function variants in the gene SCN2A, which encodes the sodium channel NaV1.2, are strongly associated with autism spectrum disorder and intellectual disability. An estimated 20%–30% of children with these variants also suffer from epilepsy, with altered neuronal activity originating in neocortex, a region where NaV1.2 channels are expressed predominantly in excitatory pyramidal cells. This is paradoxical, as sodium channel loss in excitatory cells would be expected to dampen neocortical activity rather than promote seizure. Here, we examined pyramidal neurons lacking NaV1.2 channels and found that they were intrinsically hyperexcitable, firing high-frequency bursts of action potentials (APs) despite decrements in AP size and speed. Compartmental modeling and dynamic-clamp recordings revealed that NaV1.2 loss prevented potassium channels from properly repolarizing neurons between APs, increasing overall excitability by allowing neurons to reach threshold for subsequent APs more rapidly. This cell-intrinsic mechanism may, therefore, account for why SCN2A loss-of-function can paradoxically promote seizure.

The Influence of Bulb Position on Hydraulic Performance of Submersible Tubular Pump Device

In order to study the influence of the position of the bulb on the hydraulic performance of a submersible tubular pump device, based on a large-scale pumping station, two schemes—involving a front-mounted bulb and a rear-mounted bulb, respectively—were designed. The front-mounted scheme uses the GL-2008-03 hydraulic model and its conventional guide vane, while the rear-mounted scheme uses the optimized design of a diffuser vane. The method of combining numerical simulation and experimental testing was used to analyze the differences between the external and internal characteristics of the two schemes. The results show that, under the condition of reasonable diffusion guide vane design, the efficiency under the rear-mounted scheme is higher than that under the front-mounted scheme, where the highest efficiency difference is about 1%. Although the front-mounted bulb scheme reduces the hydraulic loss of the bulb section, the placement of the bulb on the water inlet side reduces the flow conditions of the impeller. Affected by the circulation of the guide vane outlet, the hydraulic loss of the outlet channel is greater than the rear-mounted scheme. The bulb plays a rectifying function when the bulb is placed behind, which greatly eliminates the annular volume of the guide vane outlet, and the water outlet channel has a smaller hydraulic loss. In the front-mounted scheme, the water flow inside the outlet channel squeezes to the outer wall, causing higher entropy production near the outer wall area. The entropy production of the rear-mounted scheme is mainly in the bulb section and the bulb support. This research can provide reference for the design and form selection of a submersible tubular pump device, which has great engineering significance.

Voltage-gated potassium channel dysfunction in dorsal root ganglia contributes to the exaggerated exercise pressor reflex in rats with chronic heart failure.

An exaggerated exercise pressor reflex (EPR) causes excessive sympathoexcitation and exercise intolerance during physical activity in the chronic heart failure (CHF) state. Muscle afferent sensitization contributes to the genesis of the exaggerated EPR in CHF. However, the cellular mechanisms underlying muscle afferent sensitization in CHF remain unclear. Considering that voltage-gated potassium (Kv) channels critically regulate afferent neuronal excitability, we examined the potential role of Kv channels in mediating the sensitized EPR in male rats with CHF. Real-time reverse transcription-polymerase chain reaction (RT-PCR) and Western blotting experiments demonstrate that both mRNA and protein expressions of multiple Kv channel isoforms (Kv1.4, Kv3.4, Kv4.2, and Kv4.3) were downregulated in lumbar dorsal root ganglions (DRGs) of CHF rats compared with sham rats. Immunofluorescence data demonstrate significant decreased Kv channel staining in both NF200-positive and IB4-positive lumbar DRG neurons in CHF rats compared with sham rats. Data from patch-clamp experiments demonstrate that the total Kv current, especially IA, was dramatically decreased in medium-sized IB4-negative muscle afferent neurons (a subpopulation containing mostly Aδ neurons) from CHF rats compared with sham rats, indicating a potential functional loss of Kv channels in muscle afferent Aδ neurons. In in vivo experiments, adenoviral overexpression of Kv4.3 in lumbar DRGs for 1 wk attenuated the exaggerated EPR induced by muscle static contraction and the mechanoreflex by passive stretch without affecting the blunted cardiovascular response to hindlimb arterial injection of capsaicin in CHF rats. These data suggest that Kv channel dysfunction in DRGs plays a critical role in mediating the exaggerated EPR and muscle afferent sensitization in CHF.NEW & NOTEWORTHY The primary finding of this manuscript is that voltage-gated potassium (Kv) channel dysfunction in DRGs plays a critical role in mediating the exaggerated EPR and muscle afferent sensitization in chronic heart failure (CHF). We propose that manipulation of Kv channels in DRG neurons could be considered as a potential new approach to reduce the exaggerated sympathoexcitation and to improve exercise intolerance in CHF, which can ultimately facilitate an improved quality of life and reduce mortality.

