A 47-year-old male was admitted to our hospital with numbness and clumsiness in both hands. These symptoms developed 4 months ago and exacerbated progressively. A neurological examination revealed mild distal symmetrical sensory loss. He had no apparent muscle weakness in his shoulder muscles, but the medial section of the scapula was winging, especially after 120 of elevation and when he was asked to push the wall with both hands. The patient did not complain of pain. Nerve conduction studies (Table 1) demonstrated conduction blocks, slowed nerve conduction velocities, and prolonged distal latencies and F latencies in more than 2 peripheral nerves, meeting the electrophsyiological diagnostic criteria for CIDP [1]. By needle electromyography, we observed spontaneous denervation potentials in the serratus anterior muscle. There were no abnormalities in the other neck and shoulder muscles. His spinal magnetic resonance images were within normal limits. Examination of cerebrospinal fluid (CSF) revealed nothing except elevated protein levels (80 mg/dL). The patient was negative for anti-GM1, and no elevation of monoclonal antibodies was noted by blood or urine electrophoresis. There was nothing in his medical or family history that helped us exclude underlying hereditary polyneuropathy. Due to symmetrical diffuse demyelinating involvement in the nerve conduction studies, duration of the complaints, and elevated CSF protein levels, the patient was diagnosed with CIDP after excluding paraneoplastic conditions, diabetes mellitus, vasculitis, and rheumatological and hematological diseases. The patient was treated daily with 1 mgr/kg cts. His clinical symptoms, including the winging scapula, improved by the end of Week 8. In addition, the results of the nerve conduction studies improved slightly, and the conduction block abated (Table 2). Further, there were no abnormalities by repeated needle electromyography.