Case Of Variant Angina Research Articles

Prinzmetal's angina in a patient with angiosarcoma of the right cardiac chambers

Variant (Prinzmetal's) angina pectoris is a clinical syndrome characterized by the presence of angina at rest, coinciding with a transient ST-segment elevation. This syndrome is often caused by vasospasm, either on a normal coronary artery or at the site of a coronary atherosclerotic stenosis. We describe a classic case of variant angina associated with an angiosarcoma of the right heart chambers.

A case of variant angina and coronary pulmonary artery fistula

A case of variant angina and coronary pulmonary artery fistula

Vagal nerve–mediated vasospasm–induced lethal ventricular fibrillation

We report a case of variant angina complicated by lethal ventricular fibrillation. Serial ambulatory electrocardiogram monitoring revealed high-frequency spectra reflecting augmented vagal nerve activity preceding the vasospastic episodes. Our case suggests that heart rate variability analysis can help identify potential unexpected sudden cardiac death in patients with variant angina.

Coronary artery stent placement as a treatment of acute coronary syndrome in course of variant angina

High doses of calcium antagonists and nitrates are the treatment of choice in vasospasm angina when no stenosis or mild stenosis is present. In ca. 5–30% patients this kind of treatment is not effective. We present five cases of variant angina with acute coronary syndrome in patients despite standard pharmacological treatment. These patients also did not respond for intracoronary nitroglycerine injection. They were successfully treated with stent implantation in place of vasospasm. Our observations indicate that early enough interventional treatment prevent myocardial infarction. In one case in which stent placement was performed 12 h after clinical manifestation myocardial infarction with Q waves and enzymes release occurred. In others, accurately treated with stent implantation, coronary spasm did not produce myocardial damage. We did not see any clinical and biochemical evidence of myocardial infarction.

Multivessel variant angina unresponsive to urapidil

We present a case of variant angina complicated by recurrent sudden cardiac death. During coronary angiography a diffuse 3-vessel vasoconstriction was observed progressing to a more severe vasoconstriction in the mid LAD. Intracoronary administration of urapidil did not reverse the vasoconstriction of the LAD; instead an occlusive vasospasm occurred accompanied by marked ischaemia.

A case of variant angina associated with eosinophilia

A case of variant angina associated with eosinophilia

A case of variant angina with a history of myocardial infarction: a possible role of vasospastic occlusion in the pathogenesis of myocardial infarction-induction of coronary artery spasm by acetylcholine

A case of variant angina with a history of myocardial infarction: a possible role of vasospastic occlusion in the pathogenesis of myocardial infarction-induction of coronary artery spasm by acetylcholine

A case of variant angina with hypotension after right coronary artery spasm accompanied by ST-segment elevation in lead I during attack

A case of variant angina with hypotension after right coronary artery spasm accompanied by ST-segment elevation in lead I during attack

A case of variant angina with cyclic ST segment elevation

A case of variant angina with cyclic ST segment elevation

症例 急性心筋梗塞発症後,ひき続いて異型狭心症を頻発した1症例

症例 急性心筋梗塞発症後,ひき続いて異型狭心症を頻発した1症例

A case of variant angina with normal coronary arteriogram whose attacks were induced by exercise accompanying with transient abnormal Q waves

A case of variant angina with normal coronary arteriogram whose attacks were induced by exercise accompanying with transient abnormal Q waves

A Case of Variant Angina with Normal Coronary Arteriogram Who Developed Acute Myocardial Infarction and Cerebral Embolism

We report a case of a 44-year-old male with variant angina who developed acute anterior myocardial infarction 2 weeks following demonstration of normal left coronary artery. This experience provides inferential evidence that myocardial ischemia resulting from coronary spasm may progress into myocardial infarction.

A case of variant angina with coronary artery spasm by intravenous injection of ergome trine maleate.

A case of variant angina with coronary artery spasm by intravenous injection of ergome trine maleate.