Loss of SUR1 subtype KATP channels alters antinociception and locomotor activity after opioid administration

Opioid signaling can occur through several downstream mediators and influence analgesia as well as reward mechanisms in the nervous system. KATP channels are downstream targets of the μ opioid receptor and contribute to morphine-induced antinociception. The aim of the present work was to assess the role of SUR1-subtype KATP channels in antinociception and hyperlocomotion of synthetic and semi-synthetic opioids. Adult male and female mice wild-type (WT) and SUR1 deficient (KO) mice were assessed for mechanical and thermal antinociception after administration of either buprenorphine, fentanyl, or DAMGO. Potassium flux was assessed in the dorsal root ganglia and superficial dorsal horn cells in WT and KO mice. Hyperlocomotion was also assessed in WT and KO animals after buprenorphine, fentanyl, or DAMGO administration. SUR1 KO mice had attenuated mechanical antinociception after systemic administration of buprenorphine, fentanyl, and DAMGO. Potassium flux was also attenuated in the dorsal root ganglia and spinal cord dorsal horn cells after acute administration of buprenorphine and fentanyl. Hyperlocomotion after administration of morphine and buprenorphine was potentiated in SUR1 KO mice, but was not seen after administration of fentanyl or DAMGO. These results suggest SUR1-subtype KATP channels mediate the antinociceptive response of several classes of opioids (alkaloid and synthetic/semi-synthetic), but may not contribute to the “drug-seeking” behaviors of all classes of opioids.

Unsupervised water scene dehazing network using multiple scattering model.

In water scenes, where hazy images are subject to multiple scattering and where ideal data sets are difficult to collect, many dehazing methods are not as effective as they could be. Therefore, an unsupervised water scene dehazing network using atmospheric multiple scattering model is proposed. Unlike previous image dehazing methods, our method uses the unsupervised neural network and the atmospheric multiple scattering model and solves the problem of difficult acquisition of ideal datasets and the effect of multiple scattering on the image. In our method, in order to embed the atmospheric multiple scattering model into the unsupervised dehazing network, the unsupervised dehazing network uses four branches to estimate the scene radiation layer, transmission map layer, blur kernel layer and atmospheric light layer, the hazy image is then synthesized from the four output layers, minimizing the input hazy image and the output hazy image, where the output scene radiation layer is the final dehazing image. In addition, we constructed unsupervised loss functions which applicable to image dehazing by prior knowledge, i.e., color attenuation energy loss and dark channel loss. The method has a wide range of applications, with haze being thick and variable in marine, river and lake scenes, the method can be used to assist ship vision for target detection or forward road recognition in hazy conditions. Through extensive experiments on synthetic and real-world images, the proposed method is able to recover the details, structure and texture of the water image better than five advanced dehazing methods.

Entanglement-Assisted Communication Surpassing the Ultimate Classical Capacity.

Entanglement underpins a variety of quantum-enhanced communication, sensing, and computing capabilities. Entanglement-assisted communication (EACOMM) leverages entanglement preshared by communicating parties to boost the rate of classical information transmission. Pioneering theory works showed that EACOMM can enable a communication rate well beyond the ultimate classical capacity of optical communications, but an experimental demonstration of any EACOMM advantage remains elusive. In this Letter we report the implementation of EACOMM surpassing the classical capacity over lossy and noisy bosonic channels. We construct a high-efficiency entanglement source and a phase-conjugate quantum receiver to reap the benefit of preshared entanglement, despite entanglement being broken by channel loss and noise. We show that EACOMM beats the Holevo-Schumacher-Westmoreland capacity of classical communication by up to 16.3%, when both protocols are subject to the same power constraint at the transmitter. As a practical performance benchmark, we implement a classical communication protocol with the identical characteristics for the encoded signal, showing that EACOMM can reduce the bit-error rate by up to 69% over the same bosonic channel. Our work opens a route to provable quantum advantages in a wide range of quantum information processing tasks.

