1.1. Seventy-three placentas of stillborn infants were described, sectioned, and appraised in a retrospective study of the diseases of the placenta after fetal death in utero.2.2. Moderate to marked placentitis, in which maternal and fetal response by neutrophilic infiltration had extended to the amnion, chorion, or umbilical cord was apparent in 42 per cent of the 73 placentas.3.3. Moderate to marked maceration with denudation or bullae of the skin, softness of the eyeball, red-brown fluid of the body cavity, and loss of organ weight was recorded in 39 per cent or 29 out of the 73 stillborn infants. Fetal maceration was frequently associated with erythroblastosis and diabetes mellitus.4.4. Sixty-four per cent or 47 of the 73 stillborn infants were premature (45 per cent) or immature (19 per cent).5.5. Partial or complete fibrous occlusion of the fetal vessels in the stem villi was observed in 38 per cent or 28 of 73 placentas of stillborn infants. This vascular changes was commonly associated with moderate to marked fetal maceration. Villous infarction and thrombosis were independent of this vascular change, however.6.6. Partial or complete occlusion of the fetal vessels in the placentas of stillborn infants was often produced by subintimal proliferation of fibrous tissue and not by intimal proliferation or thrombosis.