The incidence of intestinal atresia has been variously quoted as 1/1,500 to 1/20,000 newborn infants (2). Atresias and stenoses of the colon occur in 4 to 15 per cent of these cases. Bland Sutton's (1) (1889) original classification of intestinal atresias into three different types consists of: I. Complete occlusion of the intestinal lumen by a diaphragm. II. Complete occlusion of the intestinal lumen terminating in blind ends joined by a cord-like structure. The mesentery may or may not be intact. III. Complete occlusion as in Type II, but without connection between the proximal and distal segments; and, in addition, a V-shaped defect in the mesentery. Several theories have been postulated regarding the pathogenesis of intestinal atresia. Tandler's “failure of recanaliza-tion theory” has been challenged by the finding that obliteration of the bowel lumen by epithelial proliferation takes place mostly in the duodenal, proximal jejunal, and rectal areas, between the fifth and eighth fetal weeks. This does not occur in the remainder of the bowel which is always patent (6). The common finding of bile, squamous cells, and lanugo hair distal to the point of obstruction further discredits this theory because they are not present until after the eighth week of fetal life. A more accepted theory of “vascular insufficiency,” although introduced in 1859, was only recently demonstrated by Barnard and Louw's experimental work in 1956 (3, 4). They showed that the three types of atresia could be produced by ligating the mesenteric vessels in fetal dogs. Vascular occlusion can occur at any stage of the development of the fetus, accounting for the presence of bile, squamous cells, and lanugo hair beyond the point of obstruction. The paucity of other congenital anomalies associated with colonic atresias also favors this theory (7). Prior to their report in 1963, Peck et al. (2) discovered a total of only 36 patients with colonic atresia or stenosis who had survived and added 2 additional cases. They found the defects equally distributed proximal and distal to the splenic flexure. Atresias of Types II and III were more common in the right colon, and Type I was more common on the left side. Successful treatment of colonic atresia requires an early diagnosis as there have been no survivals in infants treated beyond the third or fourth day of life (2, 17). The co-operative efforts of the pediatrician, surgeon, and radiologist can help overcome the delay in diagnosis, thereby decreasing the morbidity and mortality rate in this condition. We feel the subject warrants attention and are reporting our recent experience in 2 cases, as we have been unable to find any report of congenital colonic atresia in the American radiological literature. Case I: F. C., a 48-hour-old white male, product of a normal term delivery, was admitted to Variety Children's Hospital, Miami, Fla. He was vomiting bile-stained material, and progressive abdominal distention was noted.