Heralded entanglement distribution between two absorptive quantum memories.

Owing to the inevitable loss in communication channels, the distance of entanglement distribution is limited to approximately 100kilometres on the ground1. Quantum repeaters can circumvent this problem by using quantum memory and entanglement swapping2. As the elementary link of a quantum repeater, the heralded distribution of two-party entanglement between two remote nodes has only been realized with built-in-type quantum memories3-9. These schemes suffer from the trade-off between multiplexing capacity and deterministic properties and hence hinder the development of efficient quantum repeaters. Quantum repeaters based on absorptive quantum memories can overcome such limitations because they separate the quantum memories and the quantum light sources. Here we present an experimental demonstration of heralded entanglement between absorptive quantum memories. We build two nodes separated by 3.5metres, each containing a polarization-entangled photon-pair source and a solid-state quantum memory with bandwidth up to 1gigahertz. A joint Bell-state measurement in the middle station heralds the successful distribution of maximally entangled states between the two quantum memories with a fidelity of 80.4±2.2per cent (±1 standard deviation). The quantum nodes and channels demonstrated here can serve as an elementary link of a quantum repeater. Moreover, the wideband absorptive quantum memories used in the nodes are compatible with deterministic entanglement sources and can simultaneously support multiplexing, which paves the way for the construction of practical solid-state quantum repeaters and high-speed quantum networks.

Modeling and Simulation for Performance Evaluation of Optical Quantum Channels in Quantum key Distribution Systems

In this research work, a simulator with time-domain visualizers and configurable parameters using a continuous time simulation approach with Matlab R2019a is presented for modeling and investigating the performance of optical fiber and free-space quantum channels as a part of a generic quantum key distribution system simulator. The modeled optical fiber quantum channel is characterized with a maximum allowable distance of 150 km with 0.2 dB/km at =1550nm. While, at =900nm and =830nm the attenuation values are 2 dB/km and 3 dB/km respectively. The modeled free space quantum channel is characterized at 0.1 dB/km at =860 nm with maximum allowable distance of 150 km also. The simulator was investigated in terms of the execution of the BB84 protocol based on polarizing encoding with consideration of the optical fiber and free-space quantum channel imperfections and losses by estimating the quantum bit error rate and final secure key. This work shows a general repeatable modeling process for significant performance evaluation. The most remarkable result that emerged from the simulated data generated and detected is that the modeling process provides guidance for optical quantum channels design and characterization for other quantum key distribution protocols.

MKIPS: MKI‐based protocol steganography method in SRTP

This paper presents master key identifier based protocol steganography (MKIPS), a new approach toward creating a covert channel within the Secure Real-time Transfer Protocol, also known as SRTP. This can be achieved using the ability of the sender of Voice-over-Internet Protocol packets to select a master key from a pre-shared list of available cryptographic keys. This list is handed to the SRTP sender and receiver by an external key management protocol during session initiation. In this work, by intelligent utilization of the master key identifier field in the SRTP packet creation process, a covert channel is created. The proposed covert channel can reach a relatively high transfer rate, and its capacity may vary based on the underlying SRTP channel properties. In comparison to existing data embedding methods in SRTP, MKIPS can convey a secret message without adding to the traffic overhead of the channel and packet loss in the destination. Additionally, the proposed covert channel is as robust as its underlying user datagram protocol channel.

Detector blinding attacks on counterfactual quantum key distribution

Counterfactual quantum key distribution protocols allow two sides to establish a common secret key using an insecure channel and authenticated public communication. As opposed to many other quantum key distribution protocols, part of the quantum state used to establish each bit never leaves the transmitting side, which hinders some attacks. We show how to adapt detector blinding attacks to this setting. In blinding attacks, gated avalanche photodiode detectors are disabled or forced to activate using bright light pulses. We present two attacks that use this ability to compromise the security of counterfactual quantum key distribution. The first is a general attack but technologically demanding. (The attacker must be able to reduce the channel loss by half.) The second attack could be deployed with easily accessible technology and works for implementations where single photon sources are approximated by attenuated coherent states. The attack is a combination of a photon number splitting attack and the first blinding attack which could be deployed with easily accessible technology. The proposed attacks show counterfactual quantum key distribution is vulnerable to detector blinding and that experimental implementations should include explicit countermeasures against it.

