Congenital Intestinal Obstruction Research Articles

Enterogenous cyst causing congenital intestinal obstruction.

Enterogenous cyst causing congenital intestinal obstruction.

Atresia of the lower part of midgut and the whole of hindgut

Two cases of congenital intestinal obstruction due to intestinal atresia are described. Review of the literature and significant points in the pathological embryology are discussed.

CONGENITAL DUODENAL OBSTRUCTION

IN recent years there has been a significant improvement in the results of the surgical management of congenital duodenal obstruction. In November 1945 Swenson and Ladd 1 reported 21 cases of intrinsic duodenal obstruction in the newborn, with 13 recoveries after operation. At the same time they reported 30 cases of extrinsic obstruction, with 26 recoveries. This record is in sharp contrast to that existing only two decades ago. In 1922 Davis and Poynter 2 presented 392 cases of congenital intestinal obstruction of all parts of the intestinal tract above the anus. In this survey of the literature, based on observations either at autopsy or operation, there were only 2 recoveries, a mortality of almost 99.5 per cent. This striking decrease in mortality can to a considerable extent be attributed to the early roentgenographic diagnosis of these developmental anomalies. Swenson and Ladd 1 emphasized that not only is the mortality

CONGENITAL INTESTINAL OBSTRUCTION

The prognosis for congenital intestinal obstruction has changed materially since 1927. Prior to this date, authorities were unanimous in predicting an almost uniformly fatal outcome after a brief illness.1In view of the fact that previously only 4 patients were known to have survived, this estimate of the mortality was an understatement. Since that date, reports of increasing numbers of patients successfully treated surgically have led both pediatricians and surgeons to recommend, with some optimism, immediate surgical intervention in cases of congenital obstruction of the bowel. The first successful operations were performed in 1897,21910,31916,419245and 1926.6In 1927 there were reports of a number of similarly fortunate operative results.7Since that year, equally happy reports have appeared at not infrequent intervals. Loitman,7ain 1927, and Webb and Wangensteen,8in 1931, were the first to sound an encouraging note

Congenital obstruction of the alimentary tract

<h2>Summary</h2> Eight cases of congenital obstruction of the alimentary tract are reported.<ul><li>1.Congenital esophageal atresia with communication between the upper segment of the esophagus and the trachea.</li><li>2.Congenital esophageal atresia with probable communication between the lower segment and the trachea.</li><li>3.Partial atresia of the esophagus with narrowing of the lower one-third. The characteristic symptoms of atresia of the esophagus are immediate regurgitation with almost the first mouthful, and constant dribbling of mucus. There may or may not be violent coughing after feeding, with cyanosis.</li><li>4.Case of partial atresia of distal end of duodenum. Duodenal obstruction is to be differentiated from pyloric stenosis. The presence of bile in the vomitus, and x-ray examination with barium feeding will aid in the diagnosis.</li><li>5.A successful operative result of congenital obstruction of the ileum.</li><li>6, 7.Incomplete and complete obstruction, respectively, of the colon.</li><li>8.Obstruction due to volvulus with symptoms immediately after birth.</li></ul> It is interesting to note that of the eight cases reported seven were males. The symptoms of congenital intestinal obstruction are characterized clinically by persistent vomiting, distention, constipation or mucous stools stained with blood, anuria, and progressive loss of weight. Although congenital obstruction is very rare, it should be kept in mind whenever a newborn baby vomits. It is obviously important to recognize these cases early because operative intervention is the only treatment, however unsuccessful the results may be, or have been in the past.

CONGENITAL OBSTRUCTION OF THE SMALL INTESTINE

This paper is based on a study of sixty cases of congenital intestinal obstruction occurring in the Children's Hospital of Boston. Only those cases are included in which the obstruction was located in the small intestine and was due to one of two factors. Intrinsic obstruction is the interference in the continuity of the lumen of the intestine due to intraintestinal defects. Extrinsic obstruction is the interference in the function of the intestine by external pressure due to faulty rotation. Obstruction caused by bands which may or may not be of congenital origin and that, due to hernias and the vagaries of Meckel's diverticulum are not included. EMBRYOLOGY To understand these conditions one must consider the normal embryologic development of the intestine. Most of the embryologic events take place between the fifth and tenth weeks of fetal life. Prior to the fifth week of fetal life the intestine presents a

Primary Sarcoma of Stomach; Pyloric Stenosis of the New Born; Congenital Intestinal Obstruction in a New-Born Baby; Report of Cases

Primary Sarcoma of Stomach; Pyloric Stenosis of the New Born; Congenital Intestinal Obstruction in a New-Born Baby; Report of Cases