Impulsive Aggression Research Articles

The Relationship Between Cognitive Resource Consumption During Gameplay and Postgame Aggressive Behaviors: Between-Subjects Experiment.

The question of how video games can shape aggressive behaviors has been a focus for many researchers. Previous research has focused on how violent video game content leads to postgame aggressive behaviors. However, video games not only convey violence or prosocial content to players but also require cognitive effort from individuals. Since human cognitive resources are limited, consuming more cognitive resources in a game leads to less cognitive resources to suppress aggressive impulses. Therefore, the depletion of cognitive resources from playing video games may also lead to changes in postgame aggressive behaviors. This study aimed to examine the relationship between cognitive resources consumed in video games and postgame aggressive behaviors. A total of 60 participants (age: mean 20.22; range 18-24 y) were randomly assigned to either the high-load group or the low-load group. Participants from both groups played a video game centered around college life. In the low-load group, participants followed the gameplay instructions to complete it. In the high-load group, participants were given an extra digital memory task to complete while playing the game. Participants in both groups played the video game for about 25 minutes. A maze selection task was then conducted to measure the participants' helping and hurting behaviors. The independent samples 2-tailed t tests showed that the high-load group had significantly higher hurting scores (mean 3.13, SD 2.47) than the low-load group (mean 1.90, SD 2.12; t58=-2.07, P=.04; Cohen d=-0.535), whereas helping behaviors were not significantly affected (t58=1.52, P=.13; Cohen d=0.393). As more cognitive resources are consumed in a video game, more hurting behaviors are exhibited after the game. This finding proposes an alternative route by which video games impact aggressive behaviors, adding to previous theories and raising concerns about the popularity of cognitive training games.

The Insanity Defence: Is It Still Fit for Purpose?

The M'Naghten Rules formulated in 1843 have provided the basis for the insanity defence in many Western countries, including New Zealand. Although many candidates for the insanity defence experience psychosis, the principal determining factor is whether they knew their criminal act was morally wrong, a difficult metaethical judgement. In New Zealand the advent of methamphetamine abuse has created a significant challenge for forensic assessors in differentiating between mental disease and chronic intoxication, raising the question of whether the insanity defence as currently formulated is fit for purpose in assessing criminal culpability in such cases. The article explores this problem through an examination of a number of leading cases, noting the variable character of expert testimony on insanity where methamphetamine is involved. The article then examines the question of whether evidence of mental states falling short of insanity may be utilised to support a palliative claim reducing murder to manslaughter. A tentative new approach invites consideration of allowing investigation of insanity in cases involving meth-induced paranoia, whether or not the threshold of disease of the mind is met. In the concluding sections the article examines the impact of developments in cognitive neuroscience and asks whether neuroscience can help in determining criminal responsibility and whether it supports a "control limb" in a reformulated insanity defence. The article concludes with a brief discussion of mental disorder and impulsive aggression.

33.2 Clinical Psychopharmacology Management of Emotional Dysregulation and Impulsive Aggression in ASD

33.2 Clinical Psychopharmacology Management of Emotional Dysregulation and Impulsive Aggression in ASD

Examining Behavioral Approach and Inhibition to Further Characterize Youth With Impulsive Aggression

Examining Behavioral Approach and Inhibition to Further Characterize Youth With Impulsive Aggression

Impulsivity and Reactive-Proactive Aggression as Mechanisms of Alcohol-Related Sexual Intimate Partner Violence Perpetration among Romantic Partners

Background: Sexual intimate partner violence (S-IPV) commonly occurs within the contexts of committed romantic relationshops. Prior research has demonstrated the existence of a robust link between alcohol use and S-IPV. Despite this, few research studies have explored the etiological underpinnings of alcohol-related S-IPV perpetration, specifically. The present study examines the role of several key factors (i.e., problematic drinking, negative and positive urgency, proactive and reactive aggression) on S-IPV perpetration. Methods: Participants were 337 heavy drinking men and women in intimate relatinships who reported perpetrating some form of IPV toward their current partner within the past-year. A moderated-mediation model was used to determine how the key study variables interacted to predict S-IPV perpetration. Results: Results indicated that problematic drinking was positively correlated with both negative urgency and positive uregency. Findings also revealed that negative urgency, positive urgency, proactive aggression, and reactive aggression were all positively related to S-IPV perpetration. The indirect relationship between problematic drinking and S-IPV perpetration was mediated by positive urgency. Additionally, a significant main effect of proactive aggression on S-IPV perpetration was also detected. Conclusions: These findings suggest that impulsivity, specifically positive urgency, and proactive aggression may be under-appreciated constructs within the existing sexual aggression literature, and future research examining these variables as mechanisms explaining the association between the alcohol and S-IPV is warranted.