Speeding Up the Heart? Traditional and New Perspectives on HCN4 Function.

The sinoatrial node (SAN) is the primary pacemaker of the heart and is responsible for generating the intrinsic heartbeat. Within the SAN, spontaneously active pacemaker cells initiate the electrical activity that causes the contraction of all cardiomyocytes. The firing rate of pacemaker cells depends on the slow diastolic depolarization (SDD) and determines the intrinsic heart rate (HR). To adapt cardiac output to varying physical demands, HR is regulated by the autonomic nervous system (ANS). The sympathetic and parasympathetic branches of the ANS innervate the SAN and regulate the firing rate of pacemaker cells by accelerating or decelerating SDD–a process well-known as the chronotropic effect. Although this process is of fundamental physiological relevance, it is still incompletely understood how it is mediated at the subcellular level. Over the past 20 years, most of the work to resolve the underlying cellular mechanisms has made use of genetically engineered mouse models. In this review, we focus on the findings from these mouse studies regarding the cellular mechanisms involved in the generation and regulation of the heartbeat, with particular focus on the highly debated role of the hyperpolarization-activated cyclic nucleotide-gated cation channel HCN4 in mediating the chronotropic effect. By focusing on experimental data obtained in mice and humans, but not in other species, we outline how findings obtained in mice relate to human physiology and pathophysiology and provide specific information on how dysfunction or loss of HCN4 channels leads to human SAN disease.

Comparison of seven simple loss models for runoff prediction at the plot, hillslope and catchment scale in the semiarid southwestern U.S.

Infiltration excess overland flow is the dominant mechanism for runoff generation in many dryland watersheds. Event-based rainfall-runoff models therefore partition precipitation into two components: loss and excess precipitation. The latter is then transformed into a runoff hydrograph. Numerous loss models have been developed over the past century ranging from simple empirical to sophisticated physically based methods. Complex models can lead to equifinality and associated uncertainty at larger spatial scales with varying soil and cover conditions. Simple models are therefore widely used in hydrologic practice. In the absence of measured data in many arid and semiarid regions, model parameters are often estimated based on laboratory or field infiltrometer tests. Given the documented importance of spatial scale on the runoff response in dryland catchments, it is not clear how models parameterized at the point or soil column scale will perform at the hillslope or catchment scale under real-world conditions. In this study, we compared the performance of seven simple loss models with three or less parameters: the Philip, Smith-Parlange, Horton, Kostiakov, curve number (CN), initial and constant (IC) and the linear and constant (LC) models. The latter is a modification of the IC model introduced in this study. We estimated parameters at the plot scale (2.8 m2) using rainfall simulation and then tested model performance at the hillslope (1.5–3.7 ha) and catchment scale (2.4–2.8 km2) based on measured rainfall-runoff data at two sites in New Mexico and Arizona, U.S. Results show that rainfall simulation can be used successfully to parameterize loss models at the hillslope scale. At the catchment scale, most models showed positive bias, suggesting that other losses (such as channel or transmission losses) play an important role in determining the catchment runoff response. Rainfall intensity and temporal distribution were found to be crucial for accurate runoff prediction. Models that are sensitive to rainfall intensity during the entire simulation (Philip, Smith-Parlange, Horton, Kostiakov, LC) therefore performed better than those with an initial abstraction term (CN, IC). During intermittent rain, the best results were achieved by methods expressing infiltration capacity as a function of cumulative infiltration (LC, Smith-Parlange).

Sudden death and revival of Gaussian Einstein\u2013Podolsky\u2013Rosen steering in noisy channels

Einstein–Podolsky–Rosen (EPR) steering is a useful resource for secure quantum information tasks. It is crucial to investigate the effect of inevitable loss and noise in quantum channels on EPR steering. We analyze and experimentally demonstrate the influence of purity of quantum states and excess noise on Gaussian EPR steering by distributing a two-mode squeezed state through lossy and noisy channels, respectively. We show that the impurity of state never leads to sudden death of Gaussian EPR steering, but the noise in quantum channel can. Then we revive the disappeared Gaussian EPR steering by establishing a correlated noisy channel. Different from entanglement, the sudden death and revival of Gaussian EPR steering are directional. Our result confirms that EPR steering criteria proposed by Reid and I. Kogias et al. are equivalent in our case. The presented results pave way for asymmetric quantum information processing exploiting Gaussian EPR steering in noisy environment.

