339 During high intensity repetitive tasks (e.g. successive shifts in hockey) performance declines secondary to residual fatigue from previous efforts. To evaluate the degree to which this was related to lactate (HLa) accumulation, we studied collegiate hockey players (n=29) during repeat sprint skating (RSS). Each player performed 6 reps of a 91m skating course (end line to end line, back to blue line nearest start) with a new repetition every 30s. Intermediate times were recorded for the first 16m of the RSS). Peak blood HLa concentration (as a surrogate of muscle HLa) was measured before & after the test, & after subsequent versions when the test was stopped after 2 & 4 repetitions. From these measurements, we calculated what the effective peak blood HLa would be prior to each skating trial. Through the RSS, performance as measured by time to complete the trial deteriorated significantly for both the full RSS: 12.47(.49), 13.06(.47), 13.80(.45), 14.43(.56), 14.86(.74), & 15.12(.81)s and for the first 16m of the RSS: 3.15(.12), 3.30(.12), 3.48(.15), 3.60(.15), 3.72(.18), 3.79(.18)s.. At the same time, the calculated peak pre-repetition blood HLa increased significantly from trial to trial: 2.29(.37), 7.18(1.24), 11.48(2.05), 14.57(2.37), 16.81(2.63, 18.01(2.41) & was 18.58(2.54) mmol*1-1 following the last repetition. The mean individual correlation between calculated pre-trial blood lactate and skating time was significant for the full course (r=0.80) and for the first part of the course (r=0.77). We conclude that the pretrial peak blood Hla concentration, which we take as a surrogate of pre trial muscle lactate, significantly influences RSS. This suggests that strategies designed either to prevent the accumulation of lactate or to clear lactate from the musculature during recovery periods might serve to facilitate skating performance.
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