Prediction of Impulsive Aggression Based on Video Images.

In response to the subjectivity, low accuracy, and high concealment of existing attack behavior prediction methods, a video-based impulsive aggression prediction method that integrates physiological parameters and facial expression information is proposed. This method uses imaging equipment to capture video and facial expression information containing the subject's face and uses imaging photoplethysmography (IPPG) technology to obtain the subject's heart rate variability parameters. Meanwhile, the ResNet-34 expression recognition model was constructed to obtain the subject's facial expression information. Based on the random forest classification model, the physiological parameters and facial expression information obtained are used to predict individual impulsive aggression. Finally, an impulsive aggression induction experiment was designed to verify the method. The experimental results show that the accuracy of this method for predicting the presence or absence of impulsive aggression was 89.39%. This method proves the feasibility of applying physiological parameters and facial expression information to predict impulsive aggression. This article has important theoretical and practical value for exploring new impulsive aggression prediction methods. It also has significance in safety monitoring in special and public places such as prisons and rehabilitation centers.

Exploring the impact of childhood maltreatment and BPD on impulsivity in crimes of passion.

Crimes of passion, characterized as unpremeditated impulsive aggression, have garnered increasing attention in recent years. Impulsivity, a major factor in crimes of passion, is also a common feature of various health conditions, including Borderline Personality Disorder (BPD). Childhood maltreatment is considered a significant precursor to BPD and is closely related to impulsivity. Although prior research has affirmed the relationship between impulsivity, childhood maltreatment, BPD, and criminal behavior, few studies have examined these variables' interconnections within the context of crimes of passion. This study seeks to explore the relationship between childhood maltreatment, BPD, and impulsivity in crimes of passion, assessing the former's impact on the latter. Our research examined 133 crimes of passion offenders and 149 other offenders from the Shenzhen male prison in China. All 282 participants completed The Abbreviated Version of The Barratt Impulsiveness Scale (ABIS), The UPPS Impulsivity Scale (Urgency, Premeditation, Perseverance, Sensation Seeking), The McLean Screening Instrument for Borderline Personality Disorder (MSI-BPD), and The Childhood Trauma Questionnaire (CTQ). Our findings indicated that (1) crimes of passion offenders scored significantly higher in emotional neglect, physical neglect, and overall childhood maltreatment than did other offenders, and childhood maltreatment scores were notably higher in the high BPD trait group. (2) Crimes of passion offenders demonstrated significantly elevated impulsivity in attention and nonplanning scales compared to other offenders. Impulsivity scores across all scales were also significantly higher in the high BPD trait group. (3) Emotional neglect was found to significantly influence the urgency scale in crimes of passion offenders. An interaction effect was noted between physical abuse and high BPD traits, leading to increased impulsivity in crimes of passion offenders. This study underscores the predictive roles of childhood maltreatment and BPD in determining impulsivity within the context of crimes of passion.

Aggression as successful self‐control

AbstractA dominant narrative is that aggression starts when self‐control stops – unchecked aggressive impulses manifest in violence as self‐control fails to inhibit them. Yet this ‘low self‐control syndrome’ approach to aggression fails to accommodate numerous findings in which aggression arises from successful self‐control. I summarize these key findings while identifying how current theories of aggression can be molded to accommodate them. This balanced perspective, which allows aggression to arise from successful and unsuccessful self‐control, suggests exciting new hypotheses alongside confounding questions for aggression and self‐regulation scholars. It also supports ongoing and broader paradigm shifts away from treating self‐control as a purely adaptive and desirable psychological capacity and toward approaching self‐control interventions with greater care, as they may amplify aggression instead of reducing it, with costly consequences.