A 56-Gb/s PAM4 Receiver Analog Front-End With Fixed Peaking Frequency and Bandwidth in 40-nm CMOS

This paper discusses a 56-Gb/s PAM4 receiver analog-front end (AFE) implemented in TSMC 40-nm CMOS process. The system consists of a differential 100- Ω termination, a two-stage continuous-time linear equalizer (CTLE), a variable gain amplifier (VGA), and an output buffer. The source-degenerated transconductance stage and inverter-based transimpedance (TIA) with source follower structure are adopted for both CTLE and VGA. The utilization of source follower can solve the harsh DC operation problem in conventional inverter-based TIA and extend the bandwidth. By altering the source-degenerated resistors and capacitors in the two-stage CTLE, the proposed AFE can reach fixed peaking frequency with 9-dB compensation range at high frequency. Moreover, it can also achieve a fixed bandwidth at 14 GHz and 9.5-dB DC gain tuning range when altering the feedback resistors and negative capacitance compensation network (NCC) in VGA. Measurement results demonstrate that: it can compensate for 7.3-dB channel loss at 14-GHz Nyquist frequency and open the closed eye for 56-Gb/s PAM4 signal with BER <; 10 <sup xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink">-8</sup> from 2 <sup xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink">15</sup> - 1 PRBS input. The core of the AFE occupies 0.32-mm <sup xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink">2</sup> area and consumes 30-mW power from 1.1/1.2-V supply.

Wireless Loss Detection over Fairly Shared Heterogeneous Long Fat Networks

The quality of inter-network communication is often detrimentally affected by the large deployment of heterogeneous networks, including Long Fat Networks, as a result of wireless media introduction. Legacy transport protocols assume an independent wired connection to the network. When a loss occurs, the protocol considers it as a congestion loss, decreasing its throughput in order to reduce the network congestion without evaluating a possible channel failure. Distinct wireless transport protocols and their reference metrics are analyzed in order to design a mechanism that improves the Aggressive and Adaptative Transport Protocol (AATP) performance over Heterogeneous Long Fat Networks (HLFNs). In this paper, we present the Enhanced-AATP, which introduces the designed Loss Threshold Decision maker mechanism for the detection of different types of losses in the AATP operation. The degree to which the protocol can maintain throughput levels during channel losses or decrease production while congestion losses occur depends on the evolution of the smooth Jitter Ratio metric value. Moreover, the defined Weighted Fairness index enables the modification of protocol behavior and hence the prioritized fair use of the node’s resources. Different experiments are simulated over a network simulator to demonstrate the operation and performance improvement of the Enhanced-AATP. To conclude, the Enhanced-AATP performance is compared with other modern protocols.

Primary Neural Degeneration in Noise-Exposed Human Cochleas: Correlations with Outer Hair Cell Loss and Word-Discrimination Scores.

Animal studies suggest that cochlear nerve degeneration precedes sensory cell degeneration in both noise-induced hearing loss (NIHL) and age-related hearing loss (ARHL), producing a hearing impairment that is not reflected in audiometric thresholds. Here, we investigated the histopathology of human ARHL and NIHL by comparing loss of auditory nerve fibers (ANFs), cochlear hair cells and the stria vascularis in a group of 52 cases with noise-exposure history against an age-matched control group. Although strial atrophy increased with age, there was no effect of noise history. Outer hair cell (OHC) loss also increased with age throughout the cochlea but was unaffected by noise history in the low-frequency region (<2 kHz), while greatly exacerbated at high frequencies (≥2 kHz). Inner hair cell (IHC) loss was primarily seen at high frequencies but was unaffected by noise at either low or high frequencies. ANF loss was substantial at all cochlear frequencies and was exacerbated by noise throughout. According to a multivariable regression model, this loss of neural channels contributes to poor word discrimination among those with similar audiometric threshold losses. The histopathological patterns observed also suggest that, whereas the low-frequency OHC loss may be an unavoidable consequence of aging, the high-frequency loss, which produces the classic down-sloping audiogram of ARHL, may be partially because of avoidable ear abuse, even among those without a documented history of acoustic overexposure.SIGNIFICANCE STATEMENT As regenerative therapeutics in sensorineural hearing loss enter clinical trials, it becomes critical to infer which cochlear pathologies are present in addition to hair cell loss. Here, by analyzing human autopsy material, we show that acoustic injury accelerates age-related primary neural degeneration, but not strial degeneration, neither of which can be inferred from audiometric thresholds. It exacerbates outer hair cell (OHC) loss only in the high-frequency half of the cochlea, suggesting that the apical loss is age-related, whereas the basal loss is partially noise induced, and therefore avoidable. Statistical analysis suggests that neural loss helps explain differences in word-recognition ability among individuals with similar audiometric thresholds. The surprising correlation between neural loss and OHC loss in the cochlea's speech region also implicates neural loss in the well-known decline in word scores as thresholds deteriorate with age.

