A 58-year-old Malay man with a history of ischemic heart disease, well-controlled diabetes mellitus, and hypertension presented with sudden loss of vision after he underwent angiogram (cardiac catheterization) with contrast. Post-angiogram, his visual acuity deteriorated gradually to no perception of light bilaterally within 1 h duration. Clinically, there was no new intraocular pathology and neurologic examination was normal. An urgent noncontract computed tomography (CT) scan of the brain showed the presence of contrast in the venous sinuses but no obvious acute intracranial hemorrhage. Bilateral frontal and anterior parietal regions appeared hypodense with loss of gray white matter differentiation and relative effacement of the sulci. CT brain reported that infarction or cerebral edema may represent. Patient’s condition was observed, and after 24 h, his vision recovered to baseline and he was discharged well.