Chronic Miliary Tuberculosis Research Articles

Skin Rash with Acute Respiratory Distress Syndrome in Chronic Miliary Tuberculosis: The Role of HRCT Thorax and Bronchoscopy as Diagnostic Tools

Pulmonary tuberculosis (TB) is a leading cause of mortality and morbidity in India, presenting with diverse clinical and radiological manifestations such as consolidation, cavitation, lymphadenopathy, pleural effusion, miliary TB, and endobronchial TB. While acute miliary TB presenting as Acute respiratory distress syndrome (ARDS) is documented, the association with a skin rash mimicking viral exanthematous fever is not. This case report describes the case of a 41-year-old male patient with acute dyspnea, a maculopapular rash, and lung infiltrates on chest radiograph, initially diagnosed with acute hypoxic respiratory failure and later confirmed as ARDS. High-resolution computed tomography (HRCT) thorax revealed consolidation with nodules and a characteristic butterfly pattern sparing the apices and bases. Bronchoscopy confirmed TB etiology through MTB genome in BAL samples. The patient improved significantly with intravenous antibiotics, steroids, and HFNC, leading to a documented cure after 6 months of anti-tuberculosis treatment. This report highlights the importance of considering TB in cases with nodular consolidation and positive limiting signs and recommends ICU bronchoscopy as a reliable diagnostic tool. The Kigali modification for ARDS definition was instrumental in managing this case. Use of Kigali modification for defining ARDS in resource limited setting guided management in our case.

Hematogenous pulmonary tuberculosis. Med. Klin. 1932, 39

Assmann distinguishes between the trace, the forms of hematogenous pulmonary tuberculosis: acute, subacute, chronic miliary tuberculosis, miliaris discreta, lymphangitisreticularis. Spicy Mil. TBK occupies an insignificant place among the hematogenous forms of pulmonary TBK. Subacute and chronic mil. TBKs often have a benign course, even with common rashes, there is either resorption, or scarring of the lesions and recovery.

Radiology’sLegacy: Rummaging through the Attic

Early articles in Radiology are based on conventional radiography and provide the foundation for both the way we think about extracting diagnostically important information from images and the spectacular technologic achievements in the decades that followed; therefore, we decided to include in this issue of Radiology a pictorial essay of select figures and our thoughts on the articles themselves to give a taste of the beginnings of so many of our current imaging and research activities.

Lymphangitis reticularis tuberculosa

Summary Lymphangitis reticularis tuberculosa is the description given to the appearance in chest radiographs of an evenly distributed network of tuberculous lymphatic vessels in the lungs. A late result of this tuberculous infection in the untreated case is a fibrous network in the lungs with little or no distortion of the lung architecture. This condition is closely allied to chronic miliary tuberculosis and has in fact been described as the lymphogenous form of this disease. The literature is reviewed and evidence presented suggesting that the condition followshaematogenous dissemination of tubercle bacilli in the lungs. The theories regarding its evolution are discussed. Four illustrative cases, selected from many examples seen, are described.

Hepatomegalic liver in children with chronic miliary tuberculosis and liver biopsy findings; studies upon the nutrition of children in Hirosaki area. 11th Report.

1. In Hirosaki Area infantile pellagra or nutritional dystrophy has been prevalent.2. Marked enlargement of the liver as a prominent feature among clinical manifestations of chronic miliary tuberculosis in children in this Area should be regarded in a new light.3. Liver biopsy was carried out in 2 cases of chronic miliary tuberculosis, and it revealed miliary tubercles, portal fibrosis and marked fatty infiltration of the organ.4. Marked enlargement of the liver in these tuberculous children will be ascribed to fatty infiltration of the liver (identified by liver biopsy) probably due to dietary deficiency prior to their tuberculous infection.

Chronic miliary tuberculosis

Summary The case is recorded of a young woman who suffered from a preretinal h˦morrhage followed a few months later by neurological symptoms which proved eventually to have been due to tuberculomas of the brain and cerebellum. After having made an apparent recovery for a year, she then developed renal tuberculosis and was found to have miliary tuberculosis of the lungs and disseminated choroiditis. Death occurred twenty-three months after the first ocular manifestations; by then tuberculosis had gradually affected most of the organs. The unusual features of this case and the varied picture of h˦matogenous tuberculosis are discussed.

The prognosis of chronic miliary tuberculosis in children

Twenty-one cases of chronic miliary tuberculosis in children are analysed with regard to the course of their lung lesions, the course of their disease and the incidence among them of other tuberculous conditions. Assessment of the prognosis for such cases should be divided into two parts: (1) the immediate prognosis, depending on recovery from the initial miliary spread into the lungs, the chances of which are approximately inversely proportional to the degree of tox˦mia; (2) The late prognosis, depending upon the development of further tuberculous lesions. It is suggested that the late prognosis depends mainly on the healing of the bloodstream focus and that this focus is often a caseating, paratracheal gland.

Thoracic Manifestations of Sarcoidosis

The cutaneous lesions long known as lupus pernio and the glandular, ocular, osseous, mucous membrane, and systemic disease known as sarcoid were first described as one clinicopathological entity by Schaumann (9, 10) in 1914. He demonstrated clearly the lung alterations occurring in sarcoidosis which could be shown radiographically and rejected the concept that the changes were due to residues of pneumonia or to a dissemination of tuberculous nodules. He directed attention to the close similarity between the roentgen appearance in so-called chronic miliary tuberculosis and certain cases of sarcoidosis, although, in the latter, strandlike infiltrations radiating from the hilar regions were more characteristic. It is now well recognized that the histologic unit of sarcoidosis is the so-called “hard tubercle,” which consists of a collection of pale-staining epithelioid cells, with frequent giant cells, in which there is no peripheral inflammatory zone. There is no necrosis. Calcification, when it occurs, is v...

