Diminished quadriceps force control following ACL rupture may reflect quadriceps dyskinesia, which may be attributed to modulation or reorganization within the motor cortex. Reorganization within the motor cortex may be characterised by changes in the cortical motor representation of a muscle. Alterations in the cortical motor representation position is associated with dysfunctional motor control in a number of musculoskeltal conditions, but has not been examined following ACL rupture. PURPOSE: To determine the association between quadriceps cortical motor representation position and accuracy of quadriceps force output following ACL rupture. METHODS: Eighteen individuals (12M/6F; 29.8 ± 8.7years; 69.5 ± 42.5 days post-injury) with a unilateral isolated ACL rupture. While seated in a dynamometer, participants performed an isometric quadriceps contraction to match a fluctuating force target between 5% to 25% body weight (BW). The root mean square error (RMSE) of participants force relative to target force was used to determine accuracy. Motor representation of the quadriceps was determined using transcranial magnetic stimulation. Four stimuli at 120% of motor threshold were given 5s apart while participant contracted their quadriceps at 10% BW, starting at the identified optimal site moving in 1cm steps in anterior, then posterior directions until a motor evoked potential could no longer be elicited. This pattern was repeated for lateral, then medial sites until all map borders had been determined. Pearson product moment correlations (P<0.05) were used to assess associations between RMSE and motor representation position in the x- and y-axes. RESULTS: There were no significant associations between RMSE and y-axis center position of the motor representation in either limb of the ACL group (r=0.28 P=0.31 INV; r=0.30 P=0.32 UNIV). Also, no significant associations between RMSE and x-axis center position in either limb of the ACL group (r=0.099 P=0.73 INV; r=-0.36 P=0.27 UNINV). CONCLUSIONS: Quadriceps force control following ACL rupture was unrelated to cortical motor representation position. Alternatively, impaired quadriceps force control following ACL rupture may relate to alterations in spinal reflex excitability, or altered connectivity with higher brain centers.