A 20-Gb/s charge-steering equalizer utilizing highly-efficient charge-steering linear equalizer

The need for high-speed low-power receivers has grown due to the large increase in data rates and Internet-of-Things (IoT) applications. Equalizer-based receivers contain mainly two types of equalizers; Continuous-Time-Linear-Equalizers (CTLE) and non-linear equalizers implemented as Decision-Feedback-Equalizers (DFE). Many solutions have been introduced in the literature to enhance the power consumption of DFEs by using time interleaving or charge-steering. Still, the CTLE power consumption remains considerable a big portion of the overall power of the equalizer-based receivers. The CTLE function cannot be neglected as it equalizes the pre-cursors. This paper presents a low-power charge-steering based receiver that consists of a 1/8-rate charge-steering DFE integrated with a newly proposed Charge-Steering-Linear-Equalizer (CSLE). The proposed CSLE is explained qualitatively and quantitatively. The entire receiver chain consumes 2.9-mW running from a 0.9-V supply and can compensate for 14-dB channel loss at Nyquist-rate when processing 20-Gb/s data.

Advanced Biophysical Model to Capture Channel Variability for EQS Capacitive HBC.

Human Body Communication (HBC) has come up as a promising alternative to traditional radio frequency (RF) Wireless Body Area Network (WBAN) technologies. This is essentially due to HBC providing a broadband communication channel with enhanced signal security in the physical layer due to lower radiation from the human body as compared to its RF counterparts. An in-depth understanding of the mechanism for the channel loss variability and associated biophysical model needs to be developed before electro-quasistatic (EQS) HBC can be used more frequently in WBAN consumer and medical applications. Recent developments have shown biophysical models that capture the channel response for fixed transmitter and receiver positions on the human body which do not capture the variability in the HBC channel for varying positions of the devices with respect to the body. In this study, we provide a detailed analysis of the change in path loss in a capacitive-HBC channel in the EQS domain. Causes of channel loss variability namely: inter-device coupling and effects of fringe fields due to body's shadowing effects are investigated. FEM based simulation results are used to analyze the channel response of human body for different positions and sizes of the device which are further verified using measurement results to validate the developed biophysical model. Using the biophysical model, we develop a closed form equation for the path loss in a capacitive HBC channel which is then analyzed as a function of the geometric properties of the device and the position with respect to the human body which will help pave the path towards future EQS-HBC WBAN design.

A Mechanical Transmitter for Undersea Magnetic Induction Communication

The radio waves commonly used in terrestrial wireless communication are severely absorbed by the conductive seawater. However, the low-frequency magnetic fields can penetrate a much farther distance in seawater to realize wireless communication by magnetic induction (MI). An electromagnetic coil is generally used as a magnetic field generator, but it will consume excessive power when generating a strong magnetic field to achieve long-range communication. Instead, this study investigated the use of a motor-driven rotating permanent magnet as a mechanical transmitter for undersea MI communication. The frequency-dependent power consumption and the power-efficient operating frequency range of the mechanical transmitter were analyzed. To establish the undersea MI communication channel model, we derived exact analytic expressions for the fields generated by a rotating permanent magnet in seawater and explored the path loss of the undersea MI channel. A prototype mechanical transmitter using a cylindrical rare-earth magnet (Nd–Fe–B) with a diameter of 4 cm and a length of 15 cm driven by a servo motor was created, which consumes only about 2.74% of the power of the equivalent coil at 30 Hz. The surface-to-undersea MI communication using this prototype mechanical transmitter was demonstrated at a distance of 10 m.