Incidence Of Facial Paralysis Research Articles

Facial nerve monitoring in parotid surgery: The standard of care?

Recent reports have suggested that the incidence of permanent facial nerve paralysis or paresis after surgery for benign parotid tumors is between 3% and 5%. The intraoperative use of nerve-integrity monitors has been advocated to reduce the incidence of facial nerve paralysis. The purpose of this study was to compare postoperative facial nerve function after monitored and unmonitored parotid surgical procedures. The charts of 69 consecutive patients with parotid lesions who underwent surgery from 1987 to 1996 were retrospectively surveyed. Sixteen high-risk patients were excluded from the study. The study group consisted of 53 patients (33 unmonitored and 20 monitored) who underwent lateral parotidectomy for mobile tumors of the superficial lobe of the parotid gland. No patient demonstrated permanent facial paralysis. In 9 patients (17%), transient nerve paralysis developed: 5 (15%) of the 33 patients who underwent lateral parotidectomy without the use of a nerve-integrity monitor and 4 (20%) of the 20 patients who underwent lateral parotidectomy with the use of a nerve-integrity monitor. Nerve-integrity monitoring is optional for mobile parotid tumors of the superficial lobe. (Otolaryngol Head Neck Surg 1998;119:468-70.)

Facial nerve paralysis in chronic suppurative otitis media: Ankara Numune hospital experience

Although the incidence of facial nerve paralysis (FNP) decreased with the use of contemporary diagnostic tools and the advent of antibiotics, it is still a challenging problem. In this retrospective study, the charts of 1188 patients with chronic suppurative otitis media (CSOM), treated between 1988 and 1996 in the First ENT Clinic of Ankara Numune Hospital, Turkey, were analysed to establish the pre-operative incidence of FNP in CSOM. There were 20 (1.7%) patients with FNP secondary to CSOM. Of these, 14 (70%) had cholesteatoma. There was a defect on the fallopian canal in 14 patients (70%) and it was most commonly located on the tympanic segment of the canal (35.7%). Paralysis was incomplete in all patients. A total of 14 patients had ear drainage more than 3 years. FNP was associated with vertigo in three patients (15%) and tinnitus in one patient (5%).

Temporal bone fractures and complications: Correlation between high-resolution computed tomography and audiography

Acute head trauma in the emergency room is managed primarily by the trauma surgeon or neurosurgeon. Temporal bone fractures with the complications of hearing loss and facial nerve paralysis may not be promptly evaluated. The otolaryngologist may not be consulted until late in the hospitalization. Traditionally, longitudinal fractures have been associated with conductive hearing loss and transverse fractures with sensorineural hearing loss. The purpose of this study was to correlate the type of fracture with audiometric findings of hearing loss. We also studied the incidence of facial nerve paralysis and its associated fracture types.

急性中耳炎による乳幼児顔面神経麻ひ４症例

In general, facial nerve paralysis after the onset of acute otitis media is related to the patient's age. We have treated four children with of facial nerve paralysis associated with acute otitis media over a 10-year period. They were divided into two groups based on their outcome; excellent recovery of facial nerve function, and poor recovery of it. We reviewed difference in the treatment between these two groups as well as the incidence of facial nerve paralysis. It is concluded that facial nerve paralysis is related to the characteristics of the temporal bone in infants, the facial canal dehiscence. Further treatment for facial nerve paralysis is also discussed.

Idiopathic facial palsy and pregnancy.

Idiopathic peripheral facial paralysis is a common clinical condition, but the etiology and preferred mode of treatment are still undecided. The currently popular etiological theory centers around disturbances of microvascular circulation. The literature contains conflicting statements on the incidence of this condition in pregnancy. Six cases are reported, one bilateral, in all of which the paralysis developed within six weeks of delivery. The incidence of facial paralysis was not significantly different in our pregnant and nonpregnant populations of similar age groups. A battery of tests failed to reveal any specific etiologic factors. None of the patients had toxemia, therefore, hypertension is not a causative factor. Comparison with the Melkersson-Rosenthal syndrome would indicate that tissue edema is not involved in the pathogenesis of the idiopathic paralysis of pregnancy. Both idiopathic sudden deafness and idiopathic facial paralysis occur preferentially in the later stages of pregnancy and the early puerperium. Further research into the pathophysiology of this stage of pregnancy may clarify some of the etiological problems of these two conditions.

Management of traumatic lesions of the facial nerve.

FACIAL paralysis is not commonly encountered today in the routine practice of otolaryngology. At one time in the history of this specialty, however, it was not uncommon after mastoidectomy, and it stimulated the constructive pioneering of Drobnik, 1 Korte, 2 Bunnell, 3 and Ballance and Duel, 4 on which the treatment of traumatic lesions of the facial nerve is founded. Fortunately, as the training in otolaryngology became better, the incidence of facial paralysis following mastoidectomy decreased and, with the advent of the sulfonamides and antibiotics, it was reduced to a relatively rare occurrence by the marked reduction in mastoid surgery. Nevertheless, injuries to the facial nerve still occur. Although facial palsy following surgical intervention on the mastoid process has largely been eliminated, its incidence due to other causes has increased. Some of these are the result of progress in our civilization. Trauma to the facial nerve following surgical procedures for