The association between brain serotonin transporter binding and impulsivity and aggression in healthy individuals

The serotonin system plays a critical role in the modulation of impulsive aggression. Although serotonin transporters (SERT) are key in modulating synaptic serotonin levels, few studies have investigated the role of SERT levels in human impulsive aggression. The aim of this study was to investigate whether brain SERT levels are associated with trait impulsive aggression. We included 148 healthy individuals (mean age 29.3 ± 13.0, range 18–80 years, 91 females) who had undergone positron emission positron (PET) examinations with the SERT tracer [11C]DASB and filled in self-report questionnaires of trait aggression, trait impulsivity and state aggression. We evaluated the association between cerebral SERT binding (BPND) and trait impulsive aggression in a latent variable model, with one latent variable (LVSERT) modelled from SERT BPND in frontostriatal and frontolimbic networks implicated in impulsive aggression, and another latent variable (LVIA) modelled from various trait measures of impulsivity and aggression. The LVSERT was not significantly associated with the LVIA (p = 0.8). Post-hoc univariate analyses did not reveal any significant associations between regional SERT levels and trait aggression, trait impulsivity or state aggression, but we found that state aggression at the day of PET scan was significantly lower in LA/LA homozygotes vs S-carriers of the 5-HTTLPR gene (p = 0.008). We conclude that brain SERT binding was not related to variations in trait impulsive aggression or state aggression. Our findings do not support that SERT is involved in mediating the serotonergic effects on aggression and impulsivity, at least not in individuals with non-pathological levels of impulsive aggression.

Empathy deficit in male patients with schizophrenia and its relationships with impulsivity and premeditated violence.

To explore the pattern of empathy characteristics in male patients with schizophrenia (SCH) and to examine whether empathy deficit is associated with impulsivity and premeditated violence. One hundred and fourteen male SCH patients were enrolled in this study. The demographic data of all patients were collected and the subjects were divided into two groups, namely, the violent group, including 60 cases, and the non-violent group, comprising 54 cases, according to the Modified Overt Aggression Scale (MOAS). The Chinese version of the Interpersonal Reactivity Index-C (IRI-C) was used to evaluate empathy and the Impulsive/Predicted Aggression Scales (IPAS) was employed to assess the characteristics of aggression. Among the 60 patients in the violent group, 44 patients had impulsive aggression (IA) and 16 patients had premeditated aggression (PM) according to the IPAS scale. In the violent group, the scores of the four subfactors of the IRI-C, i.e., perspective taking (PT), fantasy (FS), personal distress (PD), and empathy concern (EC), were significantly lower than in the non-violent group. Stepwise logistic regression showed that PM was independent influencing factor for violent behaviors in SCH patients. Correlation analysis revealed that EC of affective empathy was positively correlated with PM but not with IA. SCH patients with violent behavior had more extensive empathy deficits compared with non-violent SCH patients. EC, IA and PM are independent risk factors of violence in SCH patients. Empathy concern is an important index to predict PM in male patients with SCH.

Laboratory assessment of aggression: The Taylor Aggression Paradigm in adults with and without a disorder of impulsive aggression

IntroductionThe modified Taylor Aggression Paradigm (TAP) has been used to study impulsive aggression in experimental designs and has been relatively successful in addressing critiques of aggression paradigms; however, little has been done to examine the potential of using the TAP as a direct measure of aggression. This study aimed to explore the psychometric properties of the TAP behavioral indexes as measures of aggression. MethodsA community sample of 962 adults were divided into three groups based on diagnostic assessments: Intermittent Explosive Disorder; Non-Aggressive Psychiatric Disorder; or healthy controls. Participants then completed the TAP and self-report measures to assess construct validity. A subset of 47 participants completed a second TAP within one year to assess reliability. TAP indexes were based on number of “extreme” shocks selected (high shock index), average shock levels selected (mean shock index), and shocks levels selected without provocation (unprovoked aggression). ResultsOverall, TAP indexes were consistent and reliable. IED participants had the highest high shock and mean shock indexes of all groups (X2 = 49.93, p < 0.001). High shock index was related to trait aggression (β = 0.184, p < 0.001) after including covariates; mean shock index had a trending association with trait anger (β = 0.102, p = 0.059). ConclusionTAP behavioral indexes demonstrated promising psychometrics as a measure of aggression. High shock index appears to be more strongly associated with aggressive behavior; mean shock index may better measure general hostile responding. Future research might include comparisons specifically with impulse control disorders.