Chronic miliary tuberculosis

Acta Medica ScandinavicaVolume 113, Issue 2-3 p. 135-162 Chronic miliary tuberculosis S. van CREVELD, S. van CREVELD Children's Sanatorium »Hoog-Blaricum», director dr. G. J. Huët, The NetherlandsSearch for more papers by this authorG. J. HUËT, G. J. HUËT Children's Sanatorium »Hoog-Blaricum», director dr. G. J. Huët, The NetherlandsSearch for more papers by this author S. van CREVELD, S. van CREVELD Children's Sanatorium »Hoog-Blaricum», director dr. G. J. Huët, The NetherlandsSearch for more papers by this authorG. J. HUËT, G. J. HUËT Children's Sanatorium »Hoog-Blaricum», director dr. G. J. Huët, The NetherlandsSearch for more papers by this author First published: January/December 1943 https://doi.org/10.1111/j.0954-6820.1943.tb09152.xCitations: 1AboutPDF ToolsRequest permissionExport citationAdd to favoritesTrack citation ShareShare Give accessShare full text accessShare full-text accessPlease review our Terms and Conditions of Use and check box below to share full-text version of article.I have read and accept the Wiley Online Library Terms and Conditions of UseShareable LinkUse the link below to share a full-text version of this article with your friends and colleagues. Learn more.Copy URL Share a linkShare onFacebookTwitterLinked InRedditWechat Citing Literature Volume113, Issue2-3January/December 1943Pages 135-162 RelatedInformation

Chronic Miliary Tuberculosis

Chronic Miliary Tuberculosis

Chronic miliary tuberculosis: Case report

Chronic miliary tuberculosis: Case report

Chronic Miliary Tuberculosis

Chronic Miliary Tuberculosis

On the Radiological Aspects of an Investigation into the Relationship of Chronic Irido-Cyclitis, Uveo-Parotitis and Tuberculosis

1. The radiological study of the chest in 40 unselected cases of chronic irido-cyclitis was productive of some striking findings. 2. Of the 40 cases, 24 had positive findings in the lungs; of these no less than 12 cases showed pulmonary and hilar changes, the like of which was only resembled closely by published illustrations of chronic miliary tuberculosis and sarcoidosis. 3. These 12 cases were regarded as being examples of chronic miliary tuberculosis of either hæmatogenous or lymphogenous origin, the latter cases showing changes due to lymphangitis reticularis chronica (Schurmann).

Tuberculosis of the mouth: A review, with report of a case of chronic miliary tuberculosis complicated by the condition

Tuberculosis of the mouth: A review, with report of a case of chronic miliary tuberculosis complicated by the condition

Chronic Miliary Tuberculosis

A patient with chronic miliary tuberculosis is being reported. The patient had miliary mottling of the lung, tubercular lymphadenitis, cervicitis and disseminated lupus vulgaris of skin lesions.

Chronic Miliary Tuberculosis

Chronic Miliary Tuberculosis

Chronic Miliary Tuberculosis

Chronic Miliary Tuberculosis

Chronic miliary tuberculosis in children

Chronic miliary tuberculosis in children

Chronic miliary tuberculosis

Chronic miliary tuberculosis

Chronic Miliary Tuberculosis and Healed Miliary Tuberculosis

In the past few years there have been a number of reports of healed miliary tuberculosis and chronic miliary tuberculosis, these two terms being rather loosely applied to the cases under discussion. Some of the authors have assumed the two conditions to be the same, while others appear to have made but slight distinction. The two cases here reported will probably help differentiate these two types of lesions—healed and chronic. Case 1. (Fig. 1.) C. W., male, white, age 38. Family history: Father living and well; mother died during childbirth; one brother living and well. No history of tuberculosis, cancer, etc. Chief complaint: Swelling of arm; smothering feeling in chest; shortness of breath. Has had dyspnea ever since he was gassed in 1918; type of gas unknown. In May, 1919, began to have pain in arm. The blood vessels became hard and very prominent. Now entire arm is swollen from the fingers to the shoulder. Physical examination: Blood pressure, systolic 136, diastolic 80; pulse 80. Well nourished. Head negative except pyorrhea and dental caries. Chest broad and well formed. Findings negative. Abdomen negative. Genito-urinary tract negative. Extremities, slight clubbing of fingers. Entire left arm somewhat larger than right. Left hand slightly cyanotic. Superficial veins prominent and sclerosed. Glands of left axilla moderately enlarged. Wassermann negative. Urine negative. Diagnosis: Sclerosis of vessels of left arm; tuberculous lymph nodes of chest; lymphadenitis left axillary glands; no active chest pathology present. Case 2. (Fig. 2.) T. C. B., male, white, age 42. Family history: Father killed in accident; mother died at 50 years, cause unknown; one brother and one sister living and well. Chief complaint: Dyspnea on exertion; headache; cough; pain in cardiac region. Previous history: Measles; mumps; whooping cough; chickenpox during childhood. Genito-urinary tract negative. Present illness: In September, 1918, was ill in hospital for about ten days. Some time later again sick for four or five days; had pneumonia or pleurisy. Tonsils and adenoids removed. Now unable to work as farmer on account of weakness. Physical examination: Negative throughout with exception of double mitral murmurs with slight transmission to axillary line. In Case 1, there is a history of swelling of the arm extending over a period of at least three years. The swelling of the arm came on gradually, presumably due to the increasing axillary adenopathy. In Case 2, there was a slight illness for a few days, but no severe illness of any kind. There is, then, a history covering a period of about three years in the former case, and a history of little or no illness in the latter case. In Case 1 the calcified tubercles are seen to be of varying sizes and of uneven distribution. Even their calcium content appears to vary with the different tubercles, hardly two tubercles being of the same size.