tions correctly. This applies to all phases of prosthetic dentistry, viz., bridge, partial, and full denture construction. When a patient with periodontal involvement presents himself, the dentist who has had proper periodontal training is able to make his diagnosis, or analysis of the case ; his prognosis, or prediction of the results which will be obtained ; and his recommendations for procedure with the sincere feeling of certainty that he is doing the best thing possible for that patient. He will be less hasty in advising the removal of involved teeth, and will consequently save more teeth than he had before felt possible. In short, he will be giving his patients better service and better dentistry. Consider then, the condition of the gingival tissues in cases of periodontal involvement. These gingival tissues are inflamed, and the degree of inflammation may vary clinically from acute to subactite, or to chronic. It is quite generally accepted that wherever inflammation of soft tissue exists, there is also a degree of inflammation and resultant resorption in the immediately adjacent bony structures. Thus, rheumatic patients exhibit resorption in the bone of the joints which are inflamed. Also, a simple fracture of a long bone will, within a week after the fracture, show radiographic evidence of marked resorption of each end of the fracture. Leriche and Policard’ state that rarefaction and resorption of the fragments of a fracture begin immediately after the trauma, and progress to such a degree that after seven or eight days exact anatomic coaptation of the fracture is most difficult. They further state that such resorption is manifested by enlargement of the Haversian canals and of the connective tissue spaces. The bone disappears by osteoclasis (osteoclastic resorption), much more than by osteolysis (chemical absorption), and as the circulatory conditions return to normal, the rarifaction ceases. Weinmann and Sicher’ report that necrosis of bone is always found at the frac
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