Impulsive aggression: recognition and choice of pharmacotherapy

Instrumental (controlled) aggression is observed in populations of both mentally healthy and mentally ill people. This type of aggression is not associated with any specific pathophysiological changes and cannot be considered as an independent symptom, which might be a target of pathogenetic therapy. By contrast, impulsive aggression is a paroxysm of anger, based on affectively narrowed consciousness when volition control is significantly reduced or absent. Impulsivity is different from cognate compulsivity by the loss of a stage of intellectual processing of affectively painted representations and by that the irrational attraction to action is realized involuntary, automatic. All impulsive-behavior patients are united by common pattern of neurobiological abnormalities, such as deficit of frontal inhibition of subcortical unconscious emotions and inclinations in response to relevant arousing stimuli. The results from evidence-based research, meta-analyses and Cochrane reviews of efficiency of pharmacological agents used for reduction of impulsive aggressive behavior were summarized.

Structural Brain Alterations in Borderline Personality Disorder

Borderline personality disorder (BPD) is one of three clusters of personality disorders characterized by a pattern of difficulty regulating emotion, impulse control, interpersonal relationships, and self-image. Emotional dysregulation, impulsive aggression, repetitive self-injury, and chronic suicidal tendencies make these individuals frequent users of mental health services. BPD has a variety of causes and involves several factors which interact in various ways with each other. Emotional dysregulation and impulsivity can be caused by genetic factors and traumatic childhood experiences and lead to dysfunctional behaviors, psychosocial conflicts, and deficits that may exacerbate emotional dysregulation and impulsivity. BPD has been linked to anatomical alterations in brain areas in the prefrontal cortex, cingulate cortex, hippocampus, amygdala, hypothalamus, corpus callosum, and frontolimbic network. These brain changes can lead to the manifestation of the inability to control behavior, feelings, and interpersonal relationships, which often leads to violence. These symptoms may begin at a young age or in early adulthood and tend to continue as the individual ages. The goal of this article is to review how structural changes in the brain can cause BPD. KEYWORDS Borderline personality disorder, MRI in BPD, BPD patients, personality disorders, structural imaging BPD, neurobiology BPD

Momentary assessment of the relations between narcissistic traits, interpersonal behaviors, and aggression.

This study explores the associations among narcissistic traits, interpersonal behaviors, and aggression using repeated, situation-based measurement. We examine narcissism's relations with aggression across three levels of its theorized hierarchy (level 1: narcissism; level 2: grandiose vs. vulnerable narcissism; level 3: antagonism, agentic extraversion, and narcissistic neuroticism). Using an experience-sampling approach, the current study examined the effects of narcissism and its finer-grained components on daily affective experiences and aggressive behaviors in the context of interpersonal interactions. Data were collected from 477 undergraduate students who were instructed to complete four prompts a day for ten consecutive days. Narcissism at the global construct level positively predicted multiple indices of episodic aggression (i.e., aggressive temper, aggressive urge, verbal aggression). At the dual-dimension level, grandiose narcissism specifically predicted aggression, and then at the trifurcated level, interpersonal antagonism predicted aggression by itself and in interaction with event-level negative affect. Negative affect consistently exhibited both within- and between-person effects on aggression. In real-life social interactions, narcissism dimensions differentially affect the way individuals experience social interactions and process negative affect, and thus in both research and clinical practice, narcissism is best assessed as a heterogeneous, multidimensional construct.

Le rôle de l’alexithymie dans les comportements violents : Une révision des études disponibles à la lumière du modèle de traitement de l’information sociale

ObjectivesThe literature highlights the prevalence of shortcomings in processing emotional information, especially the identification of emotions in the context of violent behavior. Alexithymia is the inability to recognize and describe emotional states and external oriented thinking. The social information processing approach offers a theoretical framework to understand how alexithymia deficiencies are linked to violence. Furthermore, this approach allows us to determine which stages of processing are associated with specific kinds of deficiencies in order to adapt treatment programs targeting violent behavior. The primary goal of this narrative review is to define and present the relationship between alexithymia and violent behavior. In addition, this review will attempt to illustrate that deficiencies caused by alexithymia are related to different stages of social information processing. MethodsThis review presents an explanation of the links between violent behavior and alexithymia according to the social information processing theory as a theoretical framework. ResultsThere are numerous studies that demonstrate the relationship between violent behavior and alexithymia in different populations (general, psychiatric, or incarcerated) and for different kinds of violence (physical, homicidal or sexual). In particular, the deficiencies caused by alexithymia primarily involved the (in)ability to identify emotional states. Indeed, the more people have difficulties in identifying their own internal emotional states, the higher the probability that they will engage in dysfunctional behavior such as violence. According to the social information processing model, these shortcomings are present in the initial stages of processing. Errors in detection and perception of social cues could produce a misinterpretation of other's intentions and could lead to a hostile attribution bias. This bias exhibits itself as a tendency to interpret the actions of others as hostile, particularly in ambiguous situations and is often linked to impulsive and reactive aggression. Due to these links between alexithymia, misinterpretation, and violent behavior, it seems essential to develop treatment programs designed to reduce the effects of alexithymia. One study demonstrated that a specific treatment program reduced alexithymic related deficiencies among individuals who had committed sexual offences. This program was based on developing the capacity to identify emotions and to use emotive language to verbalize emotional states. These results suggest that it is possible to improve emotional competence and reduce the effects of alexithymia, which could lead to a reduction in errors in social information processing and could be a major protective factor with respect to violent behavior. ConclusionsThe social information processing model allows us to understand the relationship between violent behavior and alexithymia based on the deficiencies in specific processing stages. In other words, this is an approach which examines the varying stages of social information processing which are dysfunctional and their relationship to the various dimensions of alexithymia. Importantly, it seems that the two explanatory hypotheses of alexithymia may be more complementary rather than diametrically opposed. Indeed, these two hypotheses allow us to explain particular kinds of deficiencies based on different stages of social information processing. This heuristic framework promises the possibility of understanding the study of alexithymia depending on both the nature of the shortcomings and on which social information processing steps are involved. Accordingly, it could become easier to accurately understand which processes are involved in order to adapt the treatment of these deficiencies, particularly in relation to violent behavior.

Outpatient management of REM sleep behavior disorder case in Brunner syndrome

IntroductionBrunner syndrome is a recessive X-linked disorder characterized by impulsive aggressiveness and mild mental retardation associated with Monoamine Oxidase – A (MAOA) deficiency (Brunner et al. Science 1993; 262 578-580).ObjectivesTo present a REM sleep behavior disorder (RBD) case in a patient with Brunner syndrome.MethodsThe present study is a case report of a patient followed in our hospital’s outpatient care. We also searched for previous case reports of sleep disorders and other clinical features in Brunner syndrome using a pubmed query.ResultsA 46-year-old Spanish male, diagnosed with Brunner syndrome due to the mutation c.1438A&gt;G/iVS14-2 A&gt;G, a loss-of-function mutation in the X-linked MAOA gene. He suffers from mild mental retardation and psychotic disturbances treated with SSRI and antipsychotic drugs. The patient was referred to our outpatient care to assess his sleep abnormal behaviors. He had been presenting with episodes of sleep-related vocalization and complex motor behaviors during sleep for the last 3 years, correlating with dream mentation. His relatives recounted episodes of talking, screaming, gesturing, kicking, falling out of bed and crying during sleep. Dream content referred by the patient was often related to persecutions, attacks and fights.Polysomnography revealed vocalization and gesticulation during REM sleep compatible with the diagnosis of RBD. The addition of clonazepam to his treatment at doses of 1-3 mg per day achieved significant clinical response of the sleep disorder.ConclusionsThe clinical presentation suggested the diagnosis of RBD case in a patient with Brunner syndrome. Although sleep disorders are not one of the most important or frequent clinical features in Brunner syndrome, they are described in the literature and can significantly affect the patient’s quality of life. To our knowledge, this is the first report about clinical management of RBD case in Brunner syndrome.Disclosure of InterestNone Declared

An Integrated Developmental Approach to Personality Disorders in Adolescence: Expanding Kernberg's Object Relations Theory.

This article is a tribute to Dr. Otto F. Kernberg and his contribution to the theoretical understanding of personality pathology in adolescence. In his object relations theory of the structure of personality, an integrated identity is considered central to healthy personality functioning and is contrasted with identity diffusion, which is posited to underlie the maladaptive interpersonal functioning of patients with severe personality disorders. His work provided an early theoretical foundation for the understanding and treatment of personality disorders in adults, but a need remained for a deeper understanding of the development and treatment of personality disorders during adolescence. During the past 10 years, Kernberg has led a group of clinicians and developmental researchers focusing on childhood and adolescence to elaborate an understanding of the development, assessment, and treatment of personality disorders among adolescents. He proposed that in the context of typical development, adolescence is important to the expansion of identity, because it represents the period when sexual and aggressive impulses are integrated and earlier representations of the self and others are revised and become more realistic and nuanced. Furthermore, adolescence entails a reorganization of the moral system and enrichment of the ego ideal. This article presents two methods for assessing personality disorders in adolescence that are based on Kernberg's theory and demonstrates the use of transference-focused psychotherapy for adolescents with personality disorders.

Serotonin transporter availability in physically aggressive personality disordered patients: associations with trait and state aggression, and response to fluoxetine.

Characterizing the neuroanatomical basis of serotonergic abnormalities in severe, chronic, impulsive aggression will allow for rational treatment selection, development of novel therapeutics, and biomarkers to identify at-risk individuals. The aim of this study is to identify associations between regional serotonin transporter (5-HTT) availability and trait and state aggression, as well as response to the anti-aggressive effects of fluoxetine. We examined 5-HTT availability using positron emission tomography (PET) imaging with [11C]DASB in personality disordered patients with current physical intermittent explosive disorder (IED; n = 18), and healthy comparison participants (HC; n = 11), in the anterior cingulate cortex (ACC), amygdala (AMY), ventral striatum (VST), and midbrain (MID). After PET imaging, IED patients were treated with fluoxetine 20mg daily (n = 9) or placebo (n = 6) for 12weeks. Trait and state aggression, trait callousness, and childhood trauma were assessed. In IED patients, trait aggression was positively associated with [11C]DASB binding in the ACC and VST; covarying for trait callousness and childhood trauma enhanced these correlations. Baseline state aggression was positively correlated with ACC [11C]DASB in IED patients. Greater baseline VST [11C]DASB binding predicted greater decreases in state aggression with fluoxetine treatment. Consistent with prior reports, ACC 5-HTT is related to trait aggression, and adjusting for factors related to proactive (callousness) and reactive (childhood trauma) aggression subtypes further resolves this relationship. Novel findings of the study include a better understanding of the association between regional 5-HTT availability and state aggression, and the involvement of VST 5-HTT with trait aggression, and with the anti-aggressive effects of fluoxetine.

Finding a Needed Diagnostic Home for Children with Impulsive Aggression.

Aggressive behavior is one of the most common reasons for referrals of youth to mental health treatment. While there are multiple publications describing different types of aggression in children, it remains challenging for clinicians to diagnose and treat aggressive youth, especially those with impulsively aggressive behaviors. The reason for this dilemma is that currently several psychiatric diagnoses include only some of the common symptoms of aggression in their criteria. However, no single diagnosis or diagnostic specifier adequately captures youth with impulsive aggression (IA). Here we review select current diagnostic categories, including behavior and mood disorders, and suggest that they do not provide an adequate description of youth with IA. We also specifically focus on the construct of IA as a distinct entity from other diagnoses and propose a set of initial, provisional diagnostic criteria based on the available evidence that describes youth with IA to use for future evaluation.

Trait anger and aggression among male violent offenders: The mediating effect of sadistic impulse and the moderating effect of emotion regulation

We investigated the mediating role of sadistic impulse and the moderating role of emotion regulation strategy in the relationship between trait anger and aggressive behaviour in violent offenders. A total of 492 male violent offenders (mean age = 34.06 years, SD = 6.67 years) completed the Trait Anger Scale, Emotion Regulation Questionnaire, Short Sadistic Impulse Scale, and Impulsive/Premeditated Aggression Scale. We tested a moderated mediation model by multiple linear regression analysis (Bootstrap n = 5 000). The results showed that sadistic impulse mediated the relationship between trait anger and aggressive behaviour in violent offenders. Higher level of sadistic impulse was associated with a higher likelihood of aggressive behaviour. Emotion regulation strategy moderated the relationship between trait anger and aggressive behaviour. Specifically, those with lower emotional regulation were more likely to exhibit aggressive behaviour compared to those with higher emotional regulation. The mediated moderation model applied to both impulsive and premeditated aggression. In the low emotion regulation group, the mediating effects of trait anger on impulsive and premeditated aggression were significant. In the high emotion regulation group, the mediating effects of trait anger on impulsive and premeditated aggression were lower, but still significant. Our findings are consistent with the emotion regulation theory of aggression stating that people with sadism may assault others to